University of Colorado

Neonat/Perinatology

Paul Rozance works in the field of Neonat/Perinatology. He went to Georgetown University School of Medicine. Georgetown University School of Medicine is ranked 49 in research and 84 in primary care medicine Paul Rozance is a published doctor as well. He has 38 publications published. The latest was: Increased fetal myocardial sensitivity to insulin-stimulated glucose metabolism during ovine fetal growth restriction.

Publications

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• A Sweet Addition for the Treatment of Neonatal Hypoglycemia.
• Increased fetal myocardial sensitivity to insulin-stimulated glucose metabolism during ovine fetal growth restriction.
• Increased adrenergic signaling is responsible for decreased glucose-stimulated insulin secretion in the chronically hyperinsulinemic ovine fetus.
• Coordinated changes in hepatic amino acid metabolism and endocrine signals support hepatic glucose production during fetal hypoglycemia.
• Placental Insufficiency Decreases Pancreatic Vascularity and Disrupts Hepatocyte Growth Factor Signaling in the Pancreatic Islet Endothelial Cell in Fetal Sheep.
• 50 years ago in the journal of pediatrics: the incidence of neonatal hypoglycemia in a nursery for premature infants.
• Thoracic and abdominal aortas stiffen through unique extracellular matrix changes in intrauterine growth restricted (IUGR) fetal sheep.
• Update on neonatal hypoglycemia.
• Increased amino acid supply potentiates glucose-stimulated insulin secretion but does not increase ?-cell mass in fetal sheep.
• Increased arterial stiffness and extracellular matrix reorganization in intrauterine growth-restricted fetal sheep.
• Hyperelastic remodeling in the intrauterine growth restricted (IUGR) carotid artery in the near-term fetus.
• Increased fetal insulin concentrations for one week fail to improve insulin secretion or ?-cell mass in fetal sheep with chronically reduced glucose supply.
• Increased hepatic glucose production in fetal sheep with intrauterine growth restriction is not suppressed by insulin.
• Reductions in Insulin Concentrations and ?-Cell Mass Precede Growth Restriction in Sheep Fetuses with Placental Insufficiency.
• Changes in fetal mannose and other carbohydrates induced by a maternal insulin infusion in pregnant sheep.
• A physiological increase in insulin suppresses muscle-specific ubiquitin ligase gene activation in fetal sheep with sustained hypoglycemia.
• Intrauterine growth restriction decreases pulmonary alveolar and vessel growth and causes pulmonary artery endothelial cell dysfunction in vitro in fetal sheep.
• The intrauterine growth restriction phenotype: fetal adaptations and potential implications for later life insulin resistance and diabetes.
• Glucose metabolism in the preterm infant.
• Characterization of glucose-insulin responsiveness and impact of fetal number and sex difference on insulin response in the sheep fetus.
• Maternal amino acid supplementation for intrauterine growth restriction.
• Describing hypoglycemia--definition or operational threshold?
• Continuous glucose monitoring for diagnosis and treatment of neonatal hypoglycemia.
• Consequences of a compromised intrauterine environment on islet function.
• Prolonged maternal amino acid infusion in late-gestation pregnant sheep increases fetal amino acid oxidation.
• Intrauterine growth restriction increases fetal hepatic gluconeogenic capacity and reduces messenger ribonucleic acid translation initiation and nutrient sensing in fe...
• Effects of chronic hypoglycemia and euglycemic correction on lysine metabolism in fetal sheep.
• Insulin is required for amino acid stimulation of dual pathways for translational control in skeletal muscle in the late-gestation ovine fetus.
• Glucose replacement to euglycemia causes hypoxia, acidosis, and decreased insulin secretion in fetal sheep with intrauterine growth restriction.
• An animal model of placental insufficiency-induced intrauterine growth restriction.
• Chronic late-gestation hypoglycemia upregulates hepatic PEPCK associated with increased PGC1alpha mRNA and phosphorylated CREB in fetal sheep.
• Increased insulin sensitivity and maintenance of glucose utilization rates in fetal sheep with placental insufficiency and intrauterine growth restriction.
• Chronic fetal hypoglycemia inhibits the later steps of stimulus-secretion coupling in pancreatic beta-cells.
• Decreased nutrient-stimulated insulin secretion in chronically hypoglycemic late-gestation fetal sheep is due to an intrinsic islet defect.
• Hypoglycemia in newborn infants: Features associated with adverse outcomes.
• Attenuated insulin release and storage in fetal sheep pancreatic islets with intrauterine growth restriction.
• Rapid deterioration of a newborn with congenital spinal cord astrocytoma.

Schools

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• Georgetown University School Of Medicine

• Stanford University Program

• University Of Colorado

• University Of Californiaberkeley

Doctors Specialties

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• Neonatal Medicine
• Neonat/Perinatology

Accepted Insurances

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Education

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• University of Colorado
• Stanford University
• Georgetown University School of Medicine

Hospital

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• University of Colorado Hospital
• Children's Hospital Colorado

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