Associate Professor of Surgery Director of Yale Endocrine Surgery Fellowship and Yale Endocrine Neoplasia Laboratory Department of Surgery Yale University School of Medicine Disease Team Leader (Endocrine Cancer) Smilow Cancer Hospital at Yale New
General Surgery
Dr. Tobias J. Carling, MD
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Tobias Carling works in the field of General Surgery. He received 2 awards: "CMS Meaningful Use Stage 1 Certification" and "Fellow (FACS)". Tobias J. Carling is a published doctor. He published 80 publications, including: 'Parathyroid MEN1 gene mutations in relation to clinical characteristics of nonfamilial primary hyperparathyroidism.' Tobias Carling is registered with Medicare.
Publications
- Neomorphic effects of recurrent somatic mutations in Yin Yang 1 in insulin-producing adenomas.
- Whole-exome sequencing characterizes the landscape of somatic mutations and copy number alterations in adrenocortical carcinoma.
- Frequent Silencing of RASSF1A via Promoter Methylation in Follicular Thyroid Hyperplasia: A Potential Early Epigenetic Susceptibility Event in Thyroid Carcinogenesis.
- Subclinical Cushing Syndrome: A Review.
- Surgery for Thyroid Cancer.
- Risk stratification in follicular neoplasm: A cytological assessment using the modified bethesda classification.
- Follicular Variant of Papillary Thyroid Carcinoma: Accuracy of FNA Diagnosis and Implications for Patient Management.
- Recurrent activating mutation in PRKACA in cortisol-producing adrenal tumors.
- Somatic and germline CACNA1D calcium channel mutations in aldosterone-producing adenomas and primary aldosteronism.
- Laryngeal physiology and voice acoustics are maintained after minimally invasive paraThyroidectomy.
- BRAFV600E mutation in papillary Thyroid microcarcinoma: a genotype-phenotype correlation.
- Risk of Secondary Cancer After Diagnosis of Thyroid Cancer Primary is Elevated in Microcarcinomas.
- Adrenocortical Carcinoma: Current Therapeutic State-of-the-Art.
- Surgical approach and outcomes in patients with lithium-associated hyperparaThyroidism.
- Vanishing Thyroid Tumors: A Diagnostic Dilemma Following Ultrasound Guided Fine Needle Aspiration.
- Identification of Somatic Mutations in ParaThyroid Tumors Using Whole-Exome Sequencing.
- The activating TERT promoter mutation C228T is recurrent in subsets of adrenal tumors.
- Epigenetic silencing of RASSF1A deregulates cytoskeleton and promotes malignant behavior of adrenocortical carcinoma.
- Hypermethylation in human Cancers of the RIZ1 tumor suppressor gene, a member of a histone/protein methyltransferase superfamily.
- Tumor formation and inactivation of RIZ1, an Rb-binding member of a nuclear protein-methyltransferase superfamily.
- [Primary hyperparaThyroidism is common among postmenopausal women. Identification of genetic risk factors can contribute to individualized treatment].
- Molecular pathology of paraThyroid tumors.
- Familial hypercalcemia and hypercalciuria caused by a novel mutation in the cytoplasmic tail of the calcium receptor.
- Reduced paraThyroid vitamin D receptor messenger ribonucleic acid levels in primary and secondary hyperparaThyroidism.
- Pathophysiology of primary hyperparaThyroidism.
- RAD51 as a candidate paraThyroid tumour suppressor gene on chromosome 15q: absence of somatic mutations.
- Genetic alterations on 3p, 11q13, and 18q in nonfamilial and MEN 1-associated pancreatic endocrine tumors.
- Analysis of the RAD54 gene on chromosome 1p as a potential tumor-suppressor gene in paraThyroid adenomas.
- The vitamin D receptor (VDR) start codon polymorphism in primary hyperparaThyroidism and paraThyroid VDR messenger ribonucleic acid levels.
- ParaThyroid MEN1 gene mutations in relation to clinical characteristics of nonfamilial primary hyperparaThyroidism.
- Vitamin D receptor (VDR) and paraThyroid hormone messenger ribonucleic acid levels correspond to polymorphic VDR alleles in human paraThyroid tumors.
- Endothelin-induced calcium signaling and secretion in chief cells and fibroblasts from pathological human paraThyroid glands.
- Vitamin D receptor gene polymorphism and paraThyroid calcium sensor protein (CAS/gp330) expression in primary hyperparaThyroidism.
- Regulation of gp330/megalin expression by vitamins A and D.
- Mutation of the multiple endocrine neoplasia type 1 gene in nonfamilial, malignant tumors of the endocrine pancreas.
- Estrogen receptor gene polymorphism in postmenopausal primary hyperparaThyroidism.
- Vitamin D receptor polymorphisms correlate to paraThyroid cell function in primary hyperparaThyroidism.
- Tissue distribution of human gp330/megalin, a putative Ca(2+)-sensing protein.
- Vitamin D receptor alleles b, a, and T: risk factors for sporadic primary hyperparaThyroidism (HPT) but not HPT of uremia or MEN 1.
- ParaThyroid surgery in familial hyperparaThyroid disorders.
- A deletion polymorphism in the RIZ gene, a female sex steroid hormone receptor coactivator, exhibits decreased response to estrogen in vitro and associates with low bo...
- Retroperitoneal endoscopic adrenalectomy is safe and effective (Br J Surg 2010; 97:1667-1672).
- The NeiI polymorphism in the cyclin D1 gene and sporadic primary hyperparaThyroidism.
- ParaThyroidectomy in familial hypercalcemia with clinical characteristics of primary hyperparaThyroidism and familial hypocalciuric hypercalcemia.
- Allelic loss in clinically and screening-detected primary hyperparaThyroidism.
- The tumor suppressor gene RIZ in Cancer gene therapy (review).
- Frequent Germ-Line Mutations of the MEN1, CASR, and HRPT2/CDC73 Genes in Young Patients with Clinically Non-familial Primary HyperparaThyroidism.
- Spontaneous adrenal hemorrhage with associated masses: etiology and management in 6 cases and a review of 133 reported cases.
- The role of epigenetic alterations in papillary Thyroid carcinogenesis.
- Minimally invasive paraThyroidectomy.
- The DNA methylome of benign and malignant paraThyroid tumors.
- Reflex BRAF testing in Thyroid fine-needle aspiration biopsy with equivocal and positive interpretation: a prospective study.
- K+ channel mutations in adrenal aldosterone-producing adenomas and hereditary hypertension.
- 4D paraThyroid CT as the initial localization study for patients with de novo primary hyperparaThyroidism.
- Clinical and histopathological characteristics of hyperparaThyroidism-induced hypercalcemic crisis.
- Expression and somatic mutations of SDHAF2 (SDH5), a novel endocrine tumor suppressor gene in paraThyroid tumors of primary hyperparaThyroidism.
- Insular variant of poorly differentiated Thyroid carcinoma.
- Controversy surrounding the role for routine central lymph node dissection for differentiated Thyroid cancer.
- Familial paraThyroid tumors: diagnosis and management.
- Molecular genetics of gastroenteropancreatic neuroendocrine tumors.
- Multinodular goiter and primary hyperparaThyroidism: a circuitous route to diagnosing metastatic uveal melanoma.
- Focused approach to paraThyroidectomy.
- Defining surgical therapy for pseudomembranous colitis with toxic megacolon.
- Special variants of differentiated Thyroid cancer: does it alter the extent of surgery versus well-differentiated Thyroid cancer?
- Diagnosis and treatment of interval sentinel lymph nodes in patients with cutaneous melanoma.
- Minimally invasive paraThyroidectomy using cervical block: reasons for conversion to general anesthesia.
- Follicular neoplasms of the Thyroid: what to recommend.
- Deletions and altered expression of the RIZ1 tumour suppressor gene in 1p36 in pheochromocytomas and abdominal paragangliomas.
- Multiple endocrine neoplasia syndrome: genetic basis for clinical management.
- A histone methyltransferase is required for maximal response to female sex hormones.
- Intragenic allelic loss and promoter hypermethylation of the RIZ1 tumor suppressor gene in paraThyroid tumors and pheochromocytomas.
- ParaThyroid tumors.
- In situ RNA-RNA hybridisation of phospholipase C beta 3 shows lack of expression in neuroendocrine tumours.
- Multiple endocrine neoplasia type 1 polymorphism D418D is associated with sporadic primary hyperparaThyroidism.
- Involvement of the MEN1 gene locus in familial isolated hyperparaThyroidism.
- Loss of heterozygosity in paraThyroid glands of familial hypercalcemia with hypercalciuria and point mutation in calcium receptor.
- 25-hydroxyvitamin D(3)-1alpha-hydroxylase expression in normal and pathological paraThyroid glands.
- Loss of heterozygosity on the short arm of chromosome 1 in pheochromocytoma and abdominal paraganglioma.
- Alterations of the SDHD gene locus in midgut carcinoids, Merkel cell carcinomas, pheochromocytomas, and abdominal paragangliomas.
Procedures Preformed
- Minimally Invasive Surgery
- Thyroid Surgery
Conditions Treated
- Thyroid Cancer
Doctors Specialties
- General Surgery
- Surgery
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACS)
Education
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Yale-New Haven Hospital -
Uppsala University Faculty of Medicine
Hospital
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Yale-New Haven Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1164604005 |
| Last Name Of The Provider | CARLING |
| First Name Of The Provider | TOBIAS |
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