Associate Professor, Pediatrics, University of Cincinnati College of Medicine

Neonat/Perinatology

Dr. Paul S. Kingma, MD

Paul Kingma works in the field of Neonat/Perinatology. He went to Vanderbilt University School of Medicine. Paul Kingma has written publications. He has 24 publications published with PubMed. The lastest paper is: 'The response of eukaryotic topoisomerases to DNA damage.'

Publications

Neutrophil CD64 as a Diagnostic Marker of Sepsis: Impact on Neonatal Care.
Fetal lung growth represented by longitudinal changes in MRI-derived fetal lung volume parameters predicts survival in isolated left-sided congenital diaphragmatic her...
Octreotide for chylous effusions in congenital diaphragmatic hernia.
The congenital diaphragmatic hernia composite prognostic index correlates with survival in left-sided congenital diaphragmatic hernia.
Is Premedication for Intubation of Preterm Infants the Right Choice?
Late gestation fetal magnetic resonance imaging-derived total lung volume predicts postnatal survival and need for extracorporeal membrane oxygenation support in isola...
Pulmonary maturational arrest and death in a patient with pulmonary interstitial glycogenosis.
Surfactant Protein D Deficiency Increases Lung Injury during Endotoxemia.
The pulmonary collectins and respiratory syncytial virus: is there a clinical link?
Correction of pulmonary abnormalities in Sftpd-/- mice requires the collagenous domain of surfactant protein D.
Multimerization of surfactant protein D, but not its collagen domain, is required for antiviral and opsonic activities related to influenza virus.
Communication between the ATPase and cleavage/religation domains of human topoisomerase IIalpha.
Prenatal pulmonary hypertension index: novel prenatal predictor of severe postnatal pulmonary artery hypertension in antenatally diagnosed congenital diaphragmatic her...
Binding of etoposide to topoisomerase II in the absence of DNA: decreased affinity as a mechanism of drug resistance.
The response of eukaryotic topoisomerases to DNA damage.
Topoisomerase II-mediated DNA cleavage and religation in the absence of base pairing. Abasic lesions as a tool to dissect enzyme mechanism.
Spontaneous DNA lesions poison human topoisomerase IIalpha and stimulate cleavage proximal to leukemic 11q23 chromosomal breakpoints.
Spontaneous DNA damage stimulates topoisomerase II-mediated DNA cleavage.
Apurinic sites are position-specific topoisomerase II poisons.
Topoisomerase II.etoposide interactions direct the formation of drug-induced enzyme-DNA cleavage complexes.
Purification of DNA topoisomerase II from Drosophila melanogaster.
Topoisomerase II-catalyzed ATP hydrolysis as monitored by thin-layer chromatography.
In defense of the lung: surfactant protein A and surfactant protein D.

Schools

Vanderbilt University
Childrens Hospital Med Center

Doctors Specialties

Neonatal Medicine
Neonat/Perinatology

Accepted Insurances

Education

Cincinnati Children's Hospital Medical Center
Vanderbilt University School of Medicine

Hospital

Cincinnati Children's Hospital Medical Center

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