Physician
Cardiology
Dr. Robert Coronado, MD
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Robert Coronado works in the field of Cardiology. Robert Coronado has written publications. He has 86 publications published with PubMed. The lastest paper is: 'Ca2+ sparks in embryonic mouse skeletal muscle selectively deficient in dihydropyridine receptor alpha1S or beta1a subunits.' He is registered with Medicare and accepts Medicare payments.
Publications
- Reconstitution of the ATP-sensitive potassium channel of skeletal muscle. Activation by a G protein-dependent process.
- Recent advances in planar phospholipid bilayer techniques for monitoring ion channels.
- Detection of K+ and Cl-channels from calf cardiac sarcolemma in planar lipid bilayer membranes.
- Open-channel subconductance state of K+ channel from cardiac sarcoplasmic reticulum.
- Reconstitution of ionic channels from human heart.
- Reconstitution and characterization of a calcium-activated channel from heart.
- Insulation of the conduction pathway of muscle transverse tubule calcium channels from the surface charge of bilayer phospholipid.
- Reconstitution and phosphorylation of chloride channels from airway epithelium membranes.
- Sarcoplasmic reticulum contains adenine nucleotide-activated calcium channels.
- Phospholipid bilayers made from monolayers on patch-clamp pipettes.
- Effect of divalent cations on the assembly of neutral and charged phospholipid bilayers in patch-recording pipettes.
- Planar bilayer recording of ryanodine receptors of sarcoplasmic reticulum.
- Internal and external effects of dihydropyridines in the calcium channel of skeletal muscle.
- Single potassium channels with delayed rectifier behavior from lobster axon membranes.
- Activation of the Ca2+ release channel of cardiac sarcoplasmic reticulum by volatile anesthetics.
- A K+-selective conductance sensitive to cholinergic antagonists obtained by the fusion of axonal membrane vesicles to planar bilayers.
- Sidedness of reconstituted calcium channels from muscle transverse tubules as determined by D600 and D890 blockade.
- K+ channels gated by voltage and ions.
- Potassium channel of cardiac sarcoplasmic reticulum is a multi-ion channel.
- Single channel measurements of the calcium release channel from skeletal muscle sarcoplasmic reticulum. Activation by Ca2+ and ATP and modulation by Mg2+.
- Techniques for observing calcium channels from skeletal muscle sarcoplasmic reticulum in planar lipid bilayers.
- Chloride-induced Ca2+ release from the sarcoplasmic reticulum of chemically skinned rabbit psoas fibers and isolated vesicles of terminal cisternae.
- Single-channel calcium and barium currents of large and small conductance from sarcoplasmic reticulum.
- Monovalent ion current through single calcium channels of skeletal muscle transverse tubules.
- Ryanodine receptor channel of sarcoplasmic reticulum.
- Opening of dihydropyridine calcium channels in skeletal muscle membranes by inositol trisphosphate.
- Volatile anesthetics selectively alter [3H]ryanodine binding to skeletal and cardiac ryanodine receptors.
- Calcium channel activity in a purified dihydropyridine-receptor preparation of skeletal muscle.
- Structure and function of ryanodine receptors.
- Functional expression of cDNA encoding the Ca2+ release channel (ryanodine receptor) of rabbit skeletal muscle sarcoplasmic reticulum in COS-1 cells.
- Charybdotoxin and noxiustoxin, two homologous peptide inhibitors of the K+ (Ca2+) channel.
- Monoclonal antibody specific for the transverse tubular membrane of skeletal muscle activates the dihydropyridine-sensitive Ca2+ channel.
- Inhibition of dihydropyridine-sensitive calcium channels by the plant alkaloid ryanodine.
- Cardiac sarcoplasmic reticulum phosphorylation increases Ca2+ release induced by flash photolysis of nitr-5.
- Abnormal ryanodine receptor channels in malignant hyperthermia.
- Reduction in density of transverse tubules and L-type Ca(2+) channels in canine tachycardia-induced heart failure.
- Agonists Bay-K8644 and CGP-28392 open calcium channels reconstituted from skeletal muscle transverse tubules.
- Heterogeneity of conductance states in calcium channels of skeletal muscle.
- unc-68 encodes a ryanodine receptor involved in regulating C. elegans body-wall muscle contraction.
- Altered binding site for Ca2+ in the ryanodine receptor of human malignant hyperthermia.
- Dantrolene and azumolene inhibit [3H]PN200-110 binding to porcine skeletal muscle dihydropyridine receptors.
- Scorpion toxins targeted against the sarcoplasmic reticulum Ca(2+)-release channel of skeletal and cardiac muscle.
- Calcium-dependent block of ryanodine receptor channel of swine skeletal muscle by direct binding of calmodulin.
- Purified ryanodine receptor from rabbit skeletal muscle is the calcium-release channel of sarcoplasmic reticulum.
- Ryanodine receptor of skeletal muscle is a gap junction-type channel.
- Ketamine, at clinical concentrations, does not alter the function of cardiac sarcoplasmic reticulum calcium release channels.
- L-thyroxine activates the intracellular Ca2+ release channel of skeletal muscle sarcoplasmic reticulum.
- Purification and characterization of ryanotoxin, a peptide with actions similar to those of ryanodine.
- Primary structure and properties of helothermine, a peptide toxin that blocks ryanodine receptors.
- Role of the ryanodine receptor of skeletal muscle in excitation-contraction coupling.
- Ca2+ release by inositol-trisphosphorothioate in isolated triads of rabbit skeletal muscle.
- Cyclic ADP-ribose induced Ca2+ release in rabbit skeletal muscle sarcoplasmic reticulum.
- Activation of the ryanodine receptor Ca2+ release channel of sarcoplasmic reticulum by a novel scorpion venom.
- Activation of the Ca2+ release channel of skeletal muscle sarcoplasmic reticulum by palmitoyl carnitine.
- The brain ryanodine receptor: a caffeine-sensitive calcium release channel.
- High molecular weight proteins in the nematode C. elegans bind [3H]ryanodine and form a large conductance channel.
- Fast release of 45Ca2+ induced by inositol 1,4,5-trisphosphate and Ca2+ in the sarcoplasmic reticulum of rabbit skeletal muscle: evidence for two types of Ca2+ release...
- Mechanism of chloride-dependent release of Ca2+ in the sarcoplasmic reticulum of rabbit skeletal muscle.
- Direct binding of verapamil to the ryanodine receptor channel of sarcoplasmic reticulum.
- A component of excitation-contraction coupling triggered in the absence of the T671-L690 and L720-Q765 regions of the II-III loop of the dihydropyridine receptor alpha...
- Functional analysis of a frame-shift mutant of the dihydropyridine receptor pore subunit (alpha1S) expressing two complementary protein fragments.
- Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coup...
- Involvement of the carboxy-terminus region of the dihydropyridine receptor beta1a subunit in excitation-contraction coupling of skeletal muscle.
- Contribution of ryanodine receptor type 3 to Ca(2+) sparks in embryonic mouse skeletal muscle.
- Modulation of L-type Ca2+ current but not activation of Ca2+ release by the gamma1 subunit of the dihydropyridine receptor of skeletal muscle.
- Purified ryanodine receptor from skeletal muscle sarcoplasmic reticulum is the Ca2+-permeable pore of the calcium release channel.
- Removal of Mg2+ inhibition of cardiac ryanodine receptor by palmitoyl coenzyme A.
- Intramembrane charge movements and excitation- contraction coupling expressed by two-domain fragments of the Ca2+ channel.
- Comparison of Ca(2+) sparks produced independently by two ryanodine receptor isoforms (type 1 or type 3).
- Differential regulation of skeletal muscle L-type Ca2+ current and excitation-contraction coupling by the dihydropyridine receptor beta subunit.
- Ca2+ sparks in embryonic mouse skeletal muscle selectively deficient in dihydropyridine receptor alpha1S or beta1a subunits.
- Molecular origin of the L-type Ca2+ current of skeletal muscle myotubes selectively deficient in dihydropyridine receptor beta1a subunit.
- Recovery of Ca2+ current, charge movements, and Ca2+ transients in myotubes deficient in dihydropyridine receptor beta 1 subunit transfected with beta 1 cDNA.
- Reduced Ca2+ current, charge movement, and absence of Ca2+ transients in skeletal muscle deficient in dihydropyridine receptor beta 1 subunit.
- Interaction between the dihydropyridine receptor Ca2+ channel beta-subunit and ryanodine receptor type 1 strengthens excitation-contraction coupling.
- Multiple loops of the dihydropyridine receptor pore subunit are required for full-scale excitation-contraction coupling in skeletal muscle.
- Functional equivalence of dihydropyridine receptor alpha1S and beta1a subunits in triggering excitation-contraction coupling in skeletal muscle.
- Maurocalcine and domain A of the II-III loop of the dihydropyridine receptor Cav 1.1 subunit share common binding sites on the skeletal ryanodine receptor.
- Involvement of a heptad repeat in the carboxyl terminus of the dihydropyridine receptor beta1a subunit in the mechanism of excitation-contraction coupling in skeletal ...
- Membrane cholesterol modulates dihydropyridine receptor function in mice fetal skeletal muscle cells.
- Ca2+-dependent excitation-contraction coupling triggered by the heterologous cardiac/brain DHPR beta2a-subunit in skeletal myotubes.
- Ca2+ current and charge movements in skeletal myotubes promoted by the beta-subunit of the dihydropyridine receptor in the absence of ryanodine receptor type 1.
- Truncation of the carboxyl terminus of the dihydropyridine receptor beta1a subunit promotes Ca2+ dependent excitation-contraction coupling in skeletal myotubes.
- Molecular characterization of a two-domain form of the neuronal voltage-gated P/Q-type calcium channel alpha(1)2.1 subunit.
- Gamma 1 subunit interactions within the skeletal muscle L-type voltage-gated calcium channels.
Schools
Univ Nacional Autonoma De Mexico De Estudios Profesionales Iztacala
Mt Sinai Hospital
University of Arizona
University Of California Davis
Cardiovascular Institute Of The South
Procedures Preformed
- Aortic Aneurysm Repair
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac Myocardial Perfusion Imaging
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Doctors Specialties
- Cardiology
- Interventional Cardiology
Accepted Insurances
Education
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University of California Davis -
University of Arizona -
Detroit Medical Center - Wayne State University -
Universidad Nacional Autonoma de Mexico
Hospital
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Mercy Medical Center Redding -
St. Elizabeth Community Hospital
Drug Facts
| NPI NUMBER | 1376502203 | |
| NPPES Provider LastName | CORONADO | |
| NPPES Provider FirstName | ROBERT | |
| NPPES Provider ZIPCode | 960010427 | |
| NPPES Provider State | CA | |
| Specialty Description | Cardiology | |
| Total Claim Count | 8183.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1376502203 |
| Last Name Of The Provider | CORONADO |
| First Name Of The Provider | ROBERT |
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