Assistant Professor of Pathology, University of Chicago Medicine
Pathology
Dr. Christopher R. Weber, MD
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Christopher Weber works in the field of Pathology. He went to medical school at Loyola University Chicago Stritch School of Medicine. Christopher Weber is a published MD as well. He has 36 publications published. The latest was: Spectral biomarkers for chemoprevention of colonic neoplasia: a placebo-controlled double-blinded trial with aspirin. He is registered with Medicare.
Publications
- Spectral biomarkers for chemoprevention of colonic neoplasia: a placebo-controlled double-blinded trial with aspirin.
- Molecular aspects of tight junction barrier function.
- Rectal leiomyosarcoma in association with ulcerative colitis: a rare condition with an unusual presentation.
- β-Catenin Promotes Colitis and Colon Cancer Through Imprinting of Proinflammatory Properties in T Cells.
- Occludin OCEL domain interactions are required for maintenance and regulation of the tight junction barrier to macromolecular flux.
- Chronic Pouchitis Versus Recurrent Crohn's Disease: A Diagnostic Challenge.
- Bifidobacteria Stabilize Claudins at Tight Junctions and Prevent Intestinal Barrier Dysfunction in Mouse Necrotizing Enterocolitis.
- Capsaicin induces NKCC1 internalization and inhibits chloride secretion in colonic epithelial cells independently of TRPV1.
- Topical Polyethylene Glycol as a Novel Chemopreventive Agent for Oral Cancer via Targeting of Epidermal Growth Factor Response.
- Dynamic properties of the tight junction barrier.
- Polyethylene glycol diminishes pathological effects of Citrobacter rodentium infection by blocking bacterial attachment to the colonic epithelia.
- Occludin S408 phosphorylation regulates tight junction protein interactions and barrier function.
- Tight junction pore and leak pathways: a dynamic duo.
- Tumor suppressor FOXO3 mediates signals from the EGF receptor to regulate proliferation of colonic cells.
- Phosphoinositide 3-kinase signaling mediates beta-catenin activation in intestinal epithelial stem and progenitor cells in colitis.
- MLCK-dependent exchange and actin binding region-dependent anchoring of ZO-1 regulate tight junction barrier function.
- Epithelial myosin light chain kinase activation induces mucosal interleukin-13 expression to alter tight junction ion selectivity.
- Caveolin-1-dependent occludin endocytosis is required for TNF-induced tight junction regulation in vivo.
- Tumor suppressor FOXO3 participates in the regulation of intestinal inflammation.
- Retinoic acid-induced gene-1 (RIG-I) associates with the actin cytoskeleton via caspase activation and recruitment domain-dependent interactions.
- Tumor suppressor Foxo3a is involved in the regulation of lipopolysaccharide-induced interleukin-8 in intestinal HT-29 cells.
- Claudin-1 and claudin-2 expression is elevated in inflammatory bowel disease and may contribute to early neoplastic transformation.
- The tight junction protein complex undergoes rapid and continuous molecular remodeling at steady state.
- MMP25 (MT6-MMP) is highly expressed in human colon cancer, promotes tumor growth, and exhibits unique biochemical properties.
- Modulation of the pancreatic islet beta-cell-delayed rectifier potassium channel Kv2.1 by the polyunsaturated fatty acid arachidonate.
- Dynamic regulation of sodium/calcium exchange function in human heart failure.
- Cardiac submembrane [Na+] transients sensed by Na+-Ca2+ exchange current.
- Cellular basis of abnormal calcium transients of failing human ventricular myocytes.
- Calcium influx via I(NCX) is favored in failing human ventricular myocytes.
- Na/Ca exchange function in intact ventricular myocytes.
- Simultaneous measurement of [Na]i, [Ca]i, and I(NCX) in intact cardiac myocytes.
- Modulation of contractility in failing human myocytes by reverse-mode Na/Ca exchange.
- Intracellular Na(+) concentration is elevated in heart failure but Na/K pump function is unchanged.
- Na(+)-Ca(2+) exchange current and submembrane [Ca(2+)] during the cardiac action potential.
- Genistein inhibits proliferation of colon cancer cells by attenuating a negative effect of epidermal growth factor on tumor suppressor FOXO3 activity.
Schools
Loyola University of Chicago Stritch School of Medicine
University Of Chicago Hospitals and Health System
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
Education
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University of Chicago -
Loyola University Chicago Stritch School of Medicine
Hospital
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University of Chicago Medical Center
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