Physician
Gastroenterology
Dr. Steven A. Weinman, MD
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Dr. Steven A. Weinman is an expert in the field of Gastroenterology. He studied medicine at Yale University School of Medicine. Yale University School of Medicine is a top medical college. Yale University School of Medicine is ranked 7/57 in Research/PrimaryCare. He received awards:"Regional Top Doctor" and "CMS Meaningful Use Stage 1 Certification". Doctor Steven A. Weinman, MD is a published doctor and has 65 publications published. The lastest was: 'Tumor necrosis factor-alpha-inducible IkappaBalpha proteolysis mediated by cytosolic m-calpain. A mechanism parallel to the ubiquitin-proteasome pathway for nuclear fa...'
Publications
- Hepatitis C virus structural proteins can exacerbate or ameliorate acetaminophen-induced liver injury in mice.
- Hepatitis C and Alcohol Exacerbate Liver Injury by Suppression of FOXO3.
- Donor PNPLA3 rs738409 genotype affects fibrosis progression in liver transplantation for Hepatitis C.
- Interactions Between Hepatitis C Virus and Mitochondria: Impact on Pathogenesis and Innate Immunity.
- Forkhead box class O transcription factors in liver function and Disease.
- Regulation of FOXO3 by phosphorylation and methylation in Hepatitis C virus infection and alcohol exposure.
- Increased incidence of aflatoxin B1-induced liver tumors in Hepatitis virus C transgenic mice.
- Antioxidants as therapeutic agents for liver Disease.
- Upregulation of Hedgehog pathway is associated with cellular permissiveness for Hepatitis C virus replication.
- Resistance mutations define specific antiviral effects for inhibitors of the Hepatitis C virus p7 ion channel.
- The impact of partial manganese superoxide dismutase (SOD2)-deficiency on mitochondrial oxidant stress, DNA fragmentation and liver injury during acetaminophen hepatot...
- Impact of Hepatitis C virus infection on the course and outcome of patients with acute alcoholic Hepatitis.
- Dissecting the dynamic turnover of GFP-LC3 in the autolysosome.
- Persistent expression of Hepatitis C virus non-structural proteins leads to increased autophagy and mitochondrial injury in human hepatoma cells.
- Role of Hepatitis C virus core protein in viral-induced mitochondrial dysfunction.
- Alcohol and Hepatitis C virus--interactions in immune dysfunctions and liver damage.
- Gut microbes define liver cancer risk in mice exposed to chemical and viral transgenic hepatocarcinogens.
- Intracellular proton conductance of the Hepatitis C virus p7 protein and its contribution to infectious virus production.
- Chapter 20 Effects of Hepatitis C core protein on mitochondrial electron transport and production of reactive oxygen species.
- Alcohol metabolism increases the replication of Hepatitis C virus and attenuates the antiviral action of interferon.
- Proteasome activation by Hepatitis C core protein is reversed by ethanol-induced oxidative stress.
- The ClC-3 chloride transport protein traffics through the plasma membrane via interaction of an N-terminal dileucine cluster with clathrin.
- Hepatitis C virus core protein increases mitochondrial ROS production by stimulation of Ca2+ uniporter activity.
- GB virus B disrupts RIG-I signaling by NS3/4A-mediated cleavage of the adaptor protein MAVS.
- Causes and consequences of mitochondrial reactive oxygen species generation in Hepatitis C.
- Hepatic iron overload induces hepatocellular carcinoma in transgenic mice expressing the Hepatitis C virus polyprotein.
- Viral and therapeutic control of IFN-beta promoter stimulator 1 during Hepatitis C virus infection.
- Hepatitis C infection and alcohol use: A dangerous mix for the liver and antiviral immunity.
- Hepatitis C virus core protein inhibits deoxycholic acid-mediated apoptosis despite generating mitochondrial reactive oxygen species.
- Hepatitis C virus core protein inhibits mitochondrial electron transport and increases reactive oxygen species (ROS) production.
- RSA 2004: combined basic research satellite symposium - session four: hepatitis virus and alcohol interactions in immunity and liver Disease.
- Mitochondrial dysfunction in Hepatitis C.
- Hepatitis C virus core protein, cytochrome P450 2E1, and alcohol produce combined mitochondrial injury and cytotoxicity in hepatoma cells.
- Involvement of chloride channels in hepatic copper metabolism: ClC-4 promotes copper incorporation into ceruloplasmin.
- Plasma appearance and tissue accumulation of non-esterified, free astaxanthin in C57BL/6 mice after oral dosing of a disodium disuccinate diester of astaxanthin (Heptax).
- Hepatitis C: a metabolic liver Disease.
- Hepatitis C and alcohol: fundamental and translational research directions.
- Mrp2 modulates the activity of chloride channels in isolated hepatocytes.
- The ClC-3 chloride channel promotes acidification of lysosomes in CHO-K1 and Huh-7 cells.
- Chloride channels and hepatocellular function: prospects for molecular identification.
- Porphyria cutanea tarda: multiplicity of risk factors including HFE mutations, Hepatitis C, and inherited uroporphyrinogen decarboxylase deficiency.
- Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by Hepatitis C virus core protein.
- Steatosis and liver cancer in transgenic mice expressing the structural and nonstructural proteins of Hepatitis C virus.
- Annexin expressions are temporally and spatially regulated during rat hepatocyte differentiation.
- Biophysical properties of ClC-3 differentiate it from swelling-activated chloride channels in Chinese hamster ovary-K1 cells.
- Oxidative stress as a cause of biliary lithiasis: new possibilities for medical therapy of stones or a false alarm?
- Expression and canalicular localization of two isoforms of the ClC-3 chloride channel from rat hepatocytes.
- A transgenic mouse model of steatosis and hepatocellular carcinoma associated with chronic Hepatitis C virus infection in humans.
- Tumor necrosis factor-alpha-inducible IkappaBalpha proteolysis mediated by cytosolic m-calPain. A mechanism parallel to the ubiquitin-proteasome pathway for nuclear fa...
- Bile acid uptake via the human apical sodium-bile acid cotransporter is electrogenic.
- Expression and regulation of leukotriene-synthesis enzymes in rat liver cells.
- Electrogenicity of Na(+)-coupled bile acid transporters.
- Leukotriene D4 activates a chloride conductance in hepatocytes from lipopolysaccharide-treated rats.
- Photosensitization of experimental hepatocellular carcinoma with protoporphyrin synthesized from administered delta-aminolevulinic acid: studies with cultured cells an...
- cAMP- and swelling-activated chloride conductance in rat hepatocytes.
- cAMP stimulates fluorescent bile acid uptake into hepatocytes by membrane hyperpolarization.
- Transport characteristics of three fluorescent conjugated bile acid analogs in isolated rat hepatocytes and couplets.
- Identifying the hepatic organic anion transporter: one of many?
- Free concentrations of intracellular fluorescent anions determined by cytoplasmic dialysis of isolated hepatocytes.
- Maintenance of cellular acidification in cyanide-treated hepatocytes results from inhibition of Na+/H+ exchange.
- Electrogenicity of Na-coupled bile salt transport in isolated rat hepatocytes.
- Anion channels in rat liver canalicular plasma membranes reconstituted into planar lipid bilayers.
- The interaction of nerve growth factor with peripheral neurons: properties of the plasma membrane receptor.
- Mechanisms of doxorubicin resistance in hepatocellular carcinoma.
Schools
Washington Center School of Medicine
Yale New Haven Hospital
Procedures Preformed
- Endoscopy (Esophagus, Stomach, Small Intestine)
- Liver Biopsy
Conditions Treated
- Abdominal Pain
- Anemia
- Cholecystitis and Gallstones
- Cirrhosis
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Doctors Specialties
- Gastroenterology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Preferred Care Blue PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- Coventry Health Care
- Coventry Health Care of Kansas, Inc. PPO
- First Health (Coventry Health Care) PPO
- GEHA
- Choice Care PPO
- Preferred PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
Education
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Yale University School of Medicine -
Yale New Haven Children's Center -
Washington University School of Medicine in St. Louis
Drug Facts
| NPI NUMBER | 1326109588 | |
| NPPES Provider LastName | WEINMAN | |
| NPPES Provider FirstName | STEVEN | |
| NPPES Provider ZIPCode | 66160 | |
| NPPES Provider State | KS | |
| Specialty Description | Gastroenterology | |
| Total Claim Count | 146.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1326109588 |
| Last Name Of The Provider | WEINMAN |
| First Name Of The Provider | STEVEN |
| View All |
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