Professor/Endocrinology and Metabolism
Endocrinology
Dr. William I. Sivitz, MD
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William Sivitz works in the field of Endocrinology. He researched medicine at Drexel University College of Medicine. He has 2 awards "Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". William Sivitz has written publications. He has 36 publications published with PubMed. The lastest paper is: 'Evidence for metabolic aberrations in asymptomatic persons with type 2 diabetes after initiation of simvastatin therapy.' Dr. William I. Sivitz accepts Medicare.
Publications
- Evidence for metabolic aberrations in asymptomatic persons with type 2 Diabetes after initiation of simvastatin therapy.
- A Mitochondrial-Targeted Coenzyme Q Analog Prevents Weight Gain and Ameliorates Hepatic Dysfunction in High Fat-Fed Mice.
- Joiner et al. reply.
- Dietary fat, fatty acid saturation and mitochondrial bioenergetics.
- Mitochondrial Function in Diabetes: Novel Methodology and New Insight.
- CaMKII determines mitochondrial stress responses in heart.
- Endothelial cell and platelet bioenergetics: effect of glucose and nutrient composition.
- Bioenergetic Effects of Mitochondrial Targeted Coenzyme Q Analogs in Endothelial Cells.
- Peroxisome proliferator-activated receptor γ decouples fatty acid uptake from Lipid inhibition of insulin signaling in skeletal muscle.
- Mitochondrial superoxide and Coenzyme Q in insulin deficient rats: Increased electron leak.
- Feeding frequency and appetite in lean and obese prepubertal children.
- Mitochondrial dysfunction in Diabetes: from molecular mechanisms to functional significance and therapeutic opportunities.
- Superoxide production by mitochondria of insulin-sensitive tissues: mechanistic differences and effect of early Diabetes.
- Superoxide and respiratory coupling in mitochondria of insulin-deficient Diabetic rats.
- Mitochondrial targeted coenzyme Q, superoxide, and fuel selectivity in endothelial cells.
- Endogenous peroxisome proliferator-activated receptor-gamma augments fatty acid uptake in oxidative muscle.
- Mitochondrial proton leak in obesity-resistant and obesity-prone mice.
- Obesity impairs vascular relaxation in human subjects: hyperglycemia exaggerates adrenergic vasoconstriction arterial dysfunction in obesity and Diabetes.
- Ectopic brown adipose tissue in muscle provides a mechanism for differences in risk of metabolic Syndrome in mice.
- Reactive oxygen and targeted antioxidant administration in endothelial cell mitochondria.
- Antecedent hypoglycemia, catecholamine depletion, and subsequent sympathetic neural responses.
- Respiratory uncoupling by UCP1 and UCP2 and superoxide generation in endothelial cell mitochondria.
- Free fatty acid-induced beta-cell defects are dependent on uncoupling protein 2 expression.
- Adiponectin and C-reactive protein in obesity, type 2 Diabetes, and monodrug therapy.
- Cyclic changes in glycemia assessed by continuous glucose monitoring system during multiple complete menstrual cycles in women with type 1 Diabetes.
- Understanding insulin resistance. What are the clinical implications?
- Leptin administration to normal rats does not alter catecholamine responsiveness to insulin-induced hypoglycemia.
- Leptin and body fat in type 2 Diabetes and monodrug therapy.
- Leptin potentiates thermogenic sympathetic responses to hypothermia: a receptor-mediated effect.
- Uncoupling Metabolism and coupling leptin to cardiovascular disease.
- UCP2-dependent proton leak in isolated mammalian mitochondria.
- Hemodynamic consequences of neuropeptide Y-induced obesity.
- The concept of selective leptin resistance: evidence from agouti yellow obese mice.
- Simultaneous quantification of mitochondrial ATP and ROS production.
- Human iPS cell-derived insulin producing cells form vascularized organoids under the kidney capsules of Diabetic mice.
Schools
Drexel University College of Medicine
Baystate Med Center
University Of Michigan
Conditions Treated
- Abnormal Thyroid
- Calcium Metabolism Disorders
- Craniopharyngioma
- Diabetes Mellitus, Secondary
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Doctors Specialties
- Endocrinology
Accepted Insurances
- Choice Plus POS II
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- Medicare MCR
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
- Universal American
- Alliance Select
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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Baystate Medical Center - Tufts University -
University of Michigan Hospitals and Health Centers -
Drexel University College of Medicine
Drug Facts
| NPI NUMBER | 1891783445 | |
| NPPES Provider LastName | SIVITZ | |
| NPPES Provider FirstName | WILLIAM | |
| NPPES Provider ZIPCode | 522421009 | |
| NPPES Provider State | IA | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 827.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1891783445 |
| Last Name Of The Provider | SIVITZ |
| First Name Of The Provider | WILLIAM |
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