Assistant Professor, Internal Medicine, University of Michigan Medical School
Internal Medicine
Dr. Kevin K. Kim, MD
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Kevin practices Internal Medicine. He attended University of Michigan Medical School. He has 1 awards "CMS Meaningful Use Stage 1 Certification". Kevin is a published MD. He has 17 publications published. The most current publication "Inhibition of myocardin-related transcription factor/serum response factor signaling decreases lung fibrosis and promotes mesenchymal cell apoptosis." Kevin accepts Medicare payments and is listed with Medicare.gov.
Publications
- Overexpression of Inhibitor of DNA-Binding 2 Attenuates Pulmonary Fibrosis through Regulation of c-Abl and Twist.
- Inhibition of myocardin-related transcription factor/serum response factor signaling decreases lung fibrosis and promotes mesenchymal cell apoptosis.
- Fibrocytes Are Not an Essential Source of Type I Collagen during Lung Fibrosis.
- Activated Alveolar Epithelial Cells Initiate Fibrosis Through Autocrine and Paracrine Secretion of Connective Tissue Growth Factor.
- Paracrine functions of fibrocytes to promote lung fibrosis.
- Activated Alveolar Epithelial Cells Initiate Fibrosis through Secretion of Mesenchymal Proteins.
- A TALEN Genome-Editing System for Generating Human Stem Cell-Based Disease Models.
- Axin Pathway Activity Regulates in Vivo pY654-?-catenin Accumulation and Pulmonary Fibrosis.
- The vitronectin binding function of PAI-1 exacerbates lung fibrosis in mice.
- Alveolar Epithelial Cells Express Mesenchymal Proteins in Patients With Idiopathic Pulmonary Fibrosis.
- Integrin alpha3beta1-dependent beta-catenin phosphorylation links epithelial Smad signaling to cell contacts.
- Epithelial cell alpha3beta1 integrin links beta-catenin and Smad signaling to promote myofibroblast formation and pulmonary fibrosis.
- Endothelin-1 as initiator of epithelial-mesenchymal transition: potential new role for endothelin-1 during pulmonary fibrosis.
- Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix.
- Regulation of alpha5beta1 integrin conformation and function by urokinase receptor binding.
- A plasminogen activator inhibitor-1 promoter polymorphism and idiopathic interstitial pneumonia.
Schools
University of Mi Med Sch
Doctors Specialties
- Internal Medicine
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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University of Michigan Hospitals and Health Centers -
University of Michigan Medical School
Hospital
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University of Michigan Hospitals and Health Centers
Drug Facts
| NPI NUMBER | 1598711632 | |
| NPPES Provider LastName | KIM | |
| NPPES Provider FirstName | KEVIN | |
| NPPES Provider ZIPCode | 481095360 | |
| NPPES Provider State | MI | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 88.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1598711632 |
| Last Name Of The Provider | KIM |
| First Name Of The Provider | KEVIN |
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