Professor, Internal Medicine, Virginia Commonwealth University, Medical College of Virginia School of Medicine
Internal Medicine
Dr. Robert W. Downs, MD
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Robert W. Downs practices Internal Medicine. He attended Duke University School of Medicine. Duke University School of Medicine is a top medical college. Duke University School of Medicine has a rank of 8 in research and a 29 in primary care He received 1 awards: "Top MD". Robert W. Downs, MD is also a published doctor. He has 71 paper published. The most recent paper is: Variations in physicians'' judgments about corticosteroid induced osteoporosis by physician specialty.
Publications
- A pilot study: effects of decreasing serum insulin With diazoxide on vitamin D levels in obese women With polycystic ovary syndrome.
- Complications of nephrogenic systemic fibrosis following repeated exposure to gadolinium in a man with hypoThyroidism: a case report.
- The long-term effects of gastric bypass on vitamin D Metabolism.
- Effects of gastric bypass procedures on bone mineral density, calcium, paraThyroid hormone, and vitamin D.
- Precision assessment and radiation safety for dual-energy X-ray absorptiometry: position paper of the International Society for Clinical Densitometry.
- Effect of raloxifene on the risk of new vertebral fracture in postmenopausal women With osteopenia or osteoporosis: a reanalysis of the Multiple Outcomes of Raloxifene...
- Significant differential effects of alendronate, estrogen, or combination therapy on the rate of bone loss after discontinuation of treatment of postmenopausal osteopo...
- Novel therapeutic options for Osteoporosis.
- Differential diagnosis of brown tumor vs. cystic osteitis by arteriography and computed tomography.
- Physician differences in managing postmenopausal Osteoporosis: results from the POSSIBLE US™ treatment registry study.
- Low-dose esterified estrogen therapy: effects on bone, plasma estradiol concentrations, endometrium, and Lipid levels. Estratab/Osteoporosis Study Group.
- Variations in physicians'' judgments about corticosteroid induced Osteoporosis by physician specialty.
- Persistence and switching patterns among women With varied osteoporosis medication histories: 12-month results from POSSIBLE US.
- Work in progress: Abnormal parathyroid glands. Comparison of nonselective arterial digital arteriography, selective parathyroid arteriography, and venous digital arter...
- Aspiration of enlarged paraThyroid glands for paraThyroid hormone assay.
- Alendronate and estrogen effects in postmenopausal women With low bone mineral density. Alendronate/Estrogen Study Group.
- Cyclic nucleotides regulate collagen production by human intestinal smooth muscle cells.
- Mild vitamin D deficiency and secondary hyperparaThyroidism in nursing home patients receiving adequate dietary vitamin D.
- Potentiation by glucose of lipolytic responsiveness of human adipocytes.
- Effect of short-term glucocorticoids on serum osteocalcin in healthy young men.
- Activity of the stimulatory guanine nucleotide-binding protein is reduced in erythrocytes from patients with pseudohypoparaThyroidism and pseudopseudohypoparaThyroidis...
- The inhibitory adenylate cyclase coupling protein in pseudohypoparaThyroidism.
- Impaired formation of beta-adrenergic receptor-nucleotide regulatory protein complexes in pseudohypoparaThyroidism.
- Dose-response relationships for alendronate treatment in osteoporotic elderly women. Alendronate Elderly Osteoporosis Study Centers.
- Effect of oral alendronate on bone mineral density and the incidence of fractures in postmenopausal Osteoporosis. The Alendronate Phase III Osteoporosis Treatment Stud...
- Familial hypocalciuric hypercalcemia: recognition among patients referred after unsuccessful paraThyroid exploration.
- The hypocalciuric or benign variant of familial Hypercalcemia: clinical and biochemical features in fifteen kindreds.
- The role of guanine nucleotides in regulation of adenylate cyclase activity.
- ParaThyroid imaging after intraarterial injections of [75Se]selenomethionine.
- Familial hypocalciuric hypercalcemia: the relation to primary paraThyroid hyperplasia.
- Calcium inhibition of adenylate cyclase: studies in turkey erythrocyte and S49 CYC- cell membranes.
- The effects of forskolin on adenylate cyclase in S49 wild Type and cyc- cells.
- Urinary calcium excretion in familial hypocalciuric hypercalcemia. Persistence of relative hypocalciuria after induction of hypoparaThyroidism.
- Deficient guanine nucleotide regulatory unit activity in cultured fibroblast membranes from patients with pseudohypoparaThyroidism type I. a cause of impaired synthesi...
- Resistance to multiple hormones in patients with pseudohypoparaThyroidism. Association with deficient activity of guanine nucleotide regulatory protein.
- Deficiency of hormone receptor-adenylate cyclase coupling protein: basis for hormone resistance in pseudohypoparaThyroidism.
- Vanadate stimulates adenylate cyclase via the guanine nucleotide regulatory protein by a mechanism differing from that of fluoride.
- ParaThyroid function after paraThyroidectomy: evaluation by measurement of urinary cAMP.
- Cholera-toxin-dependent ADP-ribosylation of the adenylate cyclase regulatory protein in turkey erythrocyte membranes.
- Urinary cyclic 3'',5''-adenosine monophosphate (cAMP): an index of the action of paraThyroid hormone on the kidney.
- Restoration of guanine nucleotide- and fluoride-stimulated activity to an adenylate cyclase-deficient cell line With affinity-purified guanine nucleotide regulatory pr...
- Intraoperative measurements of urinary cyclic AMP to guide surgery for primary hyperparaThyroidism.
- Deficient activity of guanine nucleotide regulatory protein in erythrocytes from patients with pseudohypoparaThyroidism.
- Adenosine 3',5'-monophosphate response to paraThyroid hormone: familial hypocalciuric hypercalcemia versus typical primary hyperparaThyroidism.
- Fluoride stimulation of adenylate cyclase is dependent on the guanine nucleotide regulatory protein.
- Clinical implications of guanine nucleotide-binding proteins as receptor-effector couplers.
- New form of pseudohypoparathyroidism with Abnormal catalytic adenylate cyclase.
- Cushing's Disease in two sisters.
- Glucocorticoids decrease vitamin D receptor number and gene expression in human osteosarcoma cells.
- Calcium-regulated paraThyroid hormone release in primary hyperparaThyroidism: studies in vitro with dispersed paraThyroid cells.
- Separation of a guanine nucleotide regulatory unit from the adenylate cyclase complex With GTP affinity chromatography.
- Direct comparison in vivo and in vitro of suppressibility of paraThyroid function by calcium in primary hyperparaThyroidism.
- Management of recurrent melanoma of the extremity.
- A familial syndrome of decrease in sensitivity to 1,25-dihydroxyvitamin D.
- Circulating paraThyroid hormone activity: familial hypocalciuric hypercalcemia versus typical primary hyperparaThyroidism.
- Human paraThyroid autografts: comparison of function in vivo and in vitro.
- Preoperative localization of Abnormal parathyroid: neck massage versus arteriography and selective venous sampling.
- Guanosine 5'', alpha-beta-methylene, triphosphate, a novel GTP analog, causes persistent activation of adenylate cyclase: evidence against pyrophosphorylation mechanism.
- An open-label extension study of alendronate treatment in elderly women With osteoporosis.
- Deficient adenylate cyclase regulatory protein in renal membranes from a patient with pseudohypoparaThyroidism.
- Persistent hyperparaThyroidism caused by incomplete paraThyroid resection and a hyperfunctioning paraThyroid autograft.
- Guanine nucleotides: key regulators of hormone receptor-adenylate cyclase interaction.
- Beta cell monoamines: further evidence for their role in modulating insulin secretion.
- Vitamin D-dependent rickets Type II. Defective induction of 25-hydroxyvitamin D3-24-hydroxylase by 1,25-dihydroxyvitamin D3 in cultured skin fibroblasts.
- Cushing's syndrome resulting from primary pigmented nodular adrenocortical Disease.
- Effects of arzoxifene on bone, lipid markers, and safety parameters in postmenopausal women With low bone mass.
- Comparison of alendronate and intranasal calcitonin for treatment of Osteoporosis in postmenopausal women.
- Ten years'' experience With alendronate for osteoporosis in postmenopausal women.
- Skeletal sites for Osteoporosis diagnosis: the 2005 ISCD Official Positions.
- Official positions of the international society for clinical densitometry.
Schools
Duke University School Of Medicine
Barnes Hospital Group
Niamdd Metabolic Diseases Branch
Nih Combined Endocrine Program
Conditions Treated
- Abnormal Thyroid
- Adrenal Incidentaloma
- Adrenal Insufficiency
- Adrenogenital Disorders
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Doctors Specialties
- Internal Medicine
Accepted Insurances
Awards
- Top MD
Education
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Washington University - Barnes-Jewish Hospital -
Duke University School of Medicine
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