Assistant Professor, Pathology, UCSF School of Medicine
Pathology
Dr. Scott A. Oakes, MD
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Scott A. Oakes, MD is an expert in the field of Pathology. He researched medicine at University of Connecticut School of Medicine. University of Connecticut School of Medicine is ranked 60 in research and 49 in primary care medicine Scott Oakes is a published physician. He published 22 publications, including: 'Fas-Activated Mitochondrial Apoptosis Culls Stalled Embryonic Stem Cells to Promote Differentiation.'
Publications
- Fas-Activated Mitochondrial Apoptosis Culls Stalled Embryonic Stem Cells to Promote Differentiation.
- CDK1 Inhibition Targets the p53-NOXA-MCL1 Axis, Selectively Kills Embryonic Stem Cells, and Prevents Teratoma Formation.
- The Role of Endoplasmic Reticulum Stress in Human Pathology.
- Immune evasion mediated by tumor-derived lactate dehydrogenase induction of NKG2D ligands on myeloid cells in glioblastoma patients.
- Allosteric Inhibition of the IRE1α RNase Preserves Cell Viability and Function during Endoplasmic Reticulum Stress.
- Divergent allosteric control of the IRE1α endoribonuclease using kinase inhibitors.
- IRE1α Cleaves Select microRNAs during ER Stress to Derepress Translation of Proapoptotic Caspase-2.
- IRE1α Induces Thioredoxin-Interacting Protein to Activate the NLRP3 Inflammasome and Promote Programmed Cell Death under Irremediable ER Stress.
- The adaptor protein CRK is a pro-apoptotic transducer of endoplasmic reticulum stress.
- Multiple defects, including premature apoptosis, prevent KSHV replication in murine cells.
- Blocking the mitochondrial apoptotic pathway preserves motor neuron viability and function in a mouse model of amyotrophic lateral sclerosis.
- Akt and autophagy cooperate to promote survival of drug-resistant glioma.
- IRE1alpha kinase activation modes control alternate endoribonuclease outputs to determine divergent cell fates.
- Caspase-2 cleavage of BID is a critical apoptotic signal downstream of endoplasmic reticulum stress.
- A kinase inhibitor activates the IRE1alpha RNase to confer cytoprotection against ER stress.
- Mitochondria control calcium entry at the immunological synapse.
- Proapoptotic BAX and BAK regulate the type 1 inositol trisphosphate receptor and calcium leak from the endoplasmic reticulum.
- Untangling the web: mitochondrial fission and apoptosis.
- BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis.
- A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis.
- Golgi fragmentation precedes neuromuscular denervation and is associated with endosome abnormalities in SOD1-ALS mouse motor neurons.
Schools
Univ Of Ct Sch Of Med
Brigham and Women39s HospHarvard
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
- Basic HMO
- Vitalidad Plus California Con Aetna
- Blue Card PPO
- First Health (Coventry Health Care) PPO
- Covered California - Bronze PPO
- Health Net PPO
- Individual and Family PPO
Education
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Brigham and Women's Hospital -
University of Connecticut School of Medicine
Hospital
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San Francisco General Hospital and Trauma Center -
UCSF Medical Center
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