Physician
Internal Medicine
Dr. William Cruikshank, MD
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William Cruikshank practices Internal Medicine. He researched medicine at University of Illinois College of Medicine. He over the years received 2 awards: "Diabetes Recognition Program" and "CMS Meaningful Use Stage 1 Certification". William Cruikshank is a published doctor. He has 99 papers published. The most current publication "Processing and release of IL-16 from CD4+ but not CD8+ T cells is activation dependent." Doctor William Cruikshank, MD accepts Medicare.
Publications
- Relation between stress and cytokine responses in inner-city mothers.
- Absence of Full-length Neurokinin-1 Receptor Protein Expression by Cutaneous T-cells: Implications for Substance P-mediated Signaling in Cutaneous T-cell Lymphoma.
- Emerging role of IL-16 in cytokine-mediated regulation of multiple sclerosis.
- An NT4/TrkB-dependent increase in innervation links early-life allergen exposure to persistent airway hyperreactivity.
- Regulation of cellular processes by interleukin-16 in homeostasis and cancer.
- Mannose-capped lipoarabinomannan from Mycobacterium tuberculosis preferentially inhibits sphingosine-1-phosphate-induced migration of Th1 cells.
- Maturation of B cell precursors is impaired in thymic-deprived nude and old mice.
- Inhibition of the elicitation phase of contact hypersensitivity by thymidine dinucleotides is in part mediated by increased expression of interleukin-10 in human kerat...
- Biological activity of interleukin-16.
- Interleukin-16 as a marker of Sézary syndrome onset and stage.
- Herbal medicine treatment reduces inflammation in a murine model of cockroach allergen-induced asthma.
- Introduction of pro-interleukin-16 inhibits T-lymphoblastic leukemia growth in mice.
- Reducing LPS content in cockroach allergens increases pulmonary cytokine production without increasing inflammation: a randomized laboratory study.
- lnterleukin-16: the ins and outs of regulating T-cell activation.
- Association of asthma with a functional promoter polymorphism in the IL16 gene.
- Preferential migration of T regulatory cells induced by IL-16.
- Igs from patients with Graves' disease induce the expression of T cell chemoattractants in their fibroblasts.
- Differential roles of IL-16 and CD28/B7 costimulation in the generation of T-lymphocyte chemotactic activity in the bronchial mucosa of mild and moderate asthmatic ind...
- Prointerleukin-16 contains a functional CcN motif that regulates nuclear localization.
- Histamine h(4) and h(2) receptors control histamine-induced interleukin-16 release from human CD8(+) T cells.
- IL-16 promotes leukotriene C(4) and IL-4 release from human eosinophils via CD4- and autocrine CCR3-chemokine-mediated signaling.
- Immunomodulatory effect of interleukin-16 on allergic airway inflammation.
- Binding of HTLV-1 tax oncoprotein to the precursor of interleukin-16, a T cell PDZ domain-containing protein.
- Tumor necrosis factor-alpha-induced synthesis of interleukin-16 in airway epithelial cells: priming for serotonin stimulation.
- Regulation of nuclear Prointerleukin-16 and p27(Kip1) in primary human T lymphocytes.
- Increased levels of bioactive IL-16 correlate with disease activity during relapsing experimental autoimmune encephalomyelitis (EAE).
- Anti-IL-16 therapy reduces CD4+ T-cell infiltration and improves paralysis and histopathology of relapsing EAE.
- Synovial fibroblasts from patients with rheumatoid arthritis, like fibroblasts from Graves' disease, express high levels of IL-16 when treated with Igs against insulin...
- The effect of interleukin-16 and its precursor on T lymphocyte activation and growth.
- Interleukin-16 and peptide derivatives as immunomodulatory therapy in allergic lung disease.
- Nuclear pro-IL-16 regulation of T cell proliferation: p27(KIP1)-dependent G0/G1 arrest mediated by inhibition of Skp2 transcription.
- Cutting edge: IL-16/CD4 preferentially induces Th1 cell migration: requirement of CCR5.
- Immunomodulatory cytokines in asthmatic inflammation.
- Sustained linked stimulation via CD3 and CD4 is required for the IL-4-independent development of IL-4 synthesizing CD4+ T cells.
- Cytokine-induced lymphocyte chemoattraction from cultured human thyrocytes: evidence for interleukin-16 and regulated upon activation, normal T cell expressed, and sec...
- Immunoglobulin activation of T cell chemoattractant expression in fibroblasts from patients with Graves' disease is mediated through the insulin-like growth factor I r...
- Histamine 4 receptor activation induces recruitment of FoxP3+ T cells and inhibits allergic asthma in a murine model.
- Standardization and performance evaluation of mononuclear cell cytokine secretion assays in a multicenter study.
- Production of IL-16 correlates with CD4+ Th1 inflammation and phosphorylation of axonal cytoskeleton in multiple sclerosis lesions.
- Chemokine receptor CXCR3 desensitization by IL-16/CD4 signaling is dependent on CCR5 and intact membrane cholesterol.
- Immunoglobulin G from patients with Graves' disease induces interleukin-16 and RANTES expression in cultured human thyrocytes: a putative mechanism for T-cell infiltra...
- Immunosuppressive effect on T cell activation by interleukin-16- and interleukin-10-cDNA-double-transfected human squamous cell line.
- Functional Th1 cells are required for surgical adhesion formation in a murine model.
- Neutralization of interleukin-16 protects nonobese diabetic mice from autoimmune type 1 diabetes by a CCL4-dependent mechanism.
- A homologous form of human interleukin 16 is implicated in microglia recruitment following nervous system injury in leech Hirudo medicinalis.
- Parental characteristics, somatic fetal growth, and season of birth influence innate and adaptive cord blood cytokine responses.
- Dermal nitrite application enhances global nitric oxide availability: new therapeutic potential for immunomodulation?
- Characterization of regulatory T cells in urban newborns.
- Increased frequency and compromised function of T regulatory cells in systemic sclerosis (SSc) is related to a diminished CD69 and TGFbeta expression.
- Human immunodeficiency virus type 1 gp120 reprogramming of CD4+ T-cell migration provides a mechanism for lymphadenopathy.
- Pro-IL-16 recruits histone deacetylase 3 to the Skp2 core promoter through interaction with transcription factor GABP.
- Lung lining fluid glutathione attenuates IL-13-induced asthma.
- Loss of nuclear pro-IL-16 facilitates cell cycle progression in human cutaneous T cell lymphoma.
- Diesel exhaust particulates exacerbate asthma-like inflammation by increasing CXC chemokines.
- Longitudinal relationship of early life immunomodulatory T cell phenotype and function to development of allergic sensitization in an urban cohort.
- Decreased renal ischemia-reperfusion injury by IL-16 inactivation.
- Transforming growth factor beta1 and laminin-111 cooperate in the induction of interleukin-16 expression in synovial fibroblasts from patients with rheumatoid arthritis.
- Measurement of interleukin 16.
- Exogenous interleukin-16 inhibits antigen-induced airway hyper-reactivity, eosinophilia and Th2-type cytokine production in mice.
- Functional characteristics of histamine receptor-bearing mononuclear cells. II. Identification and characterization of two histamine-induced human lymphokines that inh...
- Modulation of lymphocyte migration by human lymphokines. III. Characterization of a lymphocyte migration inhibitory factor (LyMIF35K).
- Chemoattractant lymphokines specific for the helper/inducer T-lymphocyte subset.
- Lymphokine activation of T4+ T lymphocytes and monocytes.
- A human T-T-cell hybridoma-derived lymphocyte chemoattractant factor.
- Lymphocyte motility and lymphocyte chemoattractant factors.
- Lymphocyte activation by HIV-1 envelope glycoprotein.
- CD4-mediated stimulation of human eosinophils: lymphocyte chemoattractant factor and other CD4-binding ligands elicit eosinophil migration.
- Identification of HIV-1 envelope glycoprotein in the serum of AIDS and ARC patients.
- Lymphocyte chemoattractant factor induces CD4-dependent intracytoplasmic signaling in lymphocytes.
- Evidence for recent as well as long term activation of T cells migrating through endothelial cell monolayers in vitro.
- Biologic activities of HIV-1 envelope glycoprotein: the effects of crosslinking.
- Prospects for IL-16 in the treatment of AIDS.
- Il-9 stimulates release of chemotactic factors from human bronchial epithelial cells.
- Activation of caspase-3 by interferon alpha causes interleukin-16 secretion but fails to modulate activation induced cell death.
- Interleukin-16 supports the migration of Langerhans cells, partly in a CD4-independent way.
- IL-16 inhibits IL-5 production by antigen-stimulated T cells in atopic subjects.
- Lipopolysaccharide binding protein potentiates airway reactivity in a murine model of allergic asthma.
- Desensitization of CXC chemokine receptor 4, mediated by IL-16/CD4, is independent of p56lck enzymatic activity.
- Nuclear translocation of the N-terminal prodomain of interleukin-16.
- B lymphocyte-derived IL-16 attracts dendritic cells and Th cells.
- Increased expression of IL-16 immunoreactivity in bronchial mucosa after segmental allergen challenge in patients with asthma.
- Interleukin 16 and T-cell chemoattractant activity in bronchoalveolar lavage 24 hours after allergen challenge in asthma.
- Immunosuppressive effect on T cell activation by interleukin- 16-cDNA-transfected human squamous cell line.
- Interleukin 16: implications for CD4 functions and HIV-1 progression.
- Role of IL-16 in delayed-type hypersensitivity reaction.
- Cultured human fibroblasts express constitutive IL-16 mRNA: cytokine induction of active IL-16 protein synthesis through a caspase-3-dependent mechanism.
- Role of B7-CD28/CTLA-4 costimulation and NF-kappa B in allergen-induced T cell chemotaxis by IL-16 and RANTES.
- Effect of interleukin-16-blocking peptide on parameters of allergic asthma in a murine model.
- Polarized type 1 cytokine profile in bronchoalveolar lavage T cells of patients with hypersensitivity pneumonitis.
- A role for IL-16 in the cross-talk between dendritic cells and T cells.
- Reciprocal desensitization of CCR5 and CD4 is mediated by IL-16 and macrophage-inflammatory protein-1 beta, respectively.
- Identification of domains in IL-16 critical for biological activity.
- Identification of a CD4 domain required for interleukin-16 binding and lymphocyte activation.
- Vbeta8+ T lymphocytes are essential in the regulation of airway hyperresponsiveness and bronchoalveolar eosinophilia but not in allergen-specific IgE in a murine model...
- Processing and release of IL-16 from CD4+ but not CD8+ T cells is activation dependent.
- Molecular cloning and expression of rhesus macaque and sooty mangabey interleukin 16: biologic activity and effect on simian immunodeficiency virus infection and/or re...
- Expression and in vitro evaluation of rhesus macaque wild type (wt) and modified CC chemokines.
- Tissue and T cell distribution of precursor and mature IL-16.
Schools
University Of Illinois College Of Medicine Chicago
Cook County Hospital
Doctors Specialties
- Internal Medicine
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- American Republic
- Bakery & Confectionary Union Plan
- Blue Card PPO
- Blue Advantage
- Blue Cross Blue Shield of Illinois HMO
- Blue Cross Blue Shield of Illinois PPO
- Blue Precision HMO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Cofinity PPO
- CoreSource
- Coventry Health Care of Illinois PPO
- Platinum PPO
- First Health (Coventry Health Care) PPO
- HealthLink PPO
- Choice Care PPO
- Preferred PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- National Elevator
- Pipefitters
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
- WellCare
Awards
- Diabetes Recognition Program
- CMS Meaningful Use Stage 1 Certification
Education
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John H. Stroger Hospital of Cook County -
University of Illinois College of Medicine
Hospital
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Advocate Christ Medical Center
Drug Facts
| NPI NUMBER | 1639104052 | |
| NPPES Provider LastName | CRUIKSHANK | |
| NPPES Provider FirstName | WILLIAM | |
| NPPES Provider ZIPCode | 604732003 | |
| NPPES Provider State | IL | |
| Specialty Description | Internal Medicine | |
| Total Claim Count | 12963.0 | |
| Distinct Opioid Count | 6.0 | |
| Opioid Claim Count | 504.0 | |
| Percent Opioid Claims | 3.89 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1639104052 |
| Last Name Of The Provider | CRUIKSHANK |
| First Name Of The Provider | WILLIAM |
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