General Psychiatry/Narcotic Addiction/Pain Management

Psychiatry

Peter Stanfield is an expert in the field of Psychiatry. Dr. Peter Stanfield has written publications. He has 92 publications published with PubMed. The lastest publication is: 'The selectivity filter of a potassium channel, murine kir2.1, investigated using scanning cysteine mutagenesis.' He is registered with Medicare.

Publications

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• Community-based health care in Kibwezi, Kenya: 10 years in retrospect.
• Overseas aid--urban hospitals or primary care?
• Letter: Treatment of diarrhoea in undernourished children.
• The prediction of normal P3 latency and the diagnosis of dementia.
• P300 and the effects of aging: relevance to the diagnosis of dementia.
• Voltage sparks a GPCR.
• TASK-5, a novel member of the tandem pore K+ channel family.
• An improved cell isolation technique for studying intracellular Ca(2+) homeostasis in neurones of the cochlear nucleus.
• The K+ channel signature sequence of murine Kir2.1: mutations that affect microscopic gating but not ionic selectivity.
• Potassium-depolarization-induced cytoplasmic [Ca2+] transient in freshly dissociated pyramidal neurones of the rat dorsal cochlear nucleus.
• Residues beyond the selectivity filter of the K+ channel kir2.1 regulate permeation and block by external Rb+ and Cs+.
• Co-localization of the inwardly rectifying potassium ion channel, Kir2.2, and the substance P receptor in single locus coeruleus neurons.
• Characterisation of Kir2.0 proteins in the rat cerebellum and hippocampus by polyclonal antibodies.
• The possible role of a disulphide bond in forming functional Kir2.1 potassium channels.
• Possible modulatory role of voltage-activated Ca(2+) currents determining the membrane properties of isolated pyramidal neurones of the rat dorsal cochlear nucleus.
• Topology of the pore region of an inward rectifier K+ channel, Kir2.1.
• The selectivity filter of a potassium channel, murine kir2.1, investigated using scanning cysteine mutagenesis.
• The dependence of Ag+ block of a potassium channel, murine kir2.1, on a cysteine residue in the selectivity filter.
• Membrane currents influencing action potential latency in granule neurons of the rat cochlear nucleus.
• Effect of adenosine and intracellular GTP on KATP channels of mammalian skeletal muscle.
• Characterization of the hyperpolarization activated nonspecific cation current (Ih) of bushy neurones from the rat anteroventral cochlear nucleus studied in a thin bra...
• The role of a single aspartate residue in ionic selectivity and block of a murine inward rectifier K+ channel Kir2.1.
• A rate theory model for Mg2+ block of ATP-dependent potassium channels of rat skeletal muscle.
• Unitary delayed rectifier channels of rat hippocampal neurons: properties of block by external tetraethylammonium ions.
• Pore region of K+ channel RACTK1.
• The intrinsic gating of inward rectifier K+ channels expressed from the murine IRK1 gene depends on voltage, K+ and Mg2+.
• Inactivating ''ball'' peptide from Shaker B blocks Ca(2+)-activated but not ATP-dependent K+ channels of rat skeletal muscle.
• A single aspartate residue is involved in both intrinsic gating and blockage by Mg2+ of the inward rectifier, IRK1.
• The effect of intracellular anions on ATP-dependent potassium channels of rat skeletal muscle.
• Protein kinase C-mediated inhibition of an inward rectifier potassium channel by substance P in nucleus basalis neurons.
• Rubidium block and rubidium permeability of the inward rectifier of frog skeletal muscle fibres.
• Calcium inactivation in skeletal muscle fibres of the stick insect, Carausius morosus.
• Inward rectification in frog skeletal muscle fibres and its dependence on membrane potential and external potassium.
• The influence of the permeant ions thallous and potassium on inward rectification in frog skeletal muscle.
• Slow changes in currents through sodium channels in frog muscle membrane.
• Lateral distribution of sodium and potassium channels in frog skeletal muscle: measurements with a patch-clamp technique.
• Tetraethylammonium ions and the potassium permeability of excitable cells.
• Calcium currents of frog and insect skeletal muscle fibres measured during voltage clamp.
• Calcium and potassium currents in muscle fibres of an insect (Carausius morosus).
• Gating of a muscle K+ channel and its dependence on the permeating ion species.
• A new preparation for recording single-channel currents from skeletal muscle.
• A binding-site model for calcium channel inactivation that depends on calcium entry.
• The effect of the tetraethylammonium ion on the inwardly rectifying potassium channel of frog sartorius muscle.
• Actions of some anions on electrical properties and mechanical threshold of frog twitch muscle.
• The differential effects of tetraethylammonium and zinc ions on the resting conductance of frog skeletal muscle.
• The effect of the tetraethylammonium ion on the delayed currents of frog skeletal muscle.
• Actions of some cations on the electrical properties and mechanical threshold of frog sartorius muscle fibers.
• Electrical properties of white and red muscle fibres of the elasmobranch fish Scyliorhinus canicula.
• The onset of the effects of zinc and tetraethylammonium ions on action potential duration and twitch amplitude of single muscle fibres.
• Rubidium ions and the gating of delayed rectifier potassium channels of frog skeletal muscle.
• Single inwardly rectifying potassium channels in cultured muscle cells from rat and mouse.
• Multiple blocking mechanisms of ATP-sensitive potassium channels of frog skeletal muscle by tetraethylammonium ions.
• Substance P raises neuronal membrane excitability by reducing inward rectification.
• Intracellular Mg2+ may act as a co-factor in ion channel function.
• The action of external tetraethylammonium ions on unitary delayed rectifier potassium channels of frog skeletal muscle.
• Voltage-dependent calcium channels of excitable membranes.
• Studies of the unitary properties of adenosine-5''-triphosphate-regulated potassium channels of frog skeletal muscle.
• Distribution and mobility of voltage-gated ion channels in skeletal muscle.
• Voltage-dependent ATP-sensitive potassium channels of skeletal muscle membrane.
• Properties of single potassium channels in vesicles formed from the sarcolemma of frog skeletal muscle.
• ATP-dependent potassium channels of muscle cells: their properties, regulation, and possible functions.
• Substance P excites cultured cholinergic neurons in the basal forebrain.
• Modulation of inwardly rectifying channels by substance P in cholinergic neurones from rat brain in culture.
• The effect of intracellular pH on ATP-dependent potassium channels of frog skeletal muscle.
• Unitary A-currents of rat locus coeruleus neurones grown in cell culture: rectification caused by internal Mg2+ and Na+.
• Activation of ATP-dependent K+ currents in intact skeletal muscle fibres by reduced intracellular pH.
• The effect of zinc ions on the gating of the delayed potassium conductance of frog sartorius muscle.
• A mechanism for the fall in resting potassium conductance of frog skeletal muscle fibres occurring under extreme hyperpolarization [proceedings].
• Potential-dependent blockade by Ba2+ of resting potassium permeability of frog sartorius [proceedings].
• The selectivity of the delayed potassium conductance of frog skeletal muscle fibers.
• Potential-dependence of the rubidium block of inward rectification in frog skeletal muscle [proceedings].
• Potassium depletion and sodium block of potassium currents under hyperpolarization in frog sartorius muscle.
• Calcium currents in insect muscle [proceedings].
• Inward rectification in skeletal muscle: a blocking particle model.
• A potential- and time-dependent blockade of inward rectification in frog skeletal muscle fibres by barium and strontium ions.
• A calcium dependent inward current in frog skeletal muscle fibres.
• Cs(+) causes a voltage-dependent block of inward K currents in resting skeletal muscle fibres.
• The selectivity filter of the tandem pore potassium channel TASK-1 and its pH-sensitivity and ionic selectivity.
• The voltage-dependent block of ATP-sensitive potassium channels of frog skeletal muscle by caesium and barium ions.
• The response of the tandem pore potassium channel TASK-3 (K(2P)9.1) to voltage: gating at the cytoplasmic mouth.
• Homomers of Kir.3.4 in atrial myocytes: their relevance to atrial fibrillation.
• Calcium dependence of the inactivation of calcium currents in skeletal muscle fibers of an insect.
• The selectivity, voltage-dependence and acid sensitivity of the tandem pore potassium channel TASK-1: contributions of the pore domains.
• Multiple residues in the p-region and m2 of murine kir 2.1 regulate blockage by external ba.
• Orexin-stimulated MAP kinase cascades are activated through multiple G-protein signalling pathways in human H295R adrenocortical cells: diverse roles for orexins A and B.
• Insight into the mechanism of inactivation and pH sensitivity in potassium channels from molecular dynamics simulations.
• Orexins stimulate steroidogenic acute regulatory protein expression through multiple signaling pathways in human adrenal H295R cells.
• Melastatin-type transient receptor potential channel 7 is required for intestinal pacemaking activity.
• Spermine is fit to block inward rectifier (Kir) channels.
• Nuclear immunostaining in rat neuronal cells using two anti-Kir2.2 ion channel polyclonal antibodies.
• Constitutively active and G-protein coupled inward rectifier K+ channels: Kir2.0 and Kir3.0.

Conditions Treated

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• Bipolar Disorder
• Schizophrenia
• Schizophreniform and Schizoaffective Disorder

Doctors Specialties

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• Pain Medicine
• Psychiatry

Accepted Insurances

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Education

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• Universidad de Granada Faculty of Medicine

Hospital

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• Baylor St. Luke's Medical Center

Drug Facts

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NPI NUMBER		1841300431
NPPES Provider LastName		STANFIELD
NPPES Provider FirstName		PETER
NPPES Provider ZIPCode		775624205
NPPES Provider State		TX
Specialty Description		General Practice
Total Claim Count		320.0
Distinct Opioid Count		0.0
Opioid Claim Count		0.0
Percent Opioid Claims		0.0

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Medicare Facts

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National Provider Identifier [NPI]	1841300431
Last Name Of The Provider	STANFIELD
First Name Of The Provider	PETER
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