Assistant Professor, Medicine, Vanderbilt University School of Medicine
Hematology
Dr. Sandra S. Zinkel, MD
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Dr. Sandra S. Zinkel is an expert in the field of Hematology. She studied medicine at University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine is ranked 10 in research and 19 in primary care medicine Sandra S. Zinkel is a published doctor and has 23 publications published. The lastest was: 'Transgenic studies with a keratin promoter-driven growth hormone transgene: prospects for gene therapy.'
Publications
- MTG16 contributes to colonic epithelial integrity in experimental colitis.
- Rejuvenating Bi(d)ology.
- The loss of the BH3-only Bcl-2 family member Bid delays T-cell leukemogenesis in Atm-/- mice.
- BID binds to replication protein A and stimulates ATR function following replicative stress.
- Bid regulates the pathogenesis of neurotropic reovirus.
- Investigation of the proapoptotic BCL-2 family member bid on the crossroad of the DNA damage response and apoptosis.
- Bid plays a role in the DNA damage response.
- A role for proapoptotic BID in the DNA-damage response.
- Conditional MLL-CBP targets GMP and models therapy-related myeloproliferative disease.
- Proapoptotic BID is required for myeloid homeostasis and tumor suppression.
- Function of BID -- a molecule of the bcl-2 family -- in ischemic cell death in the brain.
- A role for proapoptotic Bax and Bak in T-cell differentiation and transformation.
- Proapoptotic Bid mediates the Atr-directed DNA damage response to replicative stress.
- c-mos expression in mouse oocytes is controlled by initiator-related sequences immediately downstream of the transcription initiation site.
- Catabolite activator protein-induced DNA bending in transcription initiation.
- Identification of a negative regulatory element that inhibits c-mos transcription in somatic cells.
- BCL2 family in DNA damage and cell cycle control.
- BID mediates neuronal cell death after oxygen/ glucose deprivation and focal cerebral ischemia.
- Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules.
- Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis.
- Transgenic studies with a keratin promoter-driven growth hormone transgene: prospects for gene therapy.
- Skin cancer and transgenic mice.
Schools
U Of Chgo Div Of Bio Sci Pritzker Sch Of Med
Barnes Jewish Hospital
Conditions Treated
- Anemia
- Myeloma
Doctors Specialties
- Hematology
- Medical Oncology
- Oncology
Accepted Insurances
Education
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Washington University -
Washington University - Barnes-Jewish Hospital -
University of Chicago Pritzker School of Medicine
Hospital
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Vanderbilt Hospital and Clinics
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