Professor, Pathology, East Tennessee State University James H Quillen College of Medicine
Pathology
Dr. Charles E. Ganote, MD
Charles Ganote practices Pathology. He went to medical school at Vanderbilt University School of Medicine. Charles Ganote has written publications. He has 60 publications published with PubMed. The lastest publication is: 'Fostriecin, an inhibitor of protein phosphatase 2A, limits myocardial infarct size even when administered after onset of ischemia.'
Publications
- Sudden death and isolated right ventricular noncompaction cardiomyopathy: report of 2 autopsied adult cases.
- A mass of the right lacrimal sac in a 53-year-old man.
- Acute oxalate nephropathy associated with orlistat: a case report with a review of the literature.
- Cellular swelling and irreversible myocardial injury. Effects of polyethylene glycol and mannitol in perfused rat hearts.
- Ultrastructural studies of vasopressin effect on isolated perfused renal collecting tubules of the rabbit.
- Effect of a transient period of ischemia on myocardial cells. II. Fine structure during the first few minutes of reflow.
- Cell to cell interactions contributing to the "oxygen paradox".
- Effects of anoxic or oxygenated reperfusion in globally ischemic, isovolumic, perfused rat hearts.
- Experimental myocardial ischemic injury. I. A comparison of Stadie-Riggs and free-hand slicing techniques on tissue ultrastructure, water and electrolytes during in vi...
- Effects of hyperosmolar solutions of polyethylene glycol, dextran or mannitol on enzyme release from perfused rat hearts.
- Oxygen-induced enzyme release: early events and a proposed mechanism.
- What constitutes the calcium paradox?
- Experimental myocardial ischemic injury. II. Effect of in vivo ischemia on dog heart slice function in vitro.
- Effects of the free radical scavenger DMTU and mannitol on the oxygen paradox in perfused rat hearts.
- Contracture and the calcium paradox.
- Morphologic aspects of reperfusion injury.
- Increased myocyte fragility following anoxic injury.
- 2,4-Dinitrophenol (DNP)-induced injury in calcium-free hearts.
- Physical stress-mediated enzyme release from calcium-deficient hearts.
- Anoxia, calcium and contracture as mediators of myocardial enzyme release.
- Small cell carcinoma of the cervix in liquid-based Pap test: Utilization of split-sample immunocytochemical and molecular analysis.
- Oxygen-induced enzyme release after irreversible myocardial injury. Effects of cyanide in perfused rat hearts.
- Effect of a transient period of ischemia on myocardial cells. I. Effects on cell volume regulation.
- Irreversible injury of isolated adult rat myocytes. Osmotic fragility during metabolic inhibition.
- Contraction band necrosis and irreversible myocardial injury.
- Species variation in anoxic-induced damage of heart muscle.
- An in vitro model of myocardial ischemia utilizing isolated adult rat myocytes.
- Protection from irreversible hypoxic injury by potassium cardioplegia and hypothermia: effects on contracture, morphology and O2-enzyme release.
- Oxygen-induced enzyme release. Assessment of mitochondrial function in anoxic myocardial injury and effects of the mitochondrial uncoupling agent 2,4-dinitrophenol (DNP).
- Effects of the phospholipase inhibitor mepacrine on injury in ischemic and metabolically inhibited adult isolated myocytes.
- Fostriecin, an inhibitor of protein phosphatase 2A, limits myocardial infarct size even when administered after onset of ischemia.
- Effects of calcium depletion and loading on injury during metabolic inhibition of isolated adult rat myocytes.
- Effects of 2,3-butanedione monoxime (BDM) on contracture and injury of isolated rat myocytes following metabolic inhibition and ischemia.
- Adenosine and preconditioning in the rat heart.
- Ischemic loss of sarcolemmal dystrophin and spectrin: correlation with myocardial injury.
- Translocation of PKC, protein phosphatase inhibition and preconditioning of rabbit cardiomyocytes.
- Preconditioning of isolated rabbit cardiomyocytes: induction by metabolic stress and blockade by the adenosine antagonist SPT and calphostin C, a protein kinase C inhi...
- In vitro ischaemic preconditioning of isolated rabbit cardiomyocytes: effects of selective adenosine receptor blockade and calphostin C.
- Preconditioning of isolated rabbit cardiomyocytes: no evident separation of induction, memory and protection.
- Ischaemia and the myocyte cytoskeleton: review and speculation.
- Flow cytometric analysis of isolated adult cardiomyocytes: vinculin and tubulin fluorescence during metabolic inhibition and ischemia.
- Effects of the protein phosphatase inhibitors okadaic acid and calyculin A on metabolically inhibited and ischaemic isolated myocytes.
- Differential translocation or phosphorylation of alpha B crystallin cannot be detected in ischemically preconditioned rabbit cardiomyocytes.
- Concentration-response relationships for adenosine agonists during preconditioning of rabbit cardiomyocytes.
- Potassium channels and preconditioning of isolated rabbit cardiomyocytes: effects of glyburide and pinacidil.
- Adenosine and A1 selective agonists offer minimal protection against ischaemic injury to isolated rat cardiomyocytes.
- Adenosine receptor specificity in preconditioning of isolated rabbit cardiomyocytes: evidence of A3 receptor involvement.
- Phosphorylation state of hsp27 and p38 MAPK during preconditioning and protein phosphatase inhibitor protection of rabbit cardiomyocytes.
- Protein phosphatase inhibitors calyculin A and fostriecin protect rabbit cardiomyocytes in late ischemia.
- Comparison of in vitro preconditioning responses of isolated pig and rabbit cardiomyocytes: effects of a protein phosphatase inhibitor, fostriecin.
- Preconditioning of isolated rabbit cardiomyocytes: effects of glycolytic blockade, phorbol esters, and ischaemia.
- Sarcolemmal blebs and osmotic fragility as correlates of irreversible ischemic injury in preconditioned isolated rabbit cardiomyocytes.
- Negative image of blastomyces on diff-quik stain.
- Localization of cholinergic innervation in guinea pig heart by immunohistochemistry for high-affinity choline transporters.
- Effects of CCCP-induced mitochondrial uncoupling and cyclosporin A on cell volume, cell injury and preconditioning protection of isolated rabbit cardiomyocytes.
- Enhanced cell volume regulation: a key protective mechanism of ischemic preconditioning in rabbit ventricular myocytes.
- An ischemic beta-dystroglycan (betaDG) degradation product: correlation with irreversible injury in adult rabbit cardiomyocytes.
- Dystrophin-associated protein complex and heart failure.
- Fatal abdominal sarcomatosis secondary to gastrointestinal stromal tumor with bland histology.
Schools
Vanderbilt University
Doctors Specialties
- Forensic Pathology
- Pathology
Accepted Insurances
Education
-
Vanderbilt University -
Vanderbilt University School of Medicine
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