Co-Director Partners MS center
Neurology
Dr. Samia J. Khoury, MD
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Samia Khoury practices Neurology. He over the years received 2 awards: "Elected Member" and "Super Doctor". Samia J. Khoury, MD is also published. He has 69 publications published. The lastest publication: "BCL6 Controls Th9 Cell Development by Repressing Il9 Transcription.'
Publications
- BCL6 Controls Th9 Cell Development by Repressing Il9 Transcription.
- Clinical relevance and functional consequences of the TNFRSF1A Multiple sclerosis locus.
- Evaluation of circulating osteopontin levels in an unselected cohort of patients with Multiple sclerosis: relevance for biomarker development.
- Sex influences in autoimmune disease.
- IL-4 and Retinoic Acid Synergistically Induce Regulatory Dendritic Cells Expressing Aldh1a2.
- Circulating microRNAs as biomarkers for disease staging in Multiple sclerosis.
- In vitro and in vivo studies of IgG-derived Treg epitopes (Tregitopes): a promising new tool for tolerance induction and treatment of autoimmunity.
- Increased Th17 response to myelin peptides in pediatric MS.
- Galectin-1 Deactivates Classically Activated Microglia and Protects from Inflammation-Induced Neurodegeneration.
- Robust tumor immunity to melanoma mediated by interleukin-9-producing T cells.
- The Impact of Lesion In-Painting and Registration Methods on Voxel-Based Morphometry in Detecting Regional Cerebral Gray Matter Atrophy in Multiple Sclerosis.
- Notch receptors and Smad3 signaling cooperate in the induction of interleukin-9-producing T cells.
- Magnetic resonance disease severity scale (MRDSS) for patients with Multiple sclerosis: A longitudinal study.
- Therapeutic strategies in Multiple sclerosis.
- Plasticity of Ly-6Chi Myeloid Cells in T Cell Regulation.
- Notch ligand delta-like 4 blockade alleviates experimental autoimmune encephalomyelitis by promoting regulatory T cell development.
- Th17 and Treg balance in systemic Sclerosis.
- One year activity on subtraction MRI predicts subsequent 4 year activity and progression in Multiple sclerosis.
- Reversible neural stem cell niche dysfunction in a model of Multiple sclerosis.
- Brain MRI lesion load at 1.5T and 3T versus clinical status in Multiple sclerosis.
- The Relationship between Normal Cerebral Perfusion Patterns and White Matter Lesion Distribution in 1,249 Patients with Multiple Sclerosis.
- Immunopathogenesis of Multiple sclerosis.
- Multimodal coherent anti-Stokes Raman scattering microscopy reveals microglia-associated myelin and axonal dysfunction in Multiple sclerosis-like lesions in mice.
- Potential application of tregitopes as immunomodulating agents in Multiple sclerosis.
- Subventricular zone microglia transcriptional networks.
- A randomized controlled double-masked trial of albuterol add-on therapy in patients with Multiple sclerosis.
- TGF-beta induces IL-9 production from human Th17 cells.
- CD200R1 agonist attenuates mechanisms of chronic disease in a murine model of Multiple sclerosis.
- A putative Alzheimer's disease risk allele in PCK1 influences brain atrophy in Multiple sclerosis.
- Age-dependent B cell autoimmunity to a myelin surface antigen in pediatric Multiple sclerosis.
- IL-9 induces differentiation of TH17 cells and enhances function of FoxP3+ natural regulatory T cells.
- Localizing central nervous system immune surveillance: meningeal antigen-presenting cells activate T cells during experimental autoimmune encephalomyelitis.
- Rate of brain atrophy in benign vs early Multiple sclerosis.
- Deep gray matter involvement on brain MRI scans is associated with clinical progression in Multiple sclerosis.
- Antigen microarrays identify unique serum autoantibody signatures in clinical and pathologic subtypes of Multiple sclerosis.
- Predicting clinical progression in Multiple sclerosis with the magnetic resonance disease severity scale.
- Paradoxical dysregulation of the neural stem cell pathway sonic hedgehog-Gli1 in autoimmune encephalomyelitis and Multiple sclerosis.
- Immunotherapy for neurological diseases.
- Distinct functions of autoreactive memory and effector CD4+ T cells in experimental autoimmune encephalomyelitis.
- Neural stem cells and the future treatment of neurological diseases: raising the standard.
- JAGGED1 and delta1 differentially regulate the outcome of experimental autoimmune encephalomyelitis.
- CD11b+Ly-6C(hi) suppressive monocytes in experimental autoimmune encephalomyelitis.
- Modulating co-stimulation.
- Differential engagement of Tim-1 during activation can positively or negatively costimulate T cell expansion and effector function.
- Elevated neuronal expression of CD200 protects Wlds mice from inflammation-mediated neurodegeneration.
- Protecting axonal degeneration by increasing nicotinamide adenine dinucleotide levels in experimental autoimmune encephalomyelitis models.
- Innate immunity in Multiple sclerosis: myeloid dendritic cells in secondary progressive Multiple sclerosis are activated and drive a proinflammatory immune response.
- Differential role of programmed death-ligand 1 [corrected] and programmed death-ligand 2 [corrected] in regulating the susceptibility and chronic progression of experi...
- Insights into the molecular pathogenesis of progression in Multiple sclerosis: potential implications for future therapies.
- The Tim-3 ligand galectin-9 negatively regulates T helper type 1 immunity.
- A critical role for the programmed death ligand 1 in fetomaternal tolerance.
- Corticotropin-releasing hormone contributes to the peripheral inflammatory response in experimental autoimmune encephalomyelitis.
- Kinin B1 receptor expression on Multiple sclerosis mononuclear cells: correlation with magnetic resonance imaging T2-weighted lesion volume and clinical disability.
- Directed migration of neural stem cells to sites of CNS injury by the stromal cell-derived factor 1alpha/CXC chemokine receptor 4 pathway.
- Expression of Cux-1 and Cux-2 in the subventricular zone and upper layers II-IV of the cerebral cortex.
- Magnetic resonance imaging surrogates of Multiple sclerosis pathology and their relationship to central nervous system atrophy.
- The roles of the new negative T cell costimulatory pathways in regulating autoimmunity.
- Neural stem/progenitor cells express costimulatory molecules that are differentially regulated by inflammatory and apoptotic stimuli.
- Defining Th1 and Th2 immune responses in a reciprocal cytokine environment in vivo.
- Mutations in ARFGEF2 implicate vesicle trafficking in neural progenitor proliferation and migration in the human cerebral cortex.
- Role of costimulatory pathways in the pathogenesis of Multiple sclerosis and experimental autoimmune encephalomyelitis.
- Regulatory functions of CD8+CD28- T cells in an autoimmune disease model.
- Genetic programs and responses of neural stem/progenitor cells during demyelination: potential insights into repair mechanisms in Multiple sclerosis.
- Critical role of the programmed death-1 (PD-1) pathway in regulation of experimental autoimmune encephalomyelitis.
- The programmed death-1 (PD-1) pathway regulates autoimmune diabetes in nonobese diabetic (NOD) mice.
- 20. Immunologic neuromuscular disorders.
- Cytokine shifts and tolerance in experimental autoimmune encephalomyelitis.
- No sex-specific difference in disease trajectory in Multiple sclerosis patients before and after age 50.
Conditions Treated
- Epilepsy
- Migraine
- Multiple Sclerosis (MS)
Doctors Specialties
- Neurology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Commercial Insurance Company
- First Health (Coventry Health Care) PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Awards
- Elected Member
- Super Doctor
Education
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Brigham and Women's Hospital -
Case Western Reserve University - University Hospitals -
Case Western Reserve University - MetroHealth -
American University of Beirut Faculty of Medicine
Hospital
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Brigham and Women's Hospital
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