Professor, Neurology-Beth Israel Deaconess Medical Center, Harvard Medical School
Neurology
Dr. Thomas E. Scammell, MD
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Thomas E. Scammell practices Neurology. He went to medical school at University of Massachusetts Medical School. University of Massachusetts Medical School is ranked 49/12 in Research/PrimaryCare. He over the years received 4 awards: "Sleep Science Award", "Researcher of the Year Award", "Alpha Omega Alpha" and "CSB Distinguished Visiting Neuroscientist". Dr. Thomas E. Scammell is a published MD as well. He has 87 publications published. The latest was: Activation of ventrolateral preoptic neurons by the somnogen prostaglandin D2. He accepts Medicare.
Publications
- Dynorphin inhibits basal forebrain cholinergic neurons by pre- and postsynaptic mechanisms.
- The roles of prostaglandin E2 and D2 in lipopolysaccharide-mediated changes in Sleep.
- Carbachol excites sublaterodorsal nucleus neurons projecting to the spinal cord.
- Optogenetic-mediated release of histamine reveals distal and autoregulatory mechanisms for controlling arousal.
- Emerging therapeutics in Sleep.
- Control of arousal by the orexin neurons.
- Narcolepsy: neural mechanisms of sleepiness and cataplexy.
- Neural circuitry engaged by prostaglandins during the sickness Syndrome.
- Coexistence of Narcolepsy and Alzheimer's disease.
- Orexin receptor 2 expression in the posterior hypothalamus rescues Sleepiness in narcoleptic mice.
- Orexin receptors: pharmacology and therapeutic opportunities.
- Sleep neurobiology from a clinical perspective.
- Sleep state switching.
- The trouble with Tribbles: do antibodies against TRIB2 cause Narcolepsy?
- Abnormal Sleep/wake dynamics in orexin knockout mice.
- Loss of hypocretin (orexin) neurons with traumatic brain injury.
- Orexin neurons are necessary for the circadian control of REM Sleep.
- Is low histamine a fundamental cause of sleepiness in Narcolepsy and idiopathic hypersomnia?
- Running promotes wakefulness and increases Cataplexy in orexin knockout mice.
- Mathematical model of network dynamics governing mouse Sleep-wake behavior.
- Orexins: looking forward to Sleep, back at addiction.
- Elevated body temperature during Sleep in orexin knockout mice.
- The frustrating and mostly fruitless search for an autoimmune cause of Narcolepsy.
- Diurnal variation in CSF orexin-A in healthy male subjects.
- Afferents to the orexin neurons of the rat brain.
- Homeostatic, circadian, and emotional regulation of Sleep.
- Orexin, drugs and motivated behaviors.
- Thalamic Cav3.1 T-type Ca2+ channel plays a crucial role in stabilizing Sleep.
- Common scale-invariant patterns of Sleep-wake transitions across mammalian species.
- Behavioral state instability in orexin knock-out mice.
- Mistaken diagnosis of psychogenic gait disorder in a man with status cataplecticus ("limp man Syndrome").
- Sleep neurobiology for the clinician.
- Narcolepsy caused by acute disseminated encephalomyelitis.
- Modafinil: a drug in search of a mechanism.
- Critical role of dorsomedial hypothalamic nucleus in a wide range of behavioral circadian rhythms.
- Focal deletion of the adenosine A1 receptor in adult mice using an adeno-associated viral vector.
- The neurobiology, diagnosis, and treatment of Narcolepsy.
- Afferents to the ventrolateral preoptic nucleus.
- Orexin gene therapy restores the timing and maintenance of wakefulness in narcoleptic mice.
- Delusional confusion of dreaming and reality in Narcolepsy.
- A consensus definition of cataplexy in mouse models of Narcolepsy.
- Hcrtr1 and 2 signaling differentially regulates depression-like behaviors.
- Reward-seeking behavior in human Narcolepsy.
- Do enteric neurons make hypocretin?
- Wake-related activity of tuberomammillary neurons in rats.
- Hypothalamic regulation of Sleep and circadian rhythms.
- Parkinson''s disease, Sleepiness and hypocretin/orexin.
- Pharmacotherapy for Cataplexy.
- Regulation of daily locomotor activity and Sleep by hypothalamic EGF receptor signalling.
- Orexin/Hypocretin: wired for wakefulness.
- Hypocretin/orexin and sleep: implications for the pathophysiology and diagnosis of Narcolepsy.
- Effects of adenosine on gabaergic synaptic inputs to identified ventrolateral preoptic neurons.
- Orexin-immunoreactive inputs to rat sympathetic preganglionic neurons.
- Deletion of presynaptic adenosine A1 receptors impairs the recovery of synaptic transmission after hypoxia.
- Modafinil: a novel stimulant for the treatment of Narcolepsy.
- Modafinil more effectively induces wakefulness in orexin-null mice than in wild-type littermates.
- No evidence for a circadian rhythm of protein synthesis in the rat suprachiasmatic nuclei.
- Threat of nuclear war.
- Hyperthermia induces c-fos expression in the preoptic area.
- Inhibition of nitric oxide synthase produces hypothermia and depresses lipopolysaccharide fever.
- Ventromedial preoptic prostaglandin E2 activates fever-producing autonomic pathways.
- Distribution of Fos-like immunoreactivity in the rat brain following intravenous lipopolysaccharide administration.
- Intravenous lipopolysaccharide induces cyclooxygenase 2-like immunoreactivity in rat brain perivascular microglia and meningeal macrophages.
- Mechanisms of CNS response to systemic immune challenge: the febrile response.
- Microinjection of a cyclooxygenase inhibitor into the anteroventral preoptic region attenuates LPS fever.
- Activation of ventrolateral preoptic neurons by the somnogen prostaglandin D2.
- Narcolepsy in orexin knockout mice: molecular genetics of sleep regulation.
- Silent functional magnetic resonance imaging demonstrates focal activation in rapid eye movement Sleep.
- Cerebral deep venous thrombosis presenting as acute micrographia and hypophonia.
- Relationship of EP(1-4) prostaglandin receptors with rat hypothalamic cell groups involved in lipopolysaccharide fever responses.
- Hypothalamic arousal regions are activated during modafinil-induced wakefulness.
- Severe acute disseminated encephalomyelitis with normal MRI at presentation.
- Fos expression in orexin neurons varies with behavioral state.
- Narcolepsy and low CSF orexin (hypocretin) concentration after a diencephalic stroke.
- Orexin (hypocretin) neurons contain dynorphin.
- Wakefulness: an eye-opening perspective on orexin neurons.
- Effects of vagus nerve stimulation on respiration during Sleep.
- Melanopsin in cells of origin of the retinohypothalamic tract.
- The Sleep switch: hypothalamic control of Sleep and wakefulness.
- An adenosine A2a agonist increases Sleep and induces Fos in ventrolateral preoptic neurons.
- Polymorphisms in hypocretin/orexin pathway genes and Narcolepsy.
- Regulation of daily locomotor activity and Sleep by hypothalamic EGF receptor signaling.
- Concomitant loss of dynorphin, NARP, and orexin in Narcolepsy.
- Delayed orexin signaling consolidates wakefulness and Sleep: physiology and modeling.
- Feeding-elicited Cataplexy in orexin knockout mice.
- Activation of the basal forebrain by the orexin/hypocretin neurones.
Schools
University Of Massachusetts Medical School
University Mass Med Center
Uc San Francisco
Beth Israel Hospital
Conditions Treated
- Ataxia
- Cerebral Artery Thrombosis
- Convulsions
- Epilepsy
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Doctors Specialties
- Sleep Medicine
- Neurology
Accepted Insurances
Awards
- Sleep Science Award
- Researcher of the Year Award
- Alpha Omega Alpha
- CSB Distinguished Visiting Neuroscientist
Education
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UCSF Medical Center -
University of Massachusetts -
University of Massachusetts Medical School
Hospital
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Beth Israel Deaconess Medical Center
Drug Facts
| NPI NUMBER | 1194762724 | |
| NPPES Provider LastName | SCAMMELL | |
| NPPES Provider FirstName | THOMAS | |
| NPPES Provider ZIPCode | 022155395 | |
| NPPES Provider State | MA | |
| Specialty Description | Neurology | |
| Total Claim Count | 112.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1194762724 |
| Last Name Of The Provider | SCAMMELL |
| First Name Of The Provider | THOMAS |
| View All |
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