Professor, Medicine, Tufts Medical Center
Gastroenterology
Dr. Joel V. Weinstock, MD
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Dr. Joel V. Weinstock works in the field of Gastroenterology. He researched medicine at Wayne State University School of Medicine. Wayne State University School of Medicine has a rank of 63 in research and a 85 in primary care He received awards:"America's Top Doctors", "Boston Magazine", "CMS Meaningful Use Stage 1 Certification", "Fellow (FACP)" and "Elected Member". Joel Weinstock is also a published doctor. He has 58 publication published. The most recent publication is: Tuft cells, taste-chemosensory cells, orchestrate parasite type 2 immunity in the gut. He accepts all Medicare patients.
Publications
- Tuft cells, taste-chemosensory cells, orchestrate parasite type 2 immunity in the gut.
- Do We Need Worms to Promote Immune Health?
- Helminth Infections Decrease Host Susceptibility to Immune-Mediated Diseases.
- Innate Immunity in Disease.
- Randomised clinical trial: the safety and tolerability of Trichuris suis ova in patients with Crohn's Disease.
- Heligmosomoides polygyrus bakeri Infection Activates Colonic Foxp3+ T Cells Enhancing Their Capacity To Prevent Colitis.
- Have worms lost their luster?
- Translatability of helminth therapy in inflammatory bowel Diseases.
- Autoimmunity: The worm returns.
- Assessment of fractionated exhaled nitric oxide as a biomarker for the treatment of eosinophilic Esophagitis.
- Heligmosomoides polygyrus abrogates antigen-specific gut injury in a murine model of inflammatory bowel Disease.
- Helminth-host immunological interactions: prevention and control of immune-mediated Diseases.
- Alteration of the murine gut microbiota during infection with the parasitic helminth Heligmosomoides polygyrus.
- Heligmosomoides polygyrus infection can inhibit colitis through direct interaction with innate immunity.
- Fas ligand expression on T cells is sufficient to prevent prolonged airway inflammation in a murine model of asthma.
- TGF-beta regulates T-cell neurokinin-1 receptor internalization and function.
- Trichuris suis might be effective in treating allergic rhinitis.
- Therapeutic potential of helminth soluble proteins in TNBS-induced colitis in mice.
- Helminths and the IBD hygiene hypothesis.
- CD28 and ICOS play complementary non-overlapping roles in the development of Th2 immunity in vivo.
- Inducible costimulator expression regulates the magnitude of Th2-mediated airway inflammation by regulating the number of Th2 cells.
- Helminthic therapy: using worms to treat immune-mediated Disease.
- Coinfection with the Intestinal nematode Heligmosomoides polygyrus markedly reduces hepatic egg-induced immunopathology and proinflammatory cytokines in mouse models o...
- Colonization with Heligmosomoides polygyrus suppresses mucosal IL-17 production.
- Interleukin-12 (IL-12) and IL-23 induction of substance p synthesis in murine T cells and macrophages is subject to IL-10 and transforming growth factor beta regulation.
- ICOS costimulation expands Th2 immunity by augmenting migration of lymphocytes to draining lymph nodes.
- Heligmosomoides polygyrus promotes regulatory T-cell cytokine production in the murine normal distal intestine.
- Signaling through Fc gamma RIII is required for optimal T helper type (Th)2 responses and Th2-mediated airway inflammation.
- Therapeutic colonization with Trichuris suis.
- Induction of CD8+ regulatory T cells in the intestine by Heligmosomoides polygyrus infection.
- Intestinal helminths protect in a murine model of asthma.
- Helminths and mucosal immune modulation.
- Fas-positive T cells regulate the resolution of airway inflammation in a murine model of asthma.
- Heligmosomoides polygyrus induces TLR4 on murine mucosal T cells that produce TGFbeta after lipopolysaccharide stimulation.
- Anti-interferon-inducible chemokine, CXCL10, reduces colitis by impairing T helper-1 induction and recruitment in mice.
- Why Trichuris suis should prove safe for use in inflammatory bowel Diseases.
- IL-12 induction of mRNA encoding substance P in murine macrophages from the spleen and sites of inflammation.
- Trichuris suis therapy for active Ulcerative colitis: a randomized controlled trial.
- Helminths and the modulation of mucosal inflammation.
- CD4+ T cells from IL-10-deficient mice transfer susceptibility to NSAID-induced Rag colitis.
- Cutting edge: hemokinin has substance P-like function and expression in inflammation.
- The role of substance P, hemokinin and their receptor in governing mucosal inflammation and granulomatous responses.
- Differential regulation of CCL21 in lymphoid/nonlymphoid tissues for effectively attracting T cells to peripheral tissues.
- Treatment of Helicobacter Gastritis with IL-4 requires somatostatin.
- Substance P regulates Th1-type colitis in IL-10 knockout mice.
- Trichuris suis seems to be safe and possibly effective in the treatment of inflammatory bowel Disease.
- Substance P expression correlates with severity of diarrhea in cryptosporidiosis.
- IL-18 and IL-12 signal through the NF-kappa B pathway to induce NK-1R expression on T cells.
- Exposure to schistosome eggs protects mice from TNBS-induced colitis.
- Reduced CTL response and increased viral burden in substance P receptor-deficient mice infected with murine gamma-herpesvirus 68.
- T cell substance P receptor governs antigen-elicited IFN-gamma production.
- CD4+ TCR repertoire heterogeneity in Schistosoma mansoni-induced granulomas.
- Rapid development of colitis in NSAID-treated IL-10-deficient mice.
- Interleukin-4 receptor alpha chain and STAT6 signaling inhibit gamma interferon but not Th2 cytokine expression within schistosome granulomas.
- Established T(H1) granulomatous responses induced by active Mycobacterium avium infection switch to T(H2) following challenge with Schistosoma mansoni.
- IL-4 inhibits vasoactive Intestinal peptide production by macrophages.
- The possible link between de-worming and the emergence of immunological Disease.
Schools
Wayne State University School Of Medicine
University Hospital Of Mi
Procedures Preformed
- Biliary Drainage
- Colonoscopy, Proctosigmoidoscopy, and Sigmoidoscopy
- Endoscopy (Esophagus, Stomach, Small Intestine)
- ERCP (Endoscopic Retrograde Cholangiopancreatography)
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Conditions Treated
- Abdominal Pain
- Anemia
- Barrett's Esophagus
- Benign Neoplasm of the Digestive System
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Doctors Specialties
- Gastroenterology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- BlueConnect HMO
- Blue with Managed Care Behavioral Health HMO
- Preferred Blue PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Commercial Insurance Company
- Coventry Health Care
- Emblem Health
- Fallon Community Health Plan
- First Health (Coventry Health Care) PPO
- Harvard Pilgrim Health Care PPO
- Medicare Advantage PFFS
- MedHealthInsurance
- Medicaid
- Medical Mutual of Ohio
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Network Health
- Principal Life
- Tufts Health Plans
- Tufts PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- America's Top Doctors
- Boston Magazine
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACP)
- Elected Member
Education
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University of Michigan Hospitals and Health Centers -
Wayne State University School of Medicine
Hospital
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Tufts Medical Center
Drug Facts
| NPI NUMBER | 1760420988 | |
| NPPES Provider LastName | WEINSTOCK | |
| NPPES Provider FirstName | JOEL | |
| NPPES Provider ZIPCode | 02111 | |
| NPPES Provider State | MA | |
| Specialty Description | Gastroenterology | |
| Total Claim Count | 245.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 12.0 | |
| Percent Opioid Claims | 4.9 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1760420988 |
| Last Name Of The Provider | WEINSTOCK |
| First Name Of The Provider | JOEL |
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