Physician
Pathology
Dr. Robb E. Wilentz, MD
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Robb E. Wilentz is an expert in the field of Pathology. He researched medicine at Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is a top medical college. Johns Hopkins University School of Medicine is ranked 3 in research and 29 in primary care medicine Robb Wilentz is a published doctor. He has 60 papers published. The most current publication "Tumor-suppressor genes in pancreatic cancer." He accepts Medicare.
Publications
- Histopathological diagnosis of pancreatic intraepithelial neoplasia and intraductal papillary-mucinous neoplasms: interobserver agreement.
- RPL38, FOSL1, and UPP1 are predominantly expressed in the pancreatic ductal epithelium.
- Identification and analysis of precursors to invasive pancreatic cancer.
- Immunohistochemistry and in situ hybridization in pancreatic neoplasia.
- HLA-G upregulation in pre-malignant and malignant lesions of the gastrointestinal tract.
- A subset of pancreatic adenocarcinomas demonstrates coamplification of topoisomerase IIalpha and HER2/neu: use of immunolabeling and multicolor FISH for potential pati...
- Aberrant expression of maspin in idiopathic inflammatory bowel disease is associated with disease activity and neoplastic transformation.
- Systemic BCNU enhances the efficacy of local delivery of a topoisomerase I inhibitor against malignant glioma.
- p16 Inactivation in pancreatic intraepithelial neoplasias (PanINs) arising in patients with chronic pancreatitis.
- Novel markers of pancreatic adenocarcinoma in fine-needle aspiration: mesothelin and prostate stem cell antigen labeling increases accuracy in cytologically borderline...
- Analysis of anaphase figures in routine histologic sections distinguishes chromosomally unstable from chromosomally stable malignancies.
- Atheroembolism in cardiac surgery.
- Lesions missed on prostate biopsies in cases sent in for consultation.
- Role of the DPC4 tumor suppressor gene in adenocarcinoma of the ampulla of Vater: analysis of 140 cases.
- Pancreaticoduodenectomy (Whipple resections) in patients without malignancy: are they all 'chronic pancreatitis'?
- Premalignant conditions of the pancreas.
- Telomere shortening is nearly universal in pancreatic intraepithelial neoplasia.
- The dichotomy in the preinvasive neoplasia to invasive carcinoma sequence in the pancreas: differential expression of MUC1 and MUC2 supports the existence of two separ...
- Smad4 overexpression in hepatocellular carcinoma is strongly associated with transforming growth factor beta II receptor immunolabeling.
- Cyclooxygenase 2 expression in pancreatic adenocarcinoma and pancreatic intraepithelial neoplasia: an immunohistochemical analysis with automated cellular imaging.
- Hepatocellular carcinomas show abnormal expression of fibronectin protein.
- Immunohistochemical validation of a novel epithelial and a novel stromal marker of pancreatic ductal adenocarcinoma identified by global expression microarrays: sea ur...
- Pancreatic cancer.
- Aberrant methylation of preproenkephalin and p16 genes in pancreatic intraepithelial neoplasia and pancreatic ductal adenocarcinoma.
- K-ras, p53, and DPC4 (MAD4) alterations in fine-needle aspirates of the pancreas: a molecular panel correlates with and supplements cytologic diagnosis.
- MUC4 expression increases progressively in pancreatic intraepithelial neoplasia.
- Solid-pseudopapillary tumors of the pancreas are genetically distinct from pancreatic ductal adenocarcinomas and almost always harbor beta-catenin mutations.
- Direct correlation between proliferative activity and dysplasia in pancreatic intraepithelial neoplasia (PanIN): additional evidence for a recently proposed model of p...
- Almost all infiltrating colloid carcinomas of the pancreas and periampullary region arise from in situ papillary neoplasms: a study of 39 cases.
- Immunohistochemical labeling for the Dpc4 gene product is a specific marker for adenocarcinoma in biopsy specimens of the pancreas and bile duct.
- The SMAD4 protein and prognosis of pancreatic ductal adenocarcinoma.
- Mesothelin is overexpressed in the vast majority of ductal adenocarcinomas of the pancreas: identification of a new pancreatic cancer marker by serial analysis of gene...
- Loss of Smad4 function in pancreatic tumors: C-terminal truncation leads to decreased stability.
- Immunohistochemical [corrected] detection of the alternate INK4a-encoded tumor suppressor protein p14(ARF) in archival human cancers and cell lines using commercial an...
- Molecular pathology of pancreatic cancer.
- Pancreatic intraepithelial neoplasia and infiltrating adenocarcinoma: analysis of progression and recurrence by DPC4 immunohistochemical labeling.
- Discovery of new markers of cancer through serial analysis of gene expression: prostate stem cell antigen is overexpressed in pancreatic adenocarcinoma.
- Myofibroblastoma of the male breast: imaging appearance and ultrasound-guided core biopsy diagnosis.
- Loss of heterozygosity or intragenic mutation, which comes first?
- Differing rates of loss of DPC4 expression and of p53 overexpression among carcinomas of the proximal and distal bile ducts.
- Increased risk of incident pancreatic cancer among first-degree relatives of patients with familial pancreatic cancer.
- Nuclear localization of Dpc4 (Madh4, Smad4) in colorectal carcinomas and relation to mismatch repair/transforming growth factor-beta receptor defects.
- The latest in pancreatic cancer research: Lustgarten Foundation awardees present their findings.
- Dpc4 protein in mucinous cystic neoplasms of the pancreas: frequent loss of expression in invasive carcinomas suggests a role in genetic progression.
- Lipogenic enzymes fatty acid synthase and acetyl-coenzyme A carboxylase are coexpressed with sterol regulatory element binding protein and Ki-67 in fetal tissues.
- Dpc-4 protein is expressed in virtually all human intraductal papillary mucinous neoplasms of the pancreas: comparison with conventional ductal adenocarcinomas.
- Genetic progression in the pancreatic ducts.
- Genetic, immunohistochemical, and clinical features of medullary carcinoma of the pancreas: A newly described and characterized entity.
- Loss of expression of Dpc4 in pancreatic intraepithelial neoplasia: evidence that DPC4 inactivation occurs late in neoplastic progression.
- Immunohistochemical labeling for dpc4 mirrors genetic status in pancreatic adenocarcinomas : a new marker of DPC4 inactivation.
- Pathologic examination accurately predicts prognosis in mucinous cystic neoplasms of the pancreas.
- Transforming growth factor-beta responsiveness in DPC4/SMAD4-null cancer cells.
- Pathology of incipient pancreatic cancer.
- Tumor-suppressor genes in pancreatic cancer.
- Inactivation of the p16 (INK4A) tumor-suppressor gene in pancreatic duct lesions: loss of intranuclear expression.
- Pathology of cancer of the pancreas.
- K-ras mutations in the duodenal fluid of patients with pancreatic carcinoma.
- Can K-ras codon 12 mutations be used to distinguish benign bile duct proliferations from metastases in the liver? A molecular analysis of 101 liver lesions from 93 pat...
- Clinical significance of K-ras oncogene activation in ampullary neoplasms.
Schools
Johns Hopkins University School Of Medicine
Johns Hopkins Hospital
University Miami Jackson Meml Hospital
Doctors Specialties
- Dermatopathology
- Pathology
Accepted Insurances
Education
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Jackson Memorial Hospital - Jackson Health System -
Johns Hopkins University -
Johns Hopkins University School of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1033115209 |
| Last Name Of The Provider | WILENTZ |
| First Name Of The Provider | ROBB |
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