Assistant Professor, Vascular Surgery
Vascular Surgery
Dr. Paul R. Crisostomo, MD
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Paul Crisostomo is an expert in the field of Vascular Surgery. He studied medicine at Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. He received awards:"CMS Meaningful Use Stage 1 Certification". Paul Crisostomo is a published doctor and has 70 publications published. The lastest was: 'Period frequency of iliofemoral venous occlusive disease by Doppler ultrasound and corresponding treatment in a tertiary care facility.' Doctor Paul R. Crisostomo, MD accepts Medicare.
Publications
- The effects of endogenous sex hormones and acute hypoxia on vasoconstriction in isolated rat pulmonary artery rings.
- Proestrus female rats are more resistant to right ventricular pressure overload.
- Deleterious effects of endogenous and exogenous testosterone on mesenchymal stem cell VEGF production.
- Neonatal stem cells exhibit specific characteristics in function, proliferation, and cellular signaling that distinguish them from their adult counterparts.
- Nitric oxide suppresses the secretion of Vascular endothelial growth factor and hepatocyte growth factor from human mesenchymal stem cells.
- Surgically relevant aspects of stem cell paracrine effects.
- Exogenous estrogen rapidly attenuates pulmonary artery vasoreactivity and acute hypoxic pulmonary vasoconstriction.
- Sex steroids and stem cell function.
- Estradiol-treated mesenchymal stem cells improve myocardial recovery after ischemia.
- The effects of estrogen on pulmonary artery vasoreactivity and hypoxic pulmonary vasoconstriction: potential new clinical implications for an old hormone.
- Vascular endothelial growth factor improves myocardial functional recovery following ischemia/reperfusion injury.
- Differential IL-6 and VEGF secretion in adult and neonatal mesenchymal stem cells: role of NFkB.
- beta-Blockers in sepsis: reexamining the evidence.
- Estrogen receptor beta mediates acute myocardial protection following ischemia.
- TNFR1 signaling resistance associated with female stem cell cytokine production is independent of TNFR2-mediated pathways.
- TGF-alpha increases human mesenchymal stem cell-secreted VEGF by MEK- and PI3-K- but not JNK- or ERK-dependent mechanisms.
- Stem cell therapy in myocardial repair and remodeling.
- Embryonic stem cells attenuate myocardial dysfunction and inflammation after surgical global ischemia via paracrine actions.
- Angiopoietin-1 in the treatment of ischemia and sepsis.
- Are neonatal stem cells as effective as adult stem cells in providing ischemic protection?
- Mechanisms of sex differences in TNFR2-mediated cardioprotection.
- Selective estrogen receptor-alpha and estrogen receptor-beta agonists rapidly decrease pulmonary artery vasoconstriction by a nitric oxide-dependent mechanism.
- VEGF is critical for stem cell-mediated cardioprotection and a crucial paracrine factor for defining the age threshold in adult and neonatal stem cell function.
- Left ventricular rupture: immediately before and after.
- Stem cells as a potential future treatment of pediatric intestinal disorders.
- TNF receptor 2, not TNF receptor 1, enhances mesenchymal stem cell-mediated cardiac protection following acute ischemia.
- Toll-like receptor signaling pathways and the evidence linking toll-like receptor signaling to cardiac ischemia/reperfusion injury.
- Period frequency of iliofemoral venous occlusive Disease by Doppler ultrasound and corresponding treatment in a tertiary care facility.
- Prevalence of cognitive impairment in an urban Hispanic community population.
- Validating the time and change test to screen for dementia in an older Hispanic population.
- Does endogenous testosterone mediate the lower preconditioning threshold in males?
- Zaprinast attenuates hypoxic pulmonary artery injury and causes less aortic relaxation than milrinone.
- Estrogen receptor-alpha mediates acute myocardial protection in females.
- Brief exposure to exogenous testosterone increases death signaling and adversely affects myocardial function after ischemia.
- p38 mitogen-activated protein kinase mediates the sustained phase of hypoxic pulmonary vasoconstriction and plays a role in phase I vasodilation.
- Experimental therapies for hypoxia-induced pulmonary hypertension during acute Lung injury.
- Postconditioning in females depends on injury severity.
- Preconditioning versus postconditioning: mechanisms and therapeutic potentials.
- Cytokines in necrotizing enterocolitis.
- Pretreatment with adult progenitor cells improves recovery and decreases native myocardial proinflammatory signaling after ischemia.
- Therapeutic concepts for hypoxic pulmonary vasoconstriction involving ion regulation and the smooth muscle contractile apparatus.
- Human progenitor cells from bone marrow or adipose tissue produce VEGF, HGF, and IGF-I in response to TNF by a p38 MAPK-dependent mechanism.
- Tumor necrosis factor receptor 1 signaling resistance in the female myocardium during ischemia.
- Jak/STAT/SOCS signaling circuits and associated cytokine-mediated inflammation and hypertrophy in the heart.
- Gender differences in injury induced mesenchymal stem cell apoptosis and VEGF, TNF, IL-6 expression: role of the 55 kDa TNF receptor (TNFR1).
- Deficiency of TNFR1 protects myocardium through SOCS3 and IL-6 but not p38 MAPK or IL-1beta.
- Sex dimorphisms in activated mesenchymal stem cell function.
- High passage number of stem cells adversely affects stem cell activation and myocardial protection.
- Selective protein kinase C inhibition attenuates pulmonary artery cytokine expression without affecting hypoxic pulmonary vasoconstriction.
- Iron chelation acutely stimulates fetal human intestinal cell production of IL-6 and VEGF while decreasing HGF: the roles of p38, ERK, and JNK MAPK signaling.
- The struggle for iron: gastrointestinal microbes modulate the host immune response during infection.
- STAT3 mediates bone marrow mesenchymal stem cell VEGF production.
- The critical role of vascular endothelial growth factor in pulmonary vascular remodeling after Lung injury.
- Ethyl pyruvate inhibits hypoxic pulmonary vasoconstriction and attenuates pulmonary artery cytokine expression.
- Stem cell mechanisms and paracrine effects: potential in cardiac surgery.
- Stem cells improve right ventricular functional recovery after acute pressure overload and ischemia reperfusion injury.
- Sex differences in endothelial STAT3 mediate sex differences in myocardial inflammation.
- Endogenous estrogen attenuates pulmonary artery vasoreactivity and acute hypoxic pulmonary vasoconstriction: the effects of sex and menstrual cycle.
- The effect of chronic exogenous androgen on myocardial function following acute ischemia-reperfusion in hosts with different baseline levels of sex steroids.
- Activation of individual tumor necrosis factor receptors differentially affects stem cell growth factor and cytokine production.
- Postischemic infusion of 17-beta-estradiol protects myocardial function and viability.
- Endothelial STAT3 plays a critical role in generalized myocardial proinflammatory and proapoptotic signaling.
- In the adult mesenchymal stem cell population, source gender is a biologically relevant aspect of protective power.
- Right ventricular TNF resistance during endotoxemia: the differential effects on ventricular function.
- Mesenchymal stem cells attenuate hypoxic pulmonary vasoconstriction by a paracrine mechanism.
- The right heart and its distinct mechanisms of development, function, and failure.
- Stem cell delivery to the heart: clarifying methodology and mechanism.
- Females exhibit relative resistance to depressive effects of tumor necrosis factor-alpha on the myocardium.
- Human mesenchymal stem cells stimulated by TNF-alpha, LPS, or hypoxia produce growth factors by an NF kappa B- but not JNK-dependent mechanism.
Schools
Northwestern Center Feinberg School of Medicine
Procedures Preformed
- Arteriovenous Shunt Creation
- Dressing and-or Debridement of Wound, Infection, or Burn (incl. Negative Pressure Wound Therapy)
- Peripheral Artery Bypass Procedures
- Peripheral Artery Catheterization
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Conditions Treated
- Abdominal Pain
- Atelectasis
- Collapsed Lung (Pneumothorax)
- Hyperparathyroidism
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Doctors Specialties
- General Surgery
- Vascular Surgery
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- Blue Choice
- Blue Cross Blue Shield of Illinois PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- LocalPlus
- Open Access Plus
- Choice Care PPO
- Preferred PPO
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Indiana University School of Medicine -
Northwestern University Feinberg School of Medicine
Drug Facts
| NPI NUMBER | 1851589212 | |
| NPPES Provider LastName | CRISOSTOMO | |
| NPPES Provider FirstName | PAUL | |
| NPPES Provider ZIPCode | 601533328 | |
| NPPES Provider State | IL | |
| Specialty Description | General Surgery | |
| Total Claim Count | 30.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 14.0 | |
| Percent Opioid Claims | 46.67 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1851589212 |
| Last Name Of The Provider | CRISOSTOMO |
| First Name Of The Provider | PAUL |
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