Associate Professor, Internal Medicine, Washington University School of Medicine
Oncology
Dr. Timothy A. Graubert, MD
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Timothy Graubert is an expert in the field of Oncology. He went to Harvard Medical School. Harvard Medical School is a top medical college. Harvard Medical School is ranked 1/12 in Research/PrimaryCare. Timothy A. Graubert is a published MD. He has 68 publications published. The most current publication "How do lymphocytes kill tumor cells?" He is registered with Medicare and accepts Medicare payments.
Publications
- Rapid expansion of preexisting nonleukemic hematopoietic clones frequently follows induction therapy for de novo AML.
- Genomic analysis of germline and somatic variants in familial myelodysplasia/Acute myeloid leukemia.
- Quality of life and mood of patients and family caregivers during hospitalization for hematopoietic stem cell transplantation.
- Role of TP53 mutations in the origin and evolution of therapy-related Acute myeloid leukaemia.
- U2AF1 Mutations alter sequence specificity of pre-mRNA binding and splicing.
- AML Genomics: Introduction.
- A call to action for Acute lymphoblastic leukemia.
- Clonal architecture of secondary Acute myeloid leukemia defined by single-cell sequencing.
- Functional heterogeneity of genetically defined subclones in Acute myeloid leukemia.
- Integrated analysis of germline and somatic variants in ovarian Cancer.
- Clonal diversity of recurrently mutated genes in Myelodysplastic syndromes.
- Clonal evolution in relapsed Acute myeloid leukaemia revealed by whole-genome sequencing.
- Recurrent mutations in the U2AF1 splicing factor in Myelodysplastic syndromes.
- Genomics of Acute myeloid leukemia.
- Identification of a novel TP53 Cancer susceptibility mutation through whole-genome sequencing of a patient with therapy-related AML.
- Use of whole-genome sequencing to diagnose a cryptic fusion oncogene.
- Sequencing a mouse Acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
- DNMT3A mutations in Acute myeloid leukemia.
- Murine models of human Acute myeloid leukemia.
- Next-generation sequencing of Cancer genomes: back to the future.
- Recurring mutations found by sequencing an Acute myeloid leukemia genome.
- Acquired copy number alterations in adult Acute myeloid leukemia genomes.
- Genome-wide association study to identify novel loci associated with therapy-related myeloid Leukemia susceptibility.
- The impact of copy number variation on local gene expression in mouse hematopoietic stem and progenitor cells.
- Integrated genomic analysis implicates haploinsufficiency of multiple chromosome 5q31.2 genes in de novo Myelodysplastic syndromes pathogenesis.
- Quantitative trait loci associated with susceptibility to therapy-related Acute murine promyelocytic leukemia in hCG-PML/RARA transgenic mice.
- Identification of somatic JAK1 mutations in patients with Acute myeloid leukemia.
- Somatic mutations and germline sequence variants in the expressed tyrosine kinase genes of patients with de novo Acute myeloid leukemia.
- Distinct patterns of mutations occurring in de novo AML versus AML arising in the setting of severe congenital Neutropenia.
- A high-resolution map of segmental DNA copy number variation in the mouse genome.
- Identification of candidate alkylator-induced Cancer susceptibility genes by whole genome scanning in mice.
- Dominant negative effects of the AML1/ETO fusion oncoprotein.
- Stem cell expression of the AML1/ETO fusion protein induces a Myeloproliferative disorder in mice.
- Recruitment of bone marrow-derived endothelial cells to sites of pancreatic beta-cell injury.
- A pilot study of high-throughput, sequence-based mutational profiling of primary human Acute myeloid leukemia cell genomes.
- A mouse-based strategy for cyclophosphamide pharmacogenomic discovery.
- Sca-1(pos) cells in the mouse mammary gland represent an enriched progenitor cell population.
- SWR/J mice are susceptible to alkylator-induced myeloid Leukemia.
- Reduced risk of Acute GVHD following mobilization of HLA-identical sibling donors with GM-CSF alone.
- Supraventricular tachyarrhythmias after hematopoietic stem cell transplantation: incidence, risk factors and outcomes.
- Cloning and expression of the cDNA encoding mouse neutrophil gelatinase: demonstration of coordinate secondary granule protein gene expression during terminal neutroph...
- Granulocyte colony-stimulating factor induction of normal human bone marrow progenitors results in neutrophil-specific gene expression.
- Characterization of Ly-6M, a novel member of the Ly-6 family of hematopoietic proteins.
- Granzyme A initiates an alternative pathway for granule-mediated apoptosis.
- How do lymphocytes kill tumor cells?
- How do cytotoxic lymphocytes kill their targets?
- Stochastic, stage-specific mechanisms account for the variegation of a human globin transgene.
- Perforin/granzyme-dependent and independent mechanisms are both important for the development of Graft-versus-host disease after murine bone marrow transplantation.
- The role of granzyme B in murine models of Acute graft-versus-host disease and graft rejection.
- Recombinant retroviral systems for the analysis of drug resistant HIV.
- Enhanced green fluorescent protein targeted to the Sca-1 (Ly-6A) locus in transgenic mice results in efficient marking of hematopoietic stem cells in vivo.
- Long-term outcomes of allogeneic stem cell transplant recipients after calcineurin inhibitor-induced neurotoxicity.
- Roles of Sca-1 in hematopoietic stem/progenitor cell function.
- Sca-1 negatively regulates proliferation and differentiation of muscle cells.
- Does autologous transplantation directly increase the risk of secondary Leukemia in lymphoma patients?
- Pharmacogenetics of alkylator-associated Acute myeloid leukemia.
- A randomized double-blind trial of hydroxychloroquine for the prevention of Chronic graft-versus-host disease after allogeneic peripheral blood stem cell transplantation.
- AML1 and Evi1: coconspirators in MDS/AML?
- wuHMM: a robust algorithm to detect DNA copy number variation using long oligonucleotide microarray data.
- Therapy related Acute myeloid leukemia in breast cancer survivors, a population-based study.
- DNA sequencing of a cytogenetically normal Acute myeloid leukaemia genome.
- Therapy-related Myelodysplastic syndrome: models and genetics.
- Structural variation of the mouse natural killer gene complex.
- POU4F1 is associated with t(8;21) Acute myeloid leukemia and contributes directly to its unique transcriptional signature.
- Genetics of Myelodysplastic syndromes: new insights.
- Recurrent DNMT3A mutations in patients with Myelodysplastic syndromes.
- Integrated genomics of susceptibility to alkylator-induced Leukemia in mice.
Schools
Harvard Medical School
Yale New Haven Hospital
Barnes Jewish Hospital S Campus
Procedures Preformed
- HPC Transplantation (Stem Cell Transplant)
- Plasmapheresis
- Thoracentesis
Conditions Treated
- Acute Leukemia
- Anemia
- Chronic Myeloid Leukemia (CML)
- Colorectal Cancer
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Doctors Specialties
- Medical Oncology
- Oncology
Accepted Insurances
Education
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Washington University - Barnes-Jewish Hospital -
Yale-New Haven Hospital -
Harvard Medical School
Hospital
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Massachusetts General Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1073531703 |
| Last Name Of The Provider | GRAUBERT |
| First Name Of The Provider | TIMOTHY |
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