Professor of Medicine University of Chicago
Nephrology
Dr. Elaine M. Worcester, MD
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Doctor Elaine M. Worcester, MD is an expert in the field of Nephrology. She researched medicine at University of Illinois College of Medicine. She over the years received 2 awards: "CMS Meaningful Use Stage 1 Certification" and "Fellow (ASNF)". Dr. Elaine M. Worcester is also a published doctor. She has 79 paper published. The most recent paper is: Formation of hydrated calcium oxalates in the presence of poly-L-aspartic acid. She is registered with Medicare and accepts Medicare payments.
Publications
- Nephrocalcinosis in Calcium Stone Formers Who Do Not have Systemic Disease.
- Current recommended 25-hydroxyvitamin D targets for chronic Kidney disease management may be too low.
- Micro-CT imaging of Randall's plaques.
- Mechanisms of human Kidney stone formation.
- Biopsy Proven Medullary Sponge Kidney: Clinical Findings, Histopathology, and Role of Osteogenesis in Stone and Plaque Formation.
- Calcium supplementation in chronic Kidney disease.
- Contrasting Histopathology and Crystal Deposits in Kidneys of Idiopathic Stone Formers Who Produce Hydroxy Apatite, Brushite, or Calcium Oxalate Stones.
- A test of the hypothesis that oxalate secretion produces proximal tubule crystallization in primary hyperoxaluria type I.
- Evidence for increased renal tubule and parathyroid gland sensitivity to serum Calcium in human idiopathic hypercalciuria.
- Role of proximal tubule in the hypocalciuric response to thiazide of patients With idiopathic hypercalciuria.
- Kidney stones: an update on current pharmacological management and future directions.
- Evidence for net renal tubule oxalate secretion in patients with calcium Kidney stones.
- Clinical practice. Calcium Kidney stones.
- Renal histopathology and crystal deposits in patients with small bowel resection and Calcium oxalate stone disease.
- Intra-tubular deposits, urine and stone composition are divergent in patients With ileostomy.
- A test of the hypothesis that the collecting duct calcium-sensing receptor limits rise of urine calcium molarity in hypercalciuric calcium Kidney stone formers.
- Does idiopathic hypercalciuria trigger Calcium-sensing receptor-mediated protection from urinary supersaturation?
- Evidence for increased postprandial distal nephron Calcium delivery in hypercalciuric stone-forming patients.
- Urinary cystatin C as an early biomarker of Acute kidney injury following adult cardiothoracic surgery.
- Histopathology and surgical anatomy of patients with primary hyperparathyroidism and Calcium phosphate stones.
- Renal intratubular crystals and hyaluronan staining occur in stone formers with bypass surgery but not with idiopathic Calcium oxalate stones.
- Role of interstitial apatite plaque in the pathogenesis of the common Calcium oxalate stone.
- New insights into the pathogenesis of idiopathic hypercalciuria.
- Mechanism of formation of human Calcium oxalate renal stones on Randall's plaque.
- Evidence that postprandial reduction of renal Calcium reabsorption mediates hypercalciuria of patients with Calcium nephrolithiasis.
- Renal function in patients With nephrolithiasis.
- Endoscopic evidence of calculus attachment to Randall's plaque.
- Nephrolithiasis and increased blood pressure among females With high body mass index.
- Apatite plaque particles in inner medulla of Kidneys of calcium oxalate stone formers: osteopontin localization.
- Decreased renal function among adults With a history of nephrolithiasis: a study of NHANES III.
- Crystal-associated nephropathy in patients With brushite nephrolithiasis.
- Clinical implications of abundant calcium phosphate in routinely analyzed Kidney stones.
- Parathyroid carcinoma in secondary and tertiary Hyperparathyroidism.
- Urine stone risk factors in nephrolithiasis patients with and without bowel Disease.
- Stones from bowel Disease.
- Label-free proteomic methodology for the analysis of human Kidney stone matrix composition.
- Formation of hydrated Calcium oxalates in the presence of poly-L-aspartic acid.
- Urinary biomarkers in the clinical prognosis and early detection of Acute kidney injury.
- Pathophysiology-based treatment of idiopathic calcium Kidney stones.
- Trends in Kidney donation among Kidney stone formers: a survey of US transplant centers.
- Pathophysiological correlates of two unique renal tubule lesions in rats With intestinal resection.
- Renal histopathology of stone-forming patients with distal renal tubular Acidosis.
- Evidence against a contribution of conventional urine risk factors to de novo ESRD Renal stones.
- Kidney stone disease.
- Insights on the pathology of Kidney stone formation.
- Randall's plaque: pathogenesis and role in Calcium oxalate nephrolithiasis.
- Causes and consequences of Kidney loss in patients with nephrolithiasis.
- African American ESRD patients have a high pre-dialysis prevalence of Kidney stones compared to NHANES III.
- Renal inter-alpha-trypsin inhibitor heavy chain 3 increases in Calcium oxalate stone-forming patients.
- Renal crystal deposits and histopathology in patients With cystine stones.
- Control of Calcium oxalate crystal structure and cell adherence by urinary macromolecules.
- Inhibitors of stone formation.
- Urinary oxalate excretion increases with body size and decreases with increasing dietary Calcium intake among healthy adults.
- The effect of warfarin on urine Calcium oxalate crystal growth inhibition and urinary excretion of Calcium and nephrocalcin.
- Effect of Renal transplantation on serum oxalate and urinary oxalate excretion.
- Urinary Calcium oxalate crystal growth inhibitors.
- Osteopontin inhibits nucleation of Calcium oxalate crystals.
- Upregulation of osteopontin expression by ischemia in rat Kidney.
- Osteopontin production by cultured Kidney cells.
- Expression of osteopontin, a urinary inhibitor of stone mineral crystal growth, in rat Kidney.
- Evidence that serum calcium oxalate supersaturation is a consequence of oxalate retention in patients with chronic renal Failure.
- Glycoprotein Calcium oxalate crystal growth inhibitor in urine.
- Crystal adsorption and growth slowing by nephrocalcin, albumin, and Tamm-Horsfall protein.
- Hypercalciuria and Stones.
- The calcium oxalate crystal growth inhibitor protein produced by mouse Kidney cortical cells in culture is osteopontin.
- Effects of urinary macromolecules on hydroxyapatite crystal formation.
- Role of anionic proteins in Kidney stone formation: interaction between model anionic polypeptides and calcium oxalate crystals.
- Nephrolithiasis and nephrocalcinosis in rats With small bowel resection.
- A new animal model of hyperoxaluria and nephrolithiasis in rats With small bowel resection.
- Reduced Renal function and benefits of treatment in cystinuria vs other forms of nephrolithiasis.
- Urine pH in renal Calcium stone formers who do and do not increase stone phosphate content with time.
- A formal test of the hypothesis that idiopathic Calcium oxalate stones grow on Randall''s plaque.
- Clinical and laboratory characteristics of Calcium stone-formers with and without primary hyperparathyroidism.
- Three pathways for human Kidney stone formation.
- Evidence for altered renal tubule function in idiopathic Calcium stone formers.
- Plaque and deposits in nine human stone Diseases.
- In idiopathic Calcium oxalate stone-formers, unattached stones show evidence of having originated as attached stones on Randall''s plaque.
- Comparison of the pathology of interstitial plaque in human ICSF stone patients to NHERF-1 and THP-null mice.
Schools
University Of Illinois College Of Medicine Chicago
Loyola University Med Center
University of Chicago
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Calcium Metabolism Disorders
- Chronic Kidney Diseases
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (ASNF)
Education
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University of Chicago -
Loyola University -
University of Illinois College of Medicine
Hospital
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University of Chicago Medical Center
Drug Facts
| NPI NUMBER | 1568525087 | |
| NPPES Provider LastName | WORCESTER | |
| NPPES Provider FirstName | ELAINE | |
| NPPES Provider ZIPCode | 605276686 | |
| NPPES Provider State | IL | |
| Specialty Description | Nephrology | |
| Total Claim Count | 224.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1568525087 |
| Last Name Of The Provider | WORCESTER |
| First Name Of The Provider | ELAINE |
| View All |
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