Professor of Neuropathology, University Iowa College of Medicine
Pathology
Dr. Steven A. Moore, MD
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Doctor Steven A. Moore, MD is an expert in the field of Pathology. He went to Indiana University School of Medicine. Indiana University School of Medicine has a rank of 45 in research and a 47 in primary care Steven Moore is also a published doctor. He has 52 publication published. The most recent publication is: A slowly progressive form of limb-girdle muscular dystrophy type 2C associated with founder mutation in the SGCG gene in Puerto Rican Hispanics. He accepts Medicare payments and is listed with Medicare.gov.
Publications
- X-linked myopathy with excessive autophagy: a failure of self-eating.
- Intrafamilial variability in GMPPB-associated dystroglycanopathy: Broadening of the phenotype.
- A slowly progressive form of limb-girdle muscular dystrophy type 2C associated with founder mutation in the SGCG gene in Puerto Rican Hispanics.
- Urologic and gastrointestinal symptoms in the dystroglycanopathies.
- Early-progressive dilated cardiomyopathy in a family with Becker muscular dystrophy related to a novel frameshift mutation in the dystrophin gene exon 27.
- Endogenous Glucuronyltransferase Activity of LARGE or LARGE2 Required for Functional Modification of α-Dystroglycan in Cells and Tissues.
- Joiner et al. reply.
- Late-adult onset of X-linked myopathy with excessive autophagy (XMEA).
- A novel missense mutation in POMT1 modulates the severe congenital muscular dystrophy phenotype associated with POMT1 nonsense mutations.
- LARGE glycans on dystroglycan function as a tunable matrix scaffold to prevent dystrophy.
- Astrocytes in the rat nucleus tractus solitarii are critical for cardiovascular reflex control.
- Sex chromosome complement influences functional callosal myelination.
- Novel deletion of lysine 7 expands the clinical, histopathological and genetic spectrum of TPM2-related myopathies.
- Dystroglycan on radial glia end feet is required for pial basement membrane integrity and columnar organization of the developing cerebral cortex.
- CaMKII determines mitochondrial stress responses in heart.
- ISPD loss-of-function mutations disrupt dystroglycan O-mannosylation and cause Walker-Warburg syndrome.
- Sudden Death and Myocardial Lesions after Damage to Catecholamine Neurons of the Nucleus Tractus Solitarii in Rat.
- Genetic ablation of complement C3 attenuates muscle pathology in dysferlin-deficient mice.
- Distinct functions of glial and neuronal dystroglycan in the developing and adult mouse brain.
- Elevated oxidative membrane damage associated with genetic modifiers of Lyst-mutant phenotypes.
- Cardiac pathology exceeds skeletal muscle pathology in two cases of limb-girdle muscular dystrophy type 2I.
- Visual impairment in the absence of dystroglycan.
- Allele-specific RNAi mitigates phenotypic progression in a transgenic model of Alzheimer's disease.
- Basal lamina strengthens cell membrane integrity via the laminin G domain-binding motif of alpha-dystroglycan.
- Cardiac damage after lesions of the nucleus tractus solitarii.
- Brain and eye malformations resembling Walker-Warburg syndrome are recapitulated in mice by dystroglycan deletion in the epiblast.
- Collagen VI glycine mutations: perturbed assembly and a spectrum of clinical severity.
- Dysferlin-deficient muscular dystrophy features amyloidosis.
- Progressive external ophthalmoplegia and vision and hearing loss in a patient with mutations in POLG2 and OPA1.
- Muscle fatigue increases the probability of developing hyperalgesia in mice.
- 20-Hydroxyeicosatetraenoic acid is a potent dilator of mouse basilar artery: role of cyclooxygenase.
- Hughlings Jackson and the role of the entorhinal cortex in temporal lobe epilepsy: from patient A to Doctor Z.
- Limb-girdle muscular dystrophy in the United States.
- Pharmacological characterization of endocannabinoid transport and fatty acid amide hydrolase inhibitors.
- Cannabinoids biology: the search for new therapeutic targets.
- Both laminin and Schwann cell dystroglycan are necessary for proper clustering of sodium channels at nodes of Ranvier.
- Omega-oxidation of 20-hydroxyeicosatetraenoic acid (20-HETE) in cerebral microvascular smooth muscle and endothelium by alcohol dehydrogenase 4.
- Deep white matter pathologic features in watershed regions: a novel pattern of central nervous system involvement in MELAS.
- Induction of cyclooxygenase-2 by anandamide in cerebral microvascular endothelium.
- LARGE can functionally bypass alpha-dystroglycan glycosylation defects in distinct congenital muscular dystrophies.
- Multiminicore myopathy, central core disease, malignant hyperthermia susceptibility, and RYR1 mutations: one disease with many faces?
- Metabolism of anandamide in cerebral microvascular endothelial cells.
- Unique role of dystroglycan in peripheral nerve myelination, nodal structure, and sodium channel stabilization.
- Limb-girdle muscular dystrophy.
- Identification of a functional peroxisome proliferator-activated receptor response element in the rat catalase promoter.
- The oxygen radical scavenger pyrrolidine dithiocarbamate enhances interleukin-1beta-induced cyclooxygenase-2 expression in cerebral microvascular smooth muscle cells.
- Disruption of DAG1 in differentiated skeletal muscle reveals a role for dystroglycan in muscle regeneration.
- Limb-girdle muscular dystrophies.
- Cyclooxygenase-2 expression and activity are induced by amyloid-beta 1-42 in brain derived cells.
- Glial scaffold required for cerebellar granule cell migration is dependent on dystroglycan function as a receptor for basement membrane proteins.
- Differential activation of catalase expression and activity by PPAR agonists: Implications for astrocyte protection in anti-glioma therapy.
Schools
Indiana University School of Medicine
University Iowa Hospital
Doctors Specialties
- Neuropathology
- Pathology
Accepted Insurances
Education
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University of Iowa Hospitals and Clinics -
Indiana University School of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1174511935 |
| Last Name Of The Provider | MOORE |
| First Name Of The Provider | STEVEN |
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