Professor of Medicine, University of Chicago Medicine
Nephrology
Dr. Frederick G. Toback, MD
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Doctor Frederick G. Toback, MD practices Nephrology. He went to NYU School of Medicine. He received awards:"Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Dr. Frederick G. Toback is also a published physician. He has 86 publication published. The most recent publication is: Cell-crystal interactions and kidney stone formation. He accepts Medicare.
Publications
- Angiotensin-converting enzyme inhibitors predict Acute kidney injury during chemoradiation for head and neck cancer.
- Role of AMP-18 in oral mucositis.
- AMP-18 protects barrier function of colonic epithelial cells: role of tight junction proteins.
- Renal epithelial cells constitutively produce a protein that blocks adhesion of crystals to their surface.
- Peptide fragments of AMP-18, a novel secreted gastric antrum mucosal protein, are mitogenic and motogenic.
- A novel mitogenic protein that is highly expressed in cells of the gastric antrum mucosa.
- Sialic acid-containing glycoproteins on renal cells determine nucleation of Calcium oxalate dihydrate crystals.
- Renal cell-urinary crystal interactions.
- Regulation of renal epithelial cell affinity for Calcium oxalate monohydrate crystals.
- Reduced crystallization inhibition by urine from men With nephrolithiasis.
- Cell-crystal interactions and Kidney stone formation.
- Role of Calcium oxalate monohydrate crystal interactions with renal epithelial cells in the pathogenesis of nephrolithiasis: a review.
- Direct nucleation of Calcium oxalate dihydrate crystals onto the surface of living renal epithelial cells in culture.
- Adhesion, internalization and metabolism of Calcium oxalate monohydrate crystals by renal epithelial cells.
- Adhesion of hydroxyapatite crystals to anionic sites on the surface of Renal epithelial cells.
- Renal cell osteopontin production is stimulated by Calcium oxalate monohydrate crystals.
- Interaction of urinary crystals with Renal epithelial cells in the pathogenesis of nephrolithiasis.
- Face-selective adhesion of Calcium oxalate dihydrate crystals to renal epithelial cells.
- Adhesion of Calcium oxalate monohydrate crystals to anionic sites on the surface of renal epithelial cells.
- Molecular and cell biology of Acute renal failure: new therapeutic strategies.
- Regulation of renal epithelial cell endocytosis of Calcium oxalate monohydrate crystals.
- Regeneration of Kidney tubular epithelial cells.
- Glyceraldehyde-3-phosphate dehydrogenase modifier protein is associated with microtubules in Kidney epithelial cells.
- Renal epithelial cells rapidly bind and internalize Calcium oxalate monohydrate crystals.
- Adhesion of Calcium oxalate monohydrate crystals to renal epithelial cells is inhibited by specific anions.
- Differential gene expression in migrating Renal epithelial cells after wounding.
- Calcium oxalate monohydrate crystals stimulate gene expression in renal epithelial cells.
- Enhancement of Renal medullary pyruvate kinase activity during cell proliferation induced by potassium depletion.
- Amino acid modulation of Renal phosphatidylcholine biosynthesis in the rat.
- New form of acid phosphatase during lysosome biogenesis.
- Induction of growth in Kidney epithelial cells in culture by Na+.
- Kidney epithelial cell growth is stimulated by lowering extracellular potassium concentration.
- Amino acid administration enhances renal protein metabolism after Acute tubular necrosis.
- Stimulation of DNA synthesis in Kidney epithelial cells in culture by potassium.
- Growth of Kidney epithelial cells in culture: evidence for autocrine control.
- Vasopressin stimulates growth of Renal epithelial cells in culture.
- Cell growth and net Na+ flux are inhibited by a protein produced by Kidney epithelial cells in culture.
- Appearance of a cytosolic protein that stimulates glyceraldehyde-3-phosphate dehydrogenase activity during initiation of Renal epithelial cell growth.
- Na regulates growth of Kidney epithelial cells induced by lowering extracellular K concentration.
- Phosphatidylcholine Metabolism during renal growth and regeneration.
- Effect of posture on urinary protein patterns in nonpregmant, pregnant, and toxemic women.
- Phospholipid metabolism in the initiation of renal compensatory growth after Acute reduction of renal mass.
- Effect of hyperosmolality on renal uridine Metabolism.
- Enhancement of uridine uptake in the Kidney papilla by urea.
- Uridine Metabolism during normal and compensatory renal growth.
- Alterations in Renal and plasma amino acid concentrations during Renal compensatory growth.
- Thymidine Metabolism during normal and compensatory renal growth.
- Purine nucleotides stimulate DNA synthesis in Kidney epithelial cells in culture.
- Regulation of glyceraldehyde-3-phosphate dehydrogenase by a cytosolic protein.
- ADP activates protooncogene expression in Renal epithelial cells.
- Lowering extracellular Na+ concentration releases autocrine growth factors from Renal epithelial cells.
- An alternate method utilizing small quantities of ligand for affinity purification of monospecific antibodies.
- Sequence of a human Kidney cDNA clone encoding thymosin beta 10.
- Aberrant responses to growth-regulatory signals by variant Kidney epithelial cells.
- Kinetics of a novel cytosolic protein during the onset of Renal epithelial cell growth.
- Kidney epithelial cells express c-sis protooncogene and secrete PDGF-like protein.
- A novel early growth response gene rapidly induced by fibroblast, epithelial cell and lymphocyte mitogens.
- Extracellular potassium modifies the structure of Kidney epithelial cells in culture.
- Autocrine and paracrine regulation of Kidney epithelial cell growth.
- Macula densa cells of mouse Kidney do not synthesize epidermal growth factor precursor mRNA.
- A human early response gene homologous to murine nur77 and rat NGFI-B, and related to the nuclear receptor superfamily.
- Kidney epithelial cells release growth factors in response to extracellular signals.
- Cell proliferation in the end-stage Kidney.
- Signals that release growth factors from Renal epithelial cells.
- Antimitogenic and mitogenic actions of interleukin-1 in diverse cell types are associated With induction of gro gene expression.
- Adenine nucleotides stimulate migration in wounded cultures of Kidney epithelial cells.
- Regeneration after Acute tubular necrosis.
- Phenol red inhibits growth of Renal epithelial cells.
- Calcium oxalate monohydrate crystals are endocytosed by renal epithelial cells and induce proliferation.
- Endocytosis of Calcium oxalate crystals and proliferation of renal tubular epithelial cells in a patient with type 1 primary hyperoxaluria.
- Cytokeratin reorganization induced by adenosine diphosphate in Kidney epithelial cells.
- Independence of onset of compensatory Kidney growth from changes in renal function.
- Amino acid enhancement of renal regeneration after Acute tubular necrosis.
- Stimulation of Renal phospholipid formation during potassium depletion.
- Phospholipid metabolism during renal regeneration after Acute tubular necrosis.
- Formation of Renal medullary lysosomes during potassium depletion nephropathy.
- Zonal changes in renal structure and phospholipid Metabolism during reversal of potassium depletion nephropathy.
- Metabolic response to Renal compensatory growth.
- Defective mitochondrial energy production during potassium depletion Nephropathy.
- Amino acid-mediated stimulation of renal phospholipid biosynthesis after Acute tubular necrosis.
- Altered bioenergetics in proliferating Renal cells during potassium depletion.
- The role of renal "work" in compensatory Kidney growth.
- Mechanism of enhanced phospholipid formation during potassium depletion Nephropathy.
- Functional defects in mitochondria of Renal inner red medulla during potassium depletion nephropathy.
- Zonal changes in renal structure and phospholipid Metabolism in potassium-deficient rats.
Schools
New York University School Of Medicine
Case West Res Cleveland Met Genl
Beth Israel Hospital Harvard
Boston University
Procedures Preformed
- Hemodialysis
Conditions Treated
- Acidosis
- Acute Glomerulonephritis
- Acute Kidney Failure
- Alkalosis
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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Boston University Medical Center -
Case Western Reserve University - MetroHealth -
NYU School of Medicine
Hospital
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University of Chicago Medical Center
Drug Facts
| NPI NUMBER | 1942363460 | |
| NPPES Provider LastName | TOBACK | |
| NPPES Provider FirstName | FREDERICK | |
| NPPES Provider ZIPCode | 605276686 | |
| NPPES Provider State | IL | |
| Specialty Description | Nephrology | |
| Total Claim Count | 174.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1942363460 |
| Last Name Of The Provider | TOBACK |
| First Name Of The Provider | FREDERICK |
| View All |
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