Cardiologist at Cypress Cardiology.
Cardiology
Dr. Peter Razeghi, MD
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Peter Razeghi practices Cardiology. He received 3 awards: "CMS Meaningful Use Stage 1 Certification", "Alpha Omega Alpha" and "Fellow (FACC)". Peter Razeghi has written publications. He has 43 publications published with PubMed. The lastest publication is: 'Interleukin-18 enhances lipopolysaccharide-induced interferon-gamma production in human whole blood cultures.' Peter Razeghi, MD accepts Medicare.
Publications
- Proposed regulation of gene expression by glucose in rodent Heart.
- The diagnosis and management of cardiovascular Disease in cancer patients.
- Return to the fetal gene program protects the stressed Heart: a strong hypothesis.
- Lack of NF-kappaB1 (p105/p50) attenuates unloading-induced downregulation of PPARalpha and PPARalpha-regulated gene expression in rodent Heart.
- Hypertrophy and atrophy of the Heart: the other side of remodeling.
- Mechanical unloading of the Heart activates the calpain system.
- Induction of antioxidant gene expression in a mouse model of ischemic Cardiomyopathy is dependent on reactive oxygen species.
- Transcriptional regulators of ribosomal biogenesis are increased in the unloaded Heart.
- Atrophy, hypertrophy, and hypoxemia induce transcriptional regulators of the ubiquitin proteasome system in the rat Heart.
- Atrophic remodeling of the transplanted rat Heart.
- Cardiac remodeling: UPS lost in transit.
- Metabolic energetics and genetics in the Heart.
- The FOXO3a transcription factor regulates cardiac myocyte size downstream of AKT signaling.
- PPAR-gamma agonist rosiglitazone ameliorates ventricular dysfunction in experimental Chronic mitral regurgitation.
- Linking gene expression to function: metabolic flexibility in the normal and Diseased heart.
- Heart Disease in diabetes--resist the beginnings.
- Degree of cardiac fibrosis and hypertrophy at time of implantation predicts Myocardial improvement during left ventricular assist device support.
- Hypoxia-induced decrease of UCP3 gene expression in rat Heart parallels metabolic gene switching but fails to affect mitochondrial respiratory coupling.
- Activity of the Akt/GSK-3beta pathway in the failing human Heart before and after left ventricular assist device support.
- Dynamic changes of gene expression in hypoxia-induced right ventricular hypertrophy.
- Atrophic remodeling of the Heart in vivo simultaneously activates pathways of protein synthesis and degradation.
- Regional heterogeneity in gene expression profiles: a transcript analysis in human and rat Heart.
- Mechanical unloading of the failing human Heart fails to activate the protein kinase B/Akt/glycogen synthase kinase-3beta survival pathway.
- Hypoxia-induced switches of myosin heavy chain iso-gene expression in rat Heart.
- Stanniocalcin-1 is a naturally occurring L-channel inhibitor in cardiomyocytes: relevance to human Heart failure.
- Reverse remodeling of the failing human Heart with mechanical unloading. Emerging concepts and unanswered questions.
- Downregulation of metabolic gene expression in failing human Heart before and after mechanical unloading.
- Impaired long-chain fatty acid oxidation and contractile dysfunction in the obese Zucker rat Heart.
- Downregulation of myocardial myocyte enhancer factor 2C and myocyte enhancer factor 2C-regulated gene expression in diabetic patients with nonischemic Heart failure.
- Mitochondrial proteins in hypertrophy and atrophy: a transcript analysis in rat Heart.
- Alterations of the circadian clock in the Heart by streptozotocin-induced diabetes.
- Myocardial tumor necrosis factor-alpha expression does not correlate with clinical indices of Heart failure in patients on left ventricular assist device support.
- Metabolic gene expression in fetal and failing human Heart.
- Intrinsic diurnal variations in cardiac metabolism and contractile function.
- Calcitonin gene-related peptide is not essential for the development of pressure overload-induced hypertrophy in vivo.
- Peritoneal dialysis fluids with a physiologic pH based on either lactate or bicarbonate buffer-effects on human mesothelial cells.
- Hypoxia in vivo decreases peroxisome proliferator-activated receptor alpha-regulated gene expression in rat Heart.
- Clock genes in the Heart: characterization and attenuation with hypertrophy.
- Nitric oxide production in peritoneal macrophages from peritoneal dialysis patients with bacterial peritonitis.
- Interleukin-18 enhances lipopolysaccharide-induced interferon-gamma production in human whole blood cultures.
- New polyether sulfone dialyzers attenuate passage of cytokine-inducing substances from pseudomonas aeruginosa contaminated dialysate.
- Influence of uremia on cell viability and cytokine release of human peritoneal mesothelial cells.
Procedures Preformed
- Automatic Implantable Cardioverter Defibrillator (ICD) Implantation
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac Myocardial Perfusion Imaging
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Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Dissection
- Aortic Ectasia
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Doctors Specialties
- Cardiology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Assurant Health
- Bakery & Confectionary Union Plan
- Blue Card PPO
- Blue Choice
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Commercial Insurance Company
- Coventry Health Care
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- Preferred PPO
- Mail Handlers Benefit Plan (MHBP)
- Medicare MCR
- Molina
- MultiPlan PPO
- PHCS Network PPO
- National Elevator
- Old Surety Life Insurance CompanyMedicare MCR
- Thrivent Financial
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
Awards
- CMS Meaningful Use Stage 1 Certification
- Alpha Omega Alpha
- Fellow (FACC)
Education
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University of Texas Health Science Center -
Heinrich Heine University of Dusseldorf
Drug Facts
| NPI NUMBER | 1316116981 | |
| NPPES Provider LastName | RAZEGHI | |
| NPPES Provider FirstName | PETER | |
| NPPES Provider ZIPCode | 774295884 | |
| NPPES Provider State | TX | |
| Specialty Description | Cardiology | |
| Total Claim Count | 6703.0 | |
| Distinct Opioid Count | 2.0 | |
| Opioid Claim Count | 137.0 | |
| Percent Opioid Claims | 2.04 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1316116981 |
| Last Name Of The Provider | RAZEGHI |
| First Name Of The Provider | PETER |
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