Professor, Anesthesiology, University of Miami School of Medicine
Anesthesiology
Dr. Roy C. Levitt, MD
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Levitt practices Anesthesiology. He researched medicine at University of Maryland School of Medicine. University of Maryland School of Medicine is ranked 37/52 in Research/PrimaryCare. Dr. Roy C. Levitt has written publications. He has 72 publications published with PubMed. The lastest publication is: 'Interleukin-9 promotes allergen-induced eosinophilic inflammation and airway hyperresponsiveness in transgenic mice.'
Publications
- Corneal Mechanical Thresholds Negatively Associate With Dry Eye and Ocular Pain Symptoms.
- Dry eye symptoms align more closely to non-ocular conditions than to tear film parameters.
- Agrin requires specific proteins to selectively activate γ-aminobutyric acid neurons for pain suppression.
- Neuropathic ocular pain: an important yet underevaluated feature of dry eye.
- Dry eye symptom severity and persistence are associated with symptoms of neuropathic pain.
- Interleukin 10 Mediated by Herpes Simplex Virus Vectors Suppresses Neuropathic Pain Induced by Human Immunodeficiency Virus gp120 in Rats.
- Mechanical Allodynia Induced by Nucleoside Reverse Transcriptase Inhibitor Is Suppressed by p55TNFSR Mediated by Herpes Simplex Virus Vector Through the SDF1 alpha/CXC...
- HSV-mediated p55TNFSR reduces neuropathic pain induced by HIV gp120 in rats through CXCR4 activity.
- Diabetic neuropathic pain development in type 2 diabetic mouse model and the prophylactic and therapeutic effects of coenzyme Q10.
- Prophylactic and Antinociceptive Effects of Coenzyme Q10 on Diabetic Neuropathic Pain in a Mouse Model of Type 1 Diabetes.
- TNFα and IL-1β are mediated by both TLR4 and Nod1 pathways in the cultured HAPI cells stimulated by LPS.
- Talniflumate increases survival in a cystic fibrosis mouse model of distal intestinal obstructive syndrome.
- Niflumic acid and MSI-2216 reduce TNF-alpha-induced mucin expression in human airway mucosa.
- Involvement of IL-9 in the bronchial phenotype of patients with nasal polyposis.
- Interleukin-9 induces mucous cell metaplasia independent of inflammation.
- Increased expression of interleukin-9, interleukin-9 receptor, and the calcium-activated chloride channel hCLCA1 in the upper airways of patients with cystic fibrosis.
- A polymorphism in the human UGRP1 gene promoter that regulates transcription is associated with an increased risk of asthma.
- A calcium-activated chloride channel (HCLCA1) is strongly related to IL-9 expression and mucus production in bronchial epithelium of patients with asthma.
- Carbonic Anhydrase-8 Regulates Inflammatory Pain by Inhibiting the ITPR1-Cytosolic Free Calcium Pathway.
- Atopy and bronchial hyperresponsiveness: exclusion of linkage to markers on chromosomes 11q and 6p.
- Expression of airway hyperreactivity to acetylcholine as a simple autosomal recessive trait in mice.
- Allelic loss of chromosome 18q and prognosis in colorectal cancer.
- Molecular genetic methods for mapping disease genes.
- Structure and DNA sequence of the mouse MnSOD gene.
- Genetic differences in the metabolic activation of benzo[a]pyrene in mice. Attempts to correlate tumorigenesis with binding of reactive intermediates to DNA and with m...
- Effects of environmental chemicals on the genetic regulation of microsomal enzyme systems.
- Susceptibility to ozone-induced inflammation. II. Separate loci control responses to acute and subacute exposures.
- Evidence for a locus regulating total serum IgE levels mapping to chromosome 5.
- Relationship between "aromatic hydrocarbon responsiveness" and the survival times in mice treated with various drugs and environmental compounds.
- Dinucleotide repeat polymorphism at the hormone sensitive lipase (LIPE) locus.
- Mast cells modulate acute ozone-induced inflammation of the murine lung.
- Susceptibility to ozone-induced inflammation. I. Genetic control of the response to subacute exposure.
- Linkage analysis of chromosome 12 markers in Italian families with atopic asthmatic children.
- Linkage homology for bronchial hyperresponsiveness between DNA markers on human chromosome 5q31-q33 and mouse chromosome 13.
- Linkage mapping of the human thromboxane A2 receptor (TBXA2R) to chromosome 19p13.3 using transcribed 3' untranslated DNA sequence polymorphisms.
- Two locus segregation and linkage analysis for total serum IgE levels.
- A genetic model for evaluation of susceptibility to ozone-induced inflammation.
- Mapping of the gene for hormone sensitive lipase (LIPE) to chromosome 19q13.1-->q13.2.
- G to A polymorphism in the CACNLG gene.
- Localization of the A3 adenosine receptor gene (ADORA3) to human chromosome 1p.
- Fluorescence-based resource for semiautomated genomic analyses using microsatellite markers.
- Airway hyperresponsiveness to acetylcholine: segregation analysis and evidence for linkage to murine chromosome 6.
- Evidence for linkage of total serum IgE and bronchial hyperresponsiveness to chromosome 5q: a major regulatory locus important in asthma.
- Interleukin-9-induced expression of M-Ras/R-Ras3 oncogene in T-helper clones.
- Fine-structure mapping of genes providing susceptibility to asthma on chromosome 5q31-q33.
- Linkage analysis of susceptibility to ozone-induced lung inflammation in inbred mice.
- IL-13 mediates in vivo IL-9 activities on lung epithelial cells but not on hematopoietic cells.
- Role of insulin receptor substrate-2 in interleukin-9-dependent proliferation.
- Evidence for two unlinked loci regulating total serum IgE levels.
- Genetic susceptibility to asthma--bronchial hyperresponsiveness coinherited with a major gene for atopy.
- Characterization of a calcium-activated chloride channel as a shared target of Th2 cytokine pathways and its potential involvement in asthma.
- Expression of HCLCA1 in cystic fibrosis lungs is associated with mucus overproduction.
- Interleukin 9: a candidate gene for asthma.
- Interleukin 9 promotes influx and local maturation of eosinophils.
- Functional expression of IL-9 receptor by human neutrophils from asthmatic donors: role in IL-8 release.
- Th2 cytokines and asthma. Interleukin-9 as a therapeutic target for asthma.
- Asthma and bronchial hyperresponsiveness linked to the XY long arm pseudoautosomal region.
- Molecular analysis of human interleukin-9 receptor transcripts in peripheral blood mononuclear cells. Identification of a splice variant encoding for a nonfunctional c...
- UGRP1, a uteroglobin/Clara cell secretory protein-related protein, is a novel lung-enriched downstream target gene for the T/EBP/NKX2.1 homeodomain transcription factor.
- IL-9 induces chemokine expression in lung epithelial cells and baseline airway eosinophilia in transgenic mice.
- IL-9 expression by human eosinophils: regulation by IL-1beta and TNF-alpha.
- Interleukin-9 upregulates mucus expression in the airways.
- IL-9 and its receptor in allergic and nonallergic lung disease: increased expression in asthma.
- IL-9 pathway in asthma: new therapeutic targets for allergic inflammatory disorders.
- Interleukin-9 promotes allergen-induced eosinophilic inflammation and airway hyperresponsiveness in transgenic mice.
- Glial NF-κB inhibition alters neuropeptide expression after sciatic nerve injury in mice.
- Effect of Th2 type cytokines on hCLCA1 and mucus expression in cystic fibrosis airways.
- Up-Regulation of Interleukin-9 and the Interleukin-9-Associated Calcium-Activated Chloride Channel hCLCA1 in Nasal Mucosa Following In Vivo Allergen Challenge.
- LPS-induced mucin expression in human sinus mucosa can be attenuated by hCLCA inhibitors.
- A calcium-activated chloride channel blocker inhibits goblet cell metaplasia and mucus overproduction.
- Increased expression of the calcium-activated chloride channel hCLCA1 in airways of patients with obstructive chronic bronchitis.
Schools
University of Maryland At Baltimore Professional Schools
Doctors Specialties
- Anesthesiology
- Internal Medicine
Accepted Insurances
Education
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Johns Hopkins University -
University of Maryland School of Medicine
Hospital
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University of Miami Hospital -
Miami Veterans Affairs Healthcare System
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