Assistant Professor, Medicine, University of Washington School of Medicine
Infectious Disease
Dr. Tobias M. Hohl, MD
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Tobias Hohl works in the field of Infectious Disease. He went to medical school at Weill Cornell Medical College of Cornell University. Weill Cornell Medical College of Cornell University is ranked 18 in research and 49 in primary care medicine Tobias Hohl is a published doctor and has 41 publications published. The lastest was: 'Arrangement of subunits in 20 S particles consisting of NSF, SNAPs, and SNARE complexes.' He accepts all Medicare patients.
Publications
- GM-CSF signaling regulates neutrophil antifungal activity and the oxidative burst during respiratory fungal challenge.
- Zinc and Manganese Chelation by Neutrophil S100A8/A9 (Calprotectin) Limits Extracellular Aspergillus fumigatus Hyphal Growth and Corneal Infection.
- Compartment-specific and sequential role of MyD88 and CARD9 in chemokine induction and innate defense during respiratory fungal infection.
- Myeloid Derived Hypoxia Inducible Factor 1-alpha Is Required for Protection against Pulmonary Aspergillus fumigatus Infection.
- Nitric Oxide Regulates BAFF Expression and T Cell-Independent Antibody Responses.
- Independent contribution of bronchoalveolar lavage and serum galactomannan in the diagnosis of invasive pulmonary aspergillosis.
- Overview of vertebrate animal models of fungal infection.
- Inflammatory monocytes orchestrate innate antifungal immunity in the lung.
- Reply to King, et al.
- Resident Renal Mononuclear Phagocytes Comprise Five Discrete Populations with Distinct Phenotypes and Functions.
- Inflammatory monocytes mediate early and organ-specific innate defense during systemic candidiasis.
- Hypoxia enhances innate immune activation to Aspergillus fumigatus through cell wall modulation.
- Monocyte- and Macrophage-Targeted NADPH Oxidase Mediates Antifungal Host Defense and Regulation of Acute Inflammation in Mice.
- Genetic and Pharmacological Targeting of CSF-1/CSF-1R Inhibits Tumor-Associated Macrophages and Impairs BRAF-Induced Thyroid Cancer Progression.
- Tracing Conidial Fate and Measuring Host Cell Antifungal Activity Using a Reporter of Microbial Viability in the Lung.
- Monocytic CCR2+ Myeloid Derived Suppressor Cells Promote Immune Escape By Limiting Activated CD8 T Cell Infiltration Into The Tumor Microenvironment.
- iNKTs Foil Fungi.
- Ly6G+ Neutrophils Are Dispensable for Defense against Systemic Listeria monocytogenes Infection.
- In vivo Hypoxia and a Fungal Alcohol Dehydrogenase Influence the Pathogenesis of Invasive Pulmonary Aspergillosis.
- Immune responses against Aspergillus fumigatus: what have we learned?
- Bone Marrow Mesenchymal Stem and Progenitor Cells Induce Monocyte Emigration in Response to Circulating Toll-like Receptor Ligands.
- Dectin-1 diversifies Aspergillus fumigatus-specific T cell responses by inhibiting T helper type 1 CD4 T cell differentiation.
- Monocyte trafficking to hepatic sites of bacterial infection is chemokine independent and directed by focal intercellular adhesion molecule-1 expression.
- Inflammatory monocytes facilitate adaptive CD4 T cell responses during respiratory fungal infection.
- Essential role for neutrophils but not alveolar macrophages at early time points following Aspergillus fumigatus infection.
- Selective expansion of the monocytic lineage directed by bacterial infection.
- Stage-specific innate immune recognition of Aspergillus fumigatus and modulation by echinocandin drugs.
- Monocyte-mediated defense against microbial pathogens.
- Aspergillus fumigatus: principles of pathogenesis and host defense.
- Cracking the fungal armor.
- Distinct CD4+-T-cell responses to live and heat-inactivated Aspergillus fumigatus conidia.
- Aspergillus fumigatus triggers inflammatory responses by stage-specific beta-glucan display.
- Video-rate nonlinear microscopy of neuronal membrane dynamics with genetically encoded probes.
- In vivo bioluminescence imaging and histopathopathologic analysis reveal distinct roles for resident and recruited immune effector cells in defense against invasive as...
- A revised model for the oligomeric state of the N-ethylmaleimide-sensitive fusion protein, NSF.
- Arrangement of subunits in 20 S particles consisting of NSF, SNAPs, and SNARE complexes.
- Molecular mass, stoichiometry, and assembly of 20 S particles.
- Immunity to fungi.
- Caspofungin modulates inflammatory responses to Aspergillus fumigatus through stage-specific effects on fungal beta-glucan exposure.
- Immune responses to Aspergillus fumigatus infections.
Schools
Joan Sanford I Weill Medical College Of Cornell University
Ny Presbyterian Hospital
Meml Sloan Kettering Cancer Center
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Education
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Memorial Sloan-Kettering Cancer Center -
New York Presbyterian Hospital -
Weill Cornell Medical College of Cornell University
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1023143641 |
| Last Name Of The Provider | HOHL |
| First Name Of The Provider | TOBIAS |
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