Associate Professor, Pathology, University of Pittsburgh School of Medicine
Emergency Medicine
Dr. William C. Parks, MD
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William Parks is an expert in the field of Emergency Medicine. He graduated from University of Michigan Medical School. He received awards:"Fellow (FACEP)". Dr. William C. Parks is a published physician. He has 50 publications published. The most current publication "Syndecan-1 Attenuates Lung Injury During Influenza Infection By Potentiating c-Met Signaling to Suppress Epithelial Apoptosis."
Publications
- Syndecan-1 Attenuates Lung Injury During Influenza Infection By Potentiating c-Met Signaling to Suppress Epithelial Apoptosis.
- Filamentous Bacteriophage Promote Biofilm Assembly and Function.
- CD11b(+) Mononuclear Cells Mitigate Hyperoxia-induced Lung Injury in Neonatal Mice.
- A small intestinal organoid model of non-invasive enteric pathogen-epithelial cell interactions.
- Matrix remodeling by MMPs during wound repair.
- Mechanisms of tendon injury and repair.
- Amphiphilic polymer-coated CdSe/ZnS quantum dots induce pro-inflammatory cytokine expression in mouse lung epithelial cells and macrophages.
- Diverse functions of matrix metalloproteinases during fibrosis.
- Cdc42 Inhibits ERK-Mediated Collagenase-1 (MMP-1) Expression in Collagen-Activated Human Keratinocytes.
- Tissue inhibitor of metalloproteinases-3 moderates the proinflammatory status of macrophages.
- Changes in macrophage phenotype as the immune response evolves.
- MMP28 promotes macrophage polarization toward M2 cells and augments pulmonary fibrosis.
- Matrix metalloproteinase-7 coordinates airway epithelial injury response and differentiation of ciliated cells.
- Functional Interactions between Matrix Metalloproteinases and Glycosaminoglycans.
- Constitutive coupling of a naturally occurring human alpha1a-adrenergic receptor genetic variant to EGFR transactivation pathway.
- Epilysin (Matrix Metalloproteinase-28) Contributes to Airway Epithelial Cell Survival.
- Overexpression of urokinase by plaque macrophages causes histological features of plaque rupture and increases vascular matrix metalloproteinase activity in aged apoli...
- Kinetics of chemokine-glycosaminoglycan interactions control neutrophil migration into the airspaces of the lungs.
- Tissue inhibitor of metalloproteinases 3 regulates resolution of inflammation following acute lung injury.
- Mouse models of MMP and TIMP function.
- Role of matrix metalloproteinases in epithelial migration.
- Control of promatrilysin (MMP7) activation and substrate-specific activity by sulfated glycosaminoglycans.
- Syndecan-1 shedding facilitates the resolution of neutrophilic inflammation by removing sequestered CXC chemokines.
- MMP-9 sheds the beta2 integrin subunit (CD18) from macrophages.
- Differential Processing of {alpha}- and {beta}-Defensin Precursors by Matrix Metalloproteinase-7 (MMP-7).
- Epilysin (MMP-28) restrains early macrophage recruitment in Pseudomonas aeruginosa pneumonia.
- Cytosolic SYT/SS18 isoforms are actin-associated proteins that function in matrix-specific adhesion.
- MMP7 shedding of syndecan-1 facilitates re-epithelialization by affecting alpha(2)beta(1) integrin activation.
- Matrix metalloproteinase-7 (matrilysin) controls neutrophil egress by generating chemokine gradients.
- Erectile dysfunction in the type II diabetic db/db mouse: impaired venoocclusion with altered cavernosal vasoreactivity and matrix.
- Tissue inhibitor of metalloproteinase-1 moderates airway re-epithelialization by regulating matrilysin activity.
- Individual matrix metalloproteinases control distinct transcriptional responses in airway epithelial cells infected with Pseudomonas aeruginosa.
- What is the alpha2beta1 integrin doing in the epidermis?
- Matrix metalloproteinases promote inflammation and fibrosis in asbestos-induced lung injury in mice.
- Specific sequence motifs direct the oxygenation and chlorination of tryptophan by myeloperoxidase.
- NADPH oxidase restrains the matrix metalloproteinase activity of macrophages.
- Matrix metalloproteinase-9 modulation by resident arterial cells is responsible for injury-induced accelerated atherosclerotic plaque development in apolipoprotein E-d...
- Membrane-type 1 matrix metalloproteinase is required for normal alveolar development.
- Activities of the matrix metalloproteinase stromelysin-2 (MMP-10) in matrix degradation and keratinocyte organization in wounded skin.
- Beta3-integrin mediates smooth muscle cell accumulation in neointima after carotid ligation in mice.
- Oxidative cross-linking of tryptophan to glycine restrains matrix metalloproteinase activity: specific structural motifs control protein oxidation.
- Hypochlorous acid generated by myeloperoxidase modifies adjacent tryptophan and glycine residues in the catalytic domain of matrix metalloproteinase-7 (matrilysin): an...
- A model of primary atherosclerosis and post-angioplasty restenosis in mice.
- Matrilysin (matrix metalloproteinase-7) mediates E-cadherin ectodomain shedding in injured lung epithelium.
- Matrilysin shedding of syndecan-1 regulates chemokine mobilization and transepithelial efflux of neutrophils in acute lung injury.
- A confederacy of proteinases.
- Mouse strain modulates the role of the ciliated cell in acute tracheobronchial airway injury-distal airways.
- Nicotinamide exacerbates hypoxemia in ventilator-induced lung injury independent of neutrophil infiltration.
- Systemic sensitization with the protein allergen ovalbumin augments local sensitization in atopic dermatitis.
Schools
University of Mi Med Sch
Doctors Specialties
- Emergency Medicine
- Family Medicine
Accepted Insurances
- Aetna
- American Enterprise Group
- American Republic
- Assurant Health
- Great West Healthcare-Cigna PPO
- Commercial Insurance Company
- CoreSource
- Coventry Health Care
- Golden Rule
- HealthSmart
- Humana
- Medicare MCR
- Medico
- Molina
- Old Surety Life Insurance CompanyMedicare MCR
- POMCO Group
- United Healthcare
Awards
- Fellow (FACEP)
Education
-
University of Missouri-Columbia -
University of Michigan Medical School
Drug Facts
| NPI NUMBER | 1194720854 | |
| NPPES Provider LastName | PARKS | |
| NPPES Provider FirstName | WILLIAM | |
| NPPES Provider ZIPCode | 652015844 | |
| NPPES Provider State | MO | |
| Specialty Description | Emergency Medicine | |
| Total Claim Count | 318.0 | |
| Distinct Opioid Count | 2.0 | |
| Opioid Claim Count | 67.0 | |
| Percent Opioid Claims | 21.07 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1194720854 |
| Last Name Of The Provider | PARKS |
| First Name Of The Provider | WILLIAM |
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