Professor, Medicine, UCLA School of Medicine
Dermatology
Dr. Robert L. Modlin, MD
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Robert Modlin works in the field of Dermatology. He researched medicine at NYU School of Medicine. He received 4 awards: "CMS Meaningful Use Stage 1 Certification", "Elected Member", "Super Doctor" and "Fellow (FAAD)". Dr. Robert L. Modlin has written publications. He has 84 publications published with PubMed. The lastest paper is: 'Carbohydrate Dependent Binding of Langerin to SodC, a Cell Wall Glycoprotein of Mycobacterium leprae.'
Publications
- Comparison of molecular signatures from multiple Skin diseases identifies mechanisms of immunopathogenesis.
- Carbohydrate Dependent Binding of Langerin to SodC, a Cell Wall Glycoprotein of Mycobacterium leprae.
- IL-32 is a molecular marker of a host defense network in human tuberculosis.
- TB or Not TB: That Is No Longer the Question.
- IL-1β triggers the differentiation of macrophages with enhanced capacity to present mycobacterial antigen to T Cells.
- Antimicrobial and anti-inflammatory activity of chitosan-alginate nanoparticles: a targeted therapy for cutaneous pathogens.
- Galectin-3 regulates the innate immune response of human monocytes.
- Type I interferon suppresses type II interferon-triggered human anti-mycobacterial responses.
- Propionibacterium Acnes Strain Populations in the Human Skin Microbiome Associated with Acne.
- The helicase DDX41 recognizes the bacterial secondary messengers cyclic di-GMP and cyclic di-AMP to activate a type I interferon immune response.
- NOD2 triggers an interleukin-32-dependent human dendritic Cell program in leprosy.
- Innate Immunity: Ignored for Decades, but Not Forgotten.
- Shedding light on the vitamin D-tuberculosis-HIV connection.
- Vitamin D Is Required for IFN-{gamma}-Mediated Antimicrobial Activity of Human Macrophages.
- Structural differences in lipomannans from pathogenic and non-pathogenic mycobacteria that impact CD1b-restricted T Cell responses.
- Cord Blood Vitamin D Status Impacts Innate Immune Responses.
- Noninvasive in vivo imaging to evaluate immune responses and antimicrobial therapy against Staphylococcus aureus and USA300 MRSA Skin infections.
- T-Cell cytokines differentially control human monocyte antimicrobial responses by regulating vitamin D metabolism.
- Interleukin-4 regulates the expression of CD209 and subsequent uptake of Mycobacterium leprae by Schwann Cells in human leprosy.
- Vitamin D-binding protein directs monocyte responses to 25-hydroxy- and 1,25-dihydroxyvitamin D.
- IL-17 is essential for host defense against cutaneous Staphylococcus aureus Infection in mice.
- Integrated pathways for neutrophil recruitment and inflammation in leprosy.
- Learning from leprosy: insight into the human innate immune response.
- TLR2 looks at lipoproteins.
- Divergence of macrophage phagocytic and antimicrobial programs in leprosy.
- A vitamin for autophagy.
- Activation of Fc gamma RI on monocytes triggers differentiation into immature dendritic Cells that induce autoreactive T Cell responses.
- Downstream signals for MyD88-mediated phagocytosis of Borrelia burgdorferi can be initiated by TRIF and are dependent on PI3K.
- Anti-TNF immunotherapy reduces CD8+ T Cell-mediated antimicrobial activity against Mycobacterium tuberculosis in humans.
- The vitamin D connection to pediatric Infections and immune function.
- Vitamin d-directed rheostatic regulation of monocyte antibacterial responses.
- Convergence of IL-1beta and VDR activation pathways in human TLR2/1-induced antimicrobial responses.
- DNA transportation authority.
- IL-15 links TLR2/1-induced macrophage differentiation to the vitamin D-dependent antimicrobial pathway.
- "Dermal dendritic Cells" comprise two distinct populations: CD1+ dendritic Cells and CD209+ macrophages.
- Host-derived oxidized phospholipids and HDL regulate innate immunity in human leprosy.
- Conserved mycobacterial lipoglycoproteins activate TLR2 but also require glycosylation for MHC class II-restricted T Cell activation.
- LILRA2 activation inhibits dendritic Cell differentiation and antigen presentation to T Cells.
- Angiogenesis in cutaneous lesions of leprosy: implications for treatment.
- Inflammasome-mediated production of IL-1beta is required for neutrophil recruitment against Staphylococcus aureus in vivo.
- Vitamin D in defense of the human immune response.
- Cutting edge: vitamin D-mediated human antimicrobial activity against Mycobacterium tuberculosis is dependent on the induction of cathelicidin.
- Injury enhances TLR2 function and antimicrobial peptide expression through a vitamin D-dependent mechanism.
- Human keratinocyte Toll-like receptors promote distinct immune responses.
- Cathelicidin antimicrobial peptides block dendritic Cell TLR4 activation and allergic contact sensitization.
- Ito Cells are liver-resident antigen-presenting Cells for activating T Cell responses.
- A role for IRF3-dependent RXRalpha repression in hepatotoxicity associated with viral Infections.
- Expression of CD1d molecules by human schwann Cells and potential interactions with immunoregulatory invariant NK T Cells.
- Macrophages acquire neutrophil granules for antimicrobial activity against intraCellular pathogens.
- TLR activation of Langerhans Cell-like dendritic Cells triggers an antiviral immune response.
- Toll-like receptor triggering of a vitamin D-mediated human antimicrobial response.
- Human dendritic Cell expression of HLA-DO is subset specific and regulated by maturation.
- MyD88 mediates neutrophil recruitment initiated by IL-1R but not TLR2 activation in immunity against Staphylococcus aureus.
- Coordinate expression of CC chemokine ligand 5, granulysin, and perforin in CD8+ T Cells provides a host defense mechanism against Mycobacterium tuberculosis.
- Granulysin-derived peptides demonstrate antimicrobial and anti-inflammatory effects against Propionibacterium acnes.
- Immunology. Now presenting: gammadelta T Cells.
- The role of toll-like receptors in the pathogenesis and treatment of dermatological disease.
- TLR activation triggers the rapid differentiation of monocytes into macrophages and dendritic Cells.
- TGF-alpha regulates TLR expression and function on epidermal keratinocytes.
- The human CD1-restricted T Cell repertoire is limited to cross-reactive antigens: implications for host responses against immunologically related pathogens.
- Cutting edge: all-trans retinoic acid down-regulates TLR2 expression and function.
- Breaking tolerance--another piece added to the vitiligo puzzle.
- From plankton to pathogen recognition.
- Truncated structural variants of lipoarabinomannan in Mycobacterium leprae and an ethambutol-resistant strain of Mycobacterium tuberculosis.
- Langerhans Cells utilize CD1a and langerin to efficiently present nonpeptide antigens to T Cells.
- The role of Toll-like receptors in combating mycobacteria.
- Saposin C is required for lipid presentation by human CD1b.
- Toll-like receptors induce a phagocytic gene program through p38.
- Activation of Toll-like receptor 2 on human tracheobronchial epithelial Cells induces the antimicrobial peptide human beta defensin-2.
- Use of genetic profiling in leprosy to discriminate clinical forms of the disease.
- Apoptosis facilitates antigen presentation to T lymphocytes through MHC-I and CD1 in tuberculosis.
- Specific phospholipid oxidation products inhibit ligand activation of toll-like receptors 4 and 2.
- PIASx is a transcriptional co-repressor of signal transducer and activator of transcription 4.
- Activation and regulation of Toll-like receptors 2 and 1 in human leprosy.
- A role for triggering receptor expressed on myeloid Cells-1 in host defense during the early-induced and adaptive phases of the immune response.
- Human NKT Cells express granulysin and exhibit antimycobacterial activity.
- Expression of Toll-like receptor 2 on human Schwann Cells: a mechanism of nerve damage in leprosy.
- Toll-like receptor 2 ligands as adjuvants for human Th1 responses.
- The CD1b structure: antigen presentation adapts to a high-fat diet.
- Activation of toll-like receptor 2 in acne triggers inflammatory cytokine responses.
- Cutting edge: role of Toll-like receptor 1 in mediating immune response to microbial lipoproteins.
- Mammalian toll-like receptors.
- Impact of vitamin D on immune function: lessons learned from genome-wide analysis.
Schools
New York University School Of Medicine
L a Co Usc Med Center
Nyu Hospitals Center
Procedures Preformed
- Destruction of Benign Skin Lesion
Conditions Treated
- Actinic Keratosis
- Dermatitis
- Dry Skin
- Fungal Nail Infection
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Doctors Specialties
- Dermatology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
- Super Doctor
- Fellow (FAAD)
Education
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University of Southern California - LAC+USC Medical Center -
NYU School of Medicine
Hospital
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Ronald Reagan UCLA Medical Center -
Santa Monica-UCLA Medical Center and Orthopaedic Hospital
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