Chief, Anatomic Pathology SFGH
Pathology
Dr. Walter E. Finkbeiner, MD
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Walter E. Finkbeiner, MD works in the field of Pathology. He graduated from University of Illinois College of Medicine at Chicago. University of Illinois College of Medicine at Chicago has a rank of 49 in research and a 85 in primary care Dr. Walter E. Finkbeiner is also a published MD. He has 25 paper published. The most recent paper is: Growth of alveoli during postnatal development in humans based on stereological estimation.
Publications
- Synergy-based small-molecule screen using a human lung epithelial cell line yields ΔF508-CFTR correctors that augment VX-809 maximal efficacy.
- Growth of alveoli during postnatal development in humans based on stereological estimation.
- Pivotal Role of MUC1 Glycosylation by Cigarette Smoke in Modulating Disruption of Airway Adherens Junctions In Vitro.
- Intelectin-1 is a prominent protein constituent of pathologic mucus associated with eosinophilic airway inflammation in asthma.
- Anchored PDE4 regulates chloride conductance in wild-type and ΔF508-CFTR human airway epithelia.
- Distribution and size of mucous glands in the ferret tracheobronchial tree.
- Rac1 and Cdc42 Differentially Modulate Cigarette Smoke-Induced Airway Cell Migration through p120-Catenin-Dependent and Independent Pathways.
- Calcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contraction.
- Airway Epithelial miRNA Expression is Altered in Asthma.
- Cigarette Smoke Disrupts the Integrity of Airway Adherens Junctions through the Aberrant Interaction of P120-catenin with the Cytoplasmic Tail of MUC1.
- Small-molecule activators of TMEM16A, a calcium-activated chloride channel, stimulate epithelial chloride secretion and intestinal contraction.
- CYSTIC FIBROSIS AND THE RELATIONSHIP BETWEEN MUCIN AND CHLORIDE SECRETION BY CULTURES OF HUMAN AIRWAY GLAND MUCOUS CELLS.
- Hyperviscous airway periciliary and mucous liquid layers in cystic fibrosis measured by confocal fluorescence photobleaching.
- Interleukin-13-induced mucous metaplasia increases susceptibility of human airway epithelium to rhinovirus infection.
- CFTR and calcium-activated chloride channels in primary cultures of human airway gland cells of serous or mucous phenotype.
- CFTR-adenylyl cyclase I association responsible for UTP activation of CFTR in well-differentiated primary human bronchial cell cultures.
- Airway surface liquid depth measured in ex vivo fragments of pig and human trachea: dependence on Na+ and Cl- channel function.
- In situ measurement of airway surface liquid [K+] using a ratioable K+-sensitive fluorescent dye.
- In vitro susceptibility to rhinovirus infection is greater for bronchial than for nasal airway epithelial cells in human subjects.
- Basolateral Cl channels in primary airway epithelial cultures.
- Hypertonic saline therapy in cystic fibrosis: Evidence against the proposed mechanism involving aquaporins.
- Expansion of cultures of human tracheal epithelium with maintenance of differentiated structure and function.
- Submucosal gland dysfunction as a primary defect in cystic fibrosis.
- Disseminated microsporidiosis in a pancreas/kidney transplant recipient.
Schools
University of Illinois At Chicago College of Medicine
University Ill Hospital
University Calif
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
- Basic HMO
- Vitalidad Plus California Con Aetna
Education
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University of Illinois College of Medicine at Chicago -
University of Illinois College of Medicine
Hospital
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UCSF Medical Center
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