Physician
Family Medicine
Dr. Thomas M. McIntyre, MD
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Thomas McIntyre works in the field of Family Medicine. He attended University of Mississippi School of Medicine. He received 1 awards: "Meaningful Use Stage 1 Certification". Thomas M. McIntyre, MD has written publications. He has 58 publications published with PubMed. The lastest publication is: 'Sonic hedgehog signaling in hepatocellular carcinoma: A pilot study.'
Publications
- Apolipoprotein A1 regulates coenzyme q10 absorption, mitochondrial function, and infarct size in a mouse model of myocardial infarction.
- Chronic ethanol ingestion induces oxidative kidney injury through taurine-inhibitable inflammation.
- OxNASH Score Correlates with Histologic Features and Severity of Nonalcoholic Fatty Liver Disease.
- Aspirin Hydrolysis in Plasma Is a Variable Function of Butyrylcholinesterase and Platelet-activating Factor Acetylhydrolase 1b2 (PAFAH1b2).
- Translational control of JunB, an AP-1 transcription factor, in activated human endothelial cells.
- Intracellular Erythrocyte PAF Acetylhydrolase 1 Inactivates Aspirin in Blood.
- Lipopolysaccharide Signaling without a Nucleus: Kinase Cascades Stimulate Platelet Shedding of Proinflammatory IL-1{beta}-Rich Microparticles.
- Human TMEM30a promotes uptake of antitumor and bioactive choline phospholipids into mammalian cells.
- Circulating platelet-activating factor is primarily cleared by transport, not intravascular hydrolysis by lipoprotein-associated phospholipase A2/ PAF acetylhydrolase.
- Lipid oxidation and cardiovascular disease: introduction to a review series.
- Mass spectrometric profiling of oxidized lipid products in human nonalcoholic fatty liver disease and nonalcoholic steatohepatitis.
- Chronic alcohol exposure increases circulating bioactive oxidized phospholipids.
- An antimicrobial peptide regulates tumor-associated macrophage trafficking via the chemokine receptor CCR2, a model for tumorigenesis.
- Suppression of mitochondrial function by oxidatively truncated phospholipids is reversible, aided by bid, and suppressed by Bcl-XL.
- PAF-acetylhydrolase expressed during megakaryocyte differentiation inactivates PAF-like lipids.
- The emerging roles of PAF acetylhydrolase.
- Platelet activation by low concentrations of intact oxidized LDL particles involves the PAF receptor.
- Hepatic lipid partitioning and liver damage in nonalcoholic fatty liver disease: role of stearoyl-CoA desaturase.
- Lipopolysaccharide is a direct agonist for platelet RNA splicing.
- A yeast PAF acetylhydrolase ortholog suppresses oxidative death.
- The lysosomal-mitochondrial axis in free fatty acid-induced hepatic lipotoxicity.
- Integrin alphaDbeta2 is dynamically expressed by inflamed macrophages and alters the natural history of lethal systemic infections.
- Intracellular PAF catabolism by PAF acetylhydrolase counteracts continual PAF synthesis.
- Cytotoxic phospholipid oxidation products. Cell death from mitochondrial damage and the intrinsic caspase cascade.
- mTOR-dependent synthesis of Bcl-3 controls the retraction of fibrin clots by activated human platelets.
- Phospholipase action of platelet-activating factor acetylhydrolase, but not paraoxonase-1, on long fatty acyl chain phospholipid hydroperoxides.
- Release of free F2-isoprostanes from esterified phospholipids is catalyzed by intracellular and plasma platelet-activating factor acetylhydrolases.
- Impairment of endothelium-dependent aorta relaxation by phospholipid components of oxidized low-density lipoprotein.
- Ultraviolet B radiation generates platelet-activating factor-like phospholipids underlying cutaneous damage.
- Escaping the nuclear confines: signal-dependent pre-mRNA splicing in anucleate platelets.
- Endothelial cell confluence regulates cyclooxygenase-2 and prostaglandin E2 production that modulate motility.
- Activated polymorphonuclear leukocytes rapidly synthesize retinoic acid receptor-alpha: a mechanism for translational control of transcriptional events.
- Neutrophils alter the inflammatory milieu by signal-dependent translation of constitutive messenger RNAs.
- Lysophosphatidic acid induces neointima formation through PPARgamma activation.
- Synthesis of migration-resistant hydroxyethoxy analogues of lysophosphatidic acid.
- Endotoxin contamination of ovalbumin suppresses murine immunologic responses and development of airway hyper-reactivity.
- Endotoxins stimulate neutrophil adhesion followed by synthesis and release of platelet-activating factor in microparticles.
- Synergism between platelet-activating factor-like phospholipids and peroxisome proliferator-activated receptor gamma agonists generated during low density lipoprotein ...
- Low-density lipoprotein in hypercholesterolemic human plasma induces vascular endothelial cell apoptosis by inhibiting fibroblast growth factor 2 transcription.
- Cell-cell interactions: leukocyte-endothelial interactions.
- Platelet-activating factor acetylhydrolase is increased in lung lavage fluid from patients with acute respiratory distress syndrome.
- Platelet-activating factor acetylhydrolase, and not paraoxonase-1, is the oxidized phospholipid hydrolase of high density lipoprotein particles.
- Identification of an intracellular receptor for lysophosphatidic acid (LPA): LPA is a transcellular PPARgamma agonist.
- Fluid flow regulates E-selectin protein levels in human endothelial cells by inhibiting translation.
- Platelet-activating factor, a pleiotrophic mediator of physiological and pathological processes.
- Temporal correlation of measurements of airway hyperresponsiveness in ovalbumin-sensitized mice.
- Outside-in signals delivered by matrix metalloproteinase-1 regulate platelet function.
- The platelet-activating factor signaling system and its regulators in syndromes of inflammation and thrombosis.
- Sol Sherry lecture in thrombosis: molecular events in acute inflammation.
- Monocyte chemoattractant protein-1 and 5-lipoxygenase products recruit leukocytes in response to platelet-activating factor-like lipids in oxidized low-density lipopro...
- Cyclooxygenase-2 is induced in monocytes by peroxisome proliferator activated receptor gamma and oxidized alkyl phospholipids from oxidized low density lipoprotein.
- Activation of vascular cells by PAF-like lipids in oxidized LDL.
- Retinoic acid-inducible gene-I is induced in endothelial cells by LPS and regulates expression of COX-2.
- Ceramide generation in situ alters leukocyte cytoskeletal organization and beta 2-integrin function and causes complete degranulation.
- Leukocyte adhesion deficiency syndromes: adhesion and tethering defects involving beta 2 integrins and selectin ligands.
- Sonic hedgehog signaling in hepatocellular carcinoma: A pilot study.
- Gut Microbial Metabolite TMAO Enhances Platelet Hyperreactivity and Thrombosis Risk.
Schools
Univ Of Ms Sch Of Med
Doctors Specialties
- Family Medicine
Accepted Insurances
- Choice Plus POS II
- Blue Card PPO
- Dental Blue 100/200/300
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Awards
- Meaningful Use Stage 1 Certification
Education
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University of Mississippi School of Medicine
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