Assistant Professor, Neurology, University of Michigan Medical School
Neurology
Dr. Vikram G. Shakkottai, MD
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Vikram Shakkottai practices Neurology. He attended Christian Medical College Vellore. He received awards:"CMS Meaningful Use Stage 1 Certification". Vikram Shakkottai is a published doctor. He published 24 publications, including: 'Precision medicine in spinocerebellar ataxias: treatment based on common mechanisms of disease.' He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Precision medicine in spinocerebellar ataxias: treatment based on common mechanisms of Disease.
- Neuronal Atrophy Early in Degenerative Ataxia Is a Compensatory Mechanism to Regulate Membrane Excitability.
- Clinical Evaluation of Eye Movements in Spinocerebellar Ataxias: A Prospective Multicenter Study.
- The Role for Alterations in Neuronal Activity in the Pathogenesis of Polyglutamine Repeat Disorders.
- Physiologic Changes Associated with Cerebellar Dystonia.
- Alterations in cerebellar physiology are associated with a stiff-legged Gait in Atcay(ji-hes) mice.
- Translating cerebellar Purkinje neuron physiology to progress in dominantly inherited Ataxia.
- Clinical neurogenetics: autosomal dominant spinocerebellar Ataxia.
- Toward RNAi Therapy for the Polyglutamine Disease Machado-Joseph Disease.
- Early changes in cerebellar physiology accompany motor dysfunction in the polyglutamine Disease spinocerebellar ataxia type 3.
- Temporal and cell-specific deletion establishes that neuronal Npc1 deficiency is sufficient to mediate neuroDegeneration.
- Physiologic alterations in Ataxia: channeling changes into novel therapies.
- FGF14 regulates the intrinsic excitability of cerebellar Purkinje neurons.
- SKCa channels mediate the medium but not the slow calcium-activated afterhyperpolarization in cortical neurons.
- SK3-1C, a dominant-negative suppressor of SKCa and IKCa channels.
- Enhanced neuronal excitability in the absence of neurodegeneration induces cerebellar Ataxia.
- Vascular risk factors and clinical progression in spinocerebellar Ataxias.
- Novel truncated isoform of SK3 potassium channel is a potent dominant-negative regulator of SK currents: implications in schizophrenia.
- Gramicidin S: a peptide model for protein glycation and reversal of glycation using nucleophilic amines.
- Design and characterization of a highly selective peptide inhibitor of the small conductance calcium-activated K+ channel, SkCa2.
- Genomic organization and promoter analysis of human KCNN3 gene.
- Conditional Niemann-Pick C mice demonstrate cell autonomous Purkinje cell neuroDegeneration.
- Modulators of small- and intermediate-conductance calcium-activated potassium channels and their therapeutic indications.
Procedures Preformed
- EEG (Electroencephalogram)
- EMG (Electromyography)
Conditions Treated
- Alzheimer's Disease
- Ataxia
- Autonomic Disorders
- Brain and Nervous System Cancer (incl. Gliomas, Astrocytoma, Schwannoma, Medulloblastoma, Chordoma)
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Washington University -
Christian Medical College Vellore
Hospital
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University of Michigan Hospitals and Health Centers
Drug Facts
| NPI NUMBER | 1033204615 | |
| NPPES Provider LastName | SHAKKOTTAI | |
| NPPES Provider FirstName | VIKRAM | |
| NPPES Provider ZIPCode | 481095322 | |
| NPPES Provider State | MI | |
| Specialty Description | Neurology | |
| Total Claim Count | 298.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1033204615 |
| Last Name Of The Provider | SHAKKOTTAI |
| First Name Of The Provider | VIKRAM |
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