Professor, Medicine, University of Alabama School of Medicine
Cardiology
Dr. Steven M. Pogwizd, MD
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Steven Pogwizd works in the field of Cardiology. He went to medical school at Washington University School of Medicine in St. Louis. He has 1 awards "Meaningful Use Stage 1 Certification". Steven Pogwizd is a published doctor and has 70 publications published. The lastest was: 'Mechanisms underlying spontaneous and induced ventricular arrhythmias in patients with idiopathic dilated cardiomyopathy.' Steven Pogwizd is registered with Medicare.
Publications
- Mitochondria-derived ROS Bursts Disturb Calcium Cycling and Induce Abnormal Automaticity in Guinea Pig Cardiomyocyte: a Theoretical Study.
- Imaging Cardiac Activation Sequence during Ventricular Tachycardia in a Canine Model of Nonischemic Heart Failure.
- Long Duration Ventricular Fibrillation Exhibits Two Distinct Organized States.
- Noninvasive cardiac activation imaging of ventricular arrhythmias during drug-induced QT prolongation in the rabbit Heart.
- Sex differences in repolarization and slow delayed rectifier potassium current and their regulation by sympathetic stimulation in rabbits.
- Focal Initiation of Sustained and Nonsustained Ventricular Tachycardia in a Canine Model of Ischemic Cardiomyopathy.
- Noninvasive Reconstruction of the Three-dimensional Ventricular Activation Sequence during Pacing and Ventricular Tachycardia in the Canine Heart.
- Noninvasive reconstruction of the three-dimensional ventricular activation sequence during pacing and ventricular tachycardia in the rabbit Heart.
- Enhanced activation of p21-activated kinase 1 in Heart failure contributes to dephosphorylation of connexin 43.
- Noninvasive Imaging of Three-dimensional Cardiac Activation Sequence during Pacing and Ventricular Tachycardia.
- Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and Heart failure.
- Activation becomes highly organized during long-duration ventricular fibrillation in canine Hearts.
- Can mapping differentiate microreentry from a focus in the ventricle?
- Arrhythmogenic effects of beta2-adrenergic stimulation in the failing Heart are attributable to enhanced sarcoplasmic reticulum Ca load.
- Mechanistic insights into ventricular Arrhythmias from mapping studies in humans.
- Ca2+/calmodulin-dependent protein kinase IIdelta and protein kinase D overexpression reinforce the histone deacetylase 5 redistribution in Heart failure.
- Intra-sarcoplasmic reticulum free [Ca2+] and buffering in arrhythmogenic failing rabbit Heart.
- Creating a cardiac pacemaker by gene therapy.
- Three-dimensional Myocardial activation imaging in a rabbit model.
- Ca2+/calmodulin-dependent protein kinase modulates cardiac ryanodine receptor phosphorylation and sarcoplasmic reticulum Ca2+ leak in Heart failure.
- Noninvasive three-dimensional electrocardiographic imaging of ventricular activation sequence.
- Expression and phosphorylation of the na-pump regulatory subunit phospholemman in Heart failure.
- Adenoviral gene transfer of mutant phospholamban rescues contractile dysfunction in failing rabbit myocytes with relatively preserved SERCA function.
- Connexin 43 downregulation and dephosphorylation in nonischemic Heart failure is associated with enhanced colocalized protein phosphatase type 2A.
- Cellular basis of triggered arrhythmias in Heart failure.
- Gene therapy to develop a genetically engineered cardiac pacemaker.
- Elevated sarcoplasmic reticulum Ca2+ leak in intact ventricular myocytes from rabbits in Heart failure.
- Prevention of ischemia-induced reentrant ventricular Arrhythmias by a peptide that enhances gap junctional conductance.
- Na/Ca exchange in Heart failure: contractile dysfunction and arrhythmogenesis.
- Intracellular Na(+) concentration is elevated in Heart failure but Na/K pump function is unchanged.
- Increased expression of alternatively spliced dominant-negative isoform of SRF in human failing Hearts.
- Calcium cycling in Heart failure: the arrhythmia connection.
- Morning surge of ventricular arrhythmias in a new arrhythmogenic canine model of chronic Heart failure is associated with attenuation of time-of-day dependence of hear...
- Inhibiting na+/k+ ATPase can impair mitochondrial energetics and induce abnormal Ca2+ cycling and automaticity in Guinea pig cardiomyocytes.
- Spontaneous Ca waves in ventricular myocytes from failing Hearts depend on Ca(2+)-calmodulin-dependent protein kinase II.
- Arrhythmogenesis and contractile dysfunction in Heart failure: Roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic respo...
- Increased Na(+)-Ca(2+) exchanger in the failing Heart.
- Cycle length dynamics during ventricular tachycardia in the infarcted Heart.
- Upregulation of Na(+)/Ca(2+) exchanger expression and function in an arrhythmogenic rabbit model of Heart failure.
- Mechanisms underlying spontaneous and induced ventricular Arrhythmias in patients with idiopathic dilated cardiomyopathy.
- TransMyocardial laser treatment denervates canine myocardium.
- Termination of ventricular tachycardia in the human Heart. Insights from three-dimensional mapping of nonsustained and sustained ventricular tachycardias.
- Three-dimensional mapping of the initiation of nonsustained ventricular tachycardia in the human Heart.
- Biochemical and electrophysiological alterations underlying ventricular arrhythmias in the failing Heart.
- Nonreentrant mechanisms underlying spontaneous ventricular arrhythmias in a model of nonischemic Heart failure in rabbits.
- Focal mechanisms underlying ventricular tachycardia during prolonged ischemic Cardiomyopathy.
- Influences of labetalol a combined alpha- and beta-adrenergic blocking agent on the dysrhythmias induced by Coronary occlusion and reperfusion.
- Induction of delayed afterdepolarizations and triggered activity in canine Purkinje fibers by lysophosphoglycerides.
- Mechanisms of arrhythmogenesis during Myocardial ischemia and reperfusion: a perspective of our current understanding.
- Reentrant and nonreentrant mechanisms contribute to arrhythmogenesis during early Myocardial ischemia: results using three-dimensional mapping.
- Electrophysiologic mechanisms underlying Arrhythmias due to reperfusion of ischemic myocardium.
- Mechanisms contributing to arrhythmogenesis during early Myocardial ischemia, subsequent reperfusion, and chronic infarction.
- Mechanisms underlying the development of ventricular Fibrillation during early myocardial ischemia.
- Mechanisms underlying ventricular tachycardia and fibrillation in the ischemic Heart: relation to nonlinear dynamics.
- Amphipathic lipid metabolites and their relation to arrhythmogenesis in the ischemic Heart.
- Contribution of myocardium responsible for ventricular tachycardia to abnormalities detected by analysis of signal-averaged ECGs.
- Reentrant and focal mechanisms underlying ventricular tachycardia in the human Heart.
- The contribution of nonreentrant mechanisms to malignant ventricular Arrhythmias.
- Micropatterns of propagation.
- Intracellular [Na+] and Na+ pump rate in rat and rabbit ventricular myocytes.
- Upregulated Na/Ca exchange is involved in both contractile dysfunction and arrhythmogenesis in Heart failure.
- Three-dimensional activation sequence imaging in a rabbit model.
- Iatrogenic hemolytic anemia and endocarditis in New Zealand white rabbits secondary to Achromobacter xylosoxidans infection.
- Purkinje fibers and Arrhythmias.
- Noninvasive three-dimensional cardiac activation imaging from body surface potential maps: a computational and experimental study on a rabbit model.
- Intracellular Na in animal models of hypertrophy and Heart failure: contractile function and arrhythmogenesis.
- Clinical potential of sodium-calcium exchanger inhibitors as antiarrhythmic agents.
- Noninvasive three-dimensional cardiac activation imaging on a rabbit model.
- Connexin43 knockdown or overexpression modulates cell coupling in control and failing rabbit left ventricular myocytes.
Schools
Washington University St Louis
Michael Reese Hospital and Med Center
Barnes Jewish Hospital
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
Conditions Treated
- Aneurysm and Dissection of Heart
- Angina and Acute Coronary Syndrome
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
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Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Meaningful Use Stage 1 Certification
Education
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Washington University - Barnes-Jewish Hospital -
Washington University School of Medicine in St. Louis
Hospital
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University of Alabama Hospital
Drug Facts
| NPI NUMBER | 1730293093 | |
| NPPES Provider LastName | POGWIZD | |
| NPPES Provider FirstName | STEVEN | |
| NPPES Provider ZIPCode | 352940001 | |
| NPPES Provider State | AL | |
| Specialty Description | Cardiology | |
| Total Claim Count | 571.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1730293093 |
| Last Name Of The Provider | POGWIZD |
| First Name Of The Provider | STEVEN |
| View All |
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