Physician
Radiation Oncology
Dr. Ranjit S. Bindra, MD
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Doctor Ranjit S. Bindra, MD works in the field of Radiation Oncology. He researched medicine at Yale University School of Medicine. Yale University School of Medicine is one of the top medical schools in the US. Yale University School of Medicine has a rank of 7 in research and a 57 in primary care He over the years received 1 awards: "CMS Meaningful Use Stage 1 Certification". Ranjit S. Bindra, MD is also published. He has 26 publications published. The lastest publication: "Advances in Radiation Therapy in Pediatric Neuro-oncology.' He accepts Medicare payments and is listed with Medicare.gov.
Publications
- Advances in Radiation Therapy in Pediatric Neuro-oncology.
- Identification of novel radiosensitizers in a high-throughput, cell-based screen for DSB repair inhibitors.
- Development of a novel method to create double-strand break repair fingerprints using next-generation sequencing.
- Delayed Cerebral Vasculopathy Following Cranial Radiation Therapy for Pediatric Tumors.
- Radiation sensitivity and sensitization in melanoma.
- Development of an assay to measure mutagenic non-homologous end-joining repair activity in mammalian cells.
- Inhibition of poly(ADP-ribose) polymerase down-regulates BRCA1 and RAD51 in a pathway mediated by E2F4 and p130.
- CHK2-dependent phosphorylation of BRCA1 in hypoxia.
- Basal repression of BRCA1 by multiple E2Fs and pocket proteins at adjacent E2F sites.
- Hypoxia-induced down-regulation of BRCA1 expression by E2Fs.
- Hypoxia-induced phosphorylation of Chk2 in an ataxia telangiectasia mutated-dependent manner.
- Alterations in DNA repair gene expression under hypoxia: elucidating the mechanisms of hypoxia-induced genetic instability.
- Down-regulation of Rad51 and decreased homologous recombination in hypoxic cancer cells.
- Decreased expression of the DNA mismatch repair gene Mlh1 under hypoxic stress in mammalian cells.
- Targeted disruption of the CCR5 gene in human hematopoietic stem cells stimulated by peptide nucleic acids.
- Repression of RAD51 gene expression by E2F4/p130 complexes in hypoxia.
- Chronic hypoxia decreases synthesis of homologous recombination proteins to offset chemoresistance and radioresistance.
- Hypoxia down-regulates DNA double strand break repair gene expression in prostate cancer cells.
- Genetic instability and the tumor microenvironment: towards the concept of microenvironment-induced mutagenesis.
- Regulation of DNA repair in hypoxic cancer cells.
- Co-repression of mismatch repair gene expression by hypoxia in cancer cells: role of the Myc/Max network.
- Hypoxia-induced genetic instability--a calculated mechanism underlying tumor progression.
- Introduction: the evolving picture of the hypoxic tumour microenvironment.
- Targeting the DNA damage response for cancer therapy.
- The important role of radiation therapy in early-stage diffuse large B-cell lymphoma: time to review the evidence once again.
Schools
Yale University
Doctors Specialties
- Radiation Oncology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Memorial Sloan-Kettering Cancer Center -
Yale University School of Medicine
Hospital
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Robert Wood Johnson University Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1326206855 |
| Last Name Of The Provider | BINDRA |
| First Name Of The Provider | RANJIT |
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