Professor of Medicine, Joslin Diabetes Center
Endocrinology
Dr. Steven E. Shoelson, MD
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Steven E. Shoelson, MD is an expert in the field of Endocrinology. He went to medical school at University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine has a rank of 10 in research and a 19 in primary care Dr. Steven E. Shoelson is a published doctor and has 44 publications published. The lastest was: 'Effects of the anti-inflammatory drug salsalate on bone turnover in type 2 diabetes mellitus.'
Publications
- Effects of the anti-inflammatory drug salsalate on bone turnover in type 2 diabetes mellitus.
- Insulin Receptor Activation with Transmembrane Domain Ligands.
- The Impact of Salsalate Treatment on Serum Levels of Advanced Glycation End Products in Type 2 Diabetes.
- Targeting Inflammation Using Salsalate in Patients With Type 2 Diabetes (TINSAL): Effects on flow-mediated dilation.
- Salicylate (salsalate) in patients with type 2 diabetes: a randomized trial.
- Profilin-1 haploinsufficiency protects against obesity-associated glucose intolerance and preserves adipose tissue immune homeostasis.
- A randomised trial of salsalate for insulin resistance and cardiovascular risk factors in persons with abnormal glucose tolerance.
- PPAR-γ is a major driver of the accumulation and phenotype of adipose tissue Treg cells.
- NF-{kappa}B ACTIVATION AND POLY-UBIQUITIN CONJUGATION ARE REQUIRED FOR PULMONARY INFLAMMATION INDUCED DIAPHRAGM ATROPHY.
- ASPIRIN REDUCES HYPERTRIGLYCERIDEMIA BY LOWERING VLDL-TRIGLYCERIDE PRODUCTION IN MICE FED A HIGH-FAT DIET.
- Hepatocyte-specific IKK? expression aggravates atherosclerosis development in APOE*3-Leiden mice.
- Crystal Structures of Human TBC1D1 and TBC1D4 (AS160) RabGTPase-activating Protein (RabGAP) Domains Reveal Critical Elements for GLUT4 Translocation.
- Hepatocyte-specific IKK-{beta} activation enhances VLDL-triglyceride production in APOE*3-Leiden mice.
- Therapeutic approaches to target inflammation in type 2 diabetes.
- Inflammation and adipose tissue macrophages in lipodystrophic mice.
- Multiple binding modes between HNF4alpha and the LXXLL motifs of PGC-1alpha lead to full activation.
- Expansion and contraction: treating diabetes with bariatric surgery.
- Getting away from glucose: fanning the flames of obesity-induced inflammation.
- Aire employs a histone-binding module to mediate immunological tolerance, linking chromatin regulation with organ-specific autoimmunity.
- Use of salsalate to target inflammation in the treatment of insulin resistance and type 2 diabetes.
- When a domain is not a domain.
- Salsalate improves glycemia and inflammatory parameters in obese young adults.
- Obesity, inflammation, and insulin resistance.
- Tag polymorphisms at the A20 (TNFAIP3) locus are associated with lower gene expression and increased risk of coronary artery disease in type 2 diabetes.
- Point-counterpoint: Interleukin-6 does/does not have a beneficial role in insulin sensitivity and glucose homeostasis.
- Banking on ATM as a new target in metabolic syndrome.
- Inflammation and insulin resistance.
- Regulation of IkappaB kinase and NF-kappaB in contracting adult rat skeletal muscle.
- Kinase activation through dimerization by human SH2-B.
- Local and systemic insulin resistance resulting from hepatic activation of IKK-beta and NF-kappaB.
- IKKbeta/NF-kappaB activation causes severe muscle wasting in mice.
- A phenylalanine zipper mediates APS dimerization.
- Insulin resistance due to phosphorylation of insulin receptor substrate-1 at serine 302.
- Structural basis for HNF-4alpha activation by ligand and coactivator binding.
- Two new substrates in insulin signaling, IRS5/DOK4 and IRS6/DOK5.
- Grb10 inhibits insulin-stimulated insulin receptor substrate (IRS)-phosphatidylinositol 3-kinase/Akt signaling pathway by disrupting the association of IRS-1/IRS-2 wit...
- Diabetes mutations delineate an atypical POU domain in HNF-1alpha.
- Crystal structure of the HNF4 alpha ligand binding domain in complex with endogenous fatty acid ligand.
- Structure of the globular tail of nuclear lamin.
- Mechanism by which high-dose aspirin improves glucose metabolism in type 2 diabetes.
- Genetic variability in insulin action inhibitor Ikkbeta (IKBKB) does not play a major role in the development of type 2 diabetes.
- Regulation of diet-induced adipose tissue and systemic inflammation by salicylates and pioglitazone.
- Sustained NF-κB Activation and Inhibition in β-Cells Have Minimal Effects on Function and Islet Transplant Outcomes.
Schools
U Of Chgo Div Of Bio Sci Pritzker Sch Of Med
Doctors Specialties
- Internal Medicine
- Endocrinology
Accepted Insurances
Education
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Beth Israel Deaconess Medical Center -
Brigham and Women's Hospital -
University of Chicago Pritzker School of Medicine
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