Professor of Medicine, Immune Disease Institute
Allergy & Immunology
Dr. Anne E. Goldfeld, MD
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Anne E. Goldfeld is an expert in the field of Allergy & Immunology. She graduated from Albert Einstein College of Medicine of Yeshiva University. Doctor Anne E. Goldfeld, MD is a published physician. She published 70 publications, including: 'Identification of three new single nucleotide polymorphisms in the human tumor necrosis factor-alpha gene promoter.'
Publications
- Achieving high treatment success for multidrug-resistant TB in Africa: initiation and scale-up of MDR TB care in Ethiopia-an observational cohort study.
- T cells and adaptive immunity to Mycobacterium tuberculosis in humans.
- TB-IRIS and remodelling of the T-cell compartment in highly immunosuppressed HIV+ patients with TB: the CAPRI-T (ANRS-12614) study.
- A Distal Locus Element Mediates IFN-γ Priming of Lipopolysaccharide-Stimulated TNF Gene Expression.
- Regulation of Mycobacterium tuberculosis-Dependent HIV-1 Transcription Reveals a New Role for NFAT5 in the Toll-Like Receptor Pathway.
- Monocyte-specific accessibility of a matrix attachment region in the tumor necrosis factor locus.
- The use of HaloTag-based technology in flow and laser scanning cytometry analysis of live and fixed cells.
- Arc of a vicious circle: pathways activated by Mycobacterium tuberculosis that target the HIV-1 LTR.
- HIV-1 replication is differentially regulated by distinct clinical strains of Mycobacterium tuberculosis.
- A dimer-specific function of the transcription factor NFATp.
- Activation-dependent intrachromosomal interactions formed by the TNF gene promoter and two distal enhancers.
- Treatment strategies for HIV-infected patients with tuberculosis: ongoing and planned clinical trials.
- Post-induction, stimulus-specific regulation of tumor necrosis factor mRNA expression.
- Primate TNF promoters reveal markers of phylogeny and evolution of innate immunity.
- NFAT5 regulates HIV-1 in primary monocytes via a highly conserved long terminal repeat site.
- Transactivator of transcription from HIV type 1 subtype E selectively inhibits TNF gene expression via interference with chromatin remodeling of the TNF locus.
- Geographical distribution and disease associations of the CD45 exon 6 138G variant.
- Aspartic acid homozygosity at codon 57 of HLA-DQ beta is associated with susceptibility to pulmonary tuberculosis in Cambodia.
- Identification of a macrophage-specific chromatin signature in the IL-10 locus.
- IL-10-producing and naturally occurring CD4+ Tregs: limiting collateral damage.
- A community-based tuberculosis program in Cambodia.
- Mycobacterium tuberculosis recall antigens suppress HIV-1 replication in anergic donor cells via CD8+ T cell expansion and increased IL-10 levels.
- The -1030/-862-linked TNF promoter single-nucleotide polymorphisms are associated with the inability to control HIV-1 viremia.
- T cell-specific expression of the human TNF-alpha gene involves a functional and highly conserved chromatin signature in intron 3.
- Regulation of tumor necrosis factor alpha gene expression by mycobacteria involves the assembly of a unique enhanceosome dependent on the coactivator proteins CBP/p300.
- Ethnic-specific genetic associations with pulmonary tuberculosis.
- Antigen-specific and persistent tuberculin anergy in a cohort of pulmonary tuberculosis patients from rural Cambodia.
- Inducer-specific enhanceosome formation controls tumor necrosis factor alpha gene expression in T lymphocytes.
- Efficient transcription of the Epstein-Barr virus immediate-early BZLF1 and BRLF1 genes requires protein synthesis.
- HIV-1 infection does not induce tumor necrosis factor-alpha or interferon-beta gene transcription.
- Human tumor necrosis factor alpha gene regulation in phorbol ester stimulated T and B cell lines.
- Human tumor necrosis factor alpha gene regulation by virus and lipopolysaccharide.
- Coordinate viral induction of tumor necrosis factor alpha and interferon beta in human B cells and monocytes.
- The physical and psychological sequelae of torture. Symptomatology and diagnosis.
- Murder in Guatemala.
- Recessive lethal deletion on mouse chromosome 7 affects glucocorticoid receptor binding activities.
- Genetic control of insulin receptors.
- Tumor necrosis factor alpha is an autocrine growth factor for normal human B cells.
- Cyclosporin A and FK506 block induction of the Epstein-Barr virus lytic cycle by anti-immunoglobulin.
- Transcriptional activation of the human TNF-alpha promoter by superantigen in human monocytic cells: role of NF-kappa B.
- The role of NFATp in cyclosporin A-sensitive tumor necrosis factor-alpha gene transcription.
- Calcineurin mediates human tumor necrosis factor alpha gene induction in stimulated T and B cells.
- Identification of a novel cyclosporin-sensitive element in the human tumor necrosis factor alpha gene promoter.
- Tumor necrosis factor alpha gene regulation in activated T cells involves ATF-2/Jun and NFATp.
- Cell-type-specific regulation of the human tumor necrosis factor alpha gene in B cells and T cells by NFATp and ATF-2/JUN.
- TNF-alpha and genetic susceptibility to parasitic disease.
- Association of an HLA-DQ allele with clinical tuberculosis.
- Involvement of Bruton's tyrosine kinase in FcepsilonRI-dependent mast cell degranulation and cytokine production.
- Identification of three new single nucleotide polymorphisms in the human tumor necrosis factor-alpha gene promoter.
- Signaling through the lymphotoxin-beta receptor stimulates HIV-1 replication alone and in cooperation with soluble or membrane-bound TNF-alpha.
- Engagement of tumor necrosis factor (TNF) receptor 1 leads to ATF-2- and p38 mitogen-activated protein kinase-dependent TNF-alpha gene expression.
- Stimulus-specific assembly of enhancer complexes on the tumor necrosis factor alpha gene promoter.
- A stimulus-specific role for CREB-binding protein (CBP) in T cell receptor-activated tumor necrosis factor alpha gene expression.
- IL-10-producing T cells suppress immune responses in anergic tuberculosis patients.
- Identification of phylogenetic footprints in primate tumor necrosis factor-alpha promoters.
- A lipopolysaccharide-specific enhancer complex involving Ets, Elk-1, Sp1, and CREB binding protein and p300 is recruited to the tumor necrosis factor alpha promoter in...
- Nuclear factor of activated T cells transcription factor NFATp controls superantigen-induced lethal shock.
- Post-genomics and the neutral theory: variation and conservation in the tumor necrosis factor-alpha promoter.
- Control of HIV-1 viremia and protection from AIDS are associated with HLA-Bw4 homozygosity.
- Epigenetic Control of Cytokine Gene Expression: Regulation of the TNF/LT Locus and T Helper Cell Differentiation.
- Earlier versus later start of antiretroviral therapy in HIV-infected adults with tuberculosis.
- NFAT5 binds to the TNF promoter distinctly from NFATp, c, 3 and 4, and activates TNF transcription during hypertonic stress alone.
- Transcriptional control of the TNF gene.
- Pathogenesis and management of HIV/TB co-infection in Asia.
- Lentiviral delivery of short hairpin RNAs protects CD4 T cells from multiple clades and primary isolates of HIV.
- Genetic susceptibility to pulmonary tuberculosis in Cambodia.
- TNF-alpha promoter single nucleotide polymorphisms are markers of human ancestry.
- Diagnostic and clinical implications of response to tuberculin in two ethnically distinct populations from Peru and Cambodia.
- Transcription of the tumor necrosis factor alpha gene is rapidly induced by anti-immunoglobulin and blocked by cyclosporin A and FK506 in human B cells.
Schools
Albert Einstein College of Medicine of Yeshiva University
Mass General Hospital
Doctors Specialties
- Internal Medicine
- Allergy
- Immunology
Accepted Insurances
Education
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Massachusetts General Hospital -
Albert Einstein College of Medicine of Yeshiva University
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