Professor, Medicine, UCLA School of Medicine
Infectious Disease
Dr. Otto O. Yang, MD
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Otto O. Yang is an expert in the field of Infectious Disease. He went to NYU School of Medicine. He has 1 awards "CMS Meaningful Use Stage 1 Certification". Dr. Otto O. Yang is a published MD. He published 84 publications, including: 'Supranormal thymic output up to 2 decades after HIV-1 infection.' He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Short Conserved Sequences of HIV-1 Are Highly Immunogenic and Shift Immunodominance.
- Generation of β cell-specific Human Cytotoxic T Cells by Lentiviral Transduction and Their Survival in Immunodeficient HLA-transgenic Mice.
- Immunomodulation of Antiretroviral Drug-Suppressed Chronic HIV-1 Infection in an Oral Probiotic Double-Blind Placebo-Controlled Trial.
- Seminal Plasma HIV-1 RNA Concentration is Strongly Associated with Altered Levels of Seminal Plasma IFN-γ, IL-17, and IL-5.
- Ineffectual Targeting of HIV-1 Nef by Cytotoxic T Lymphocytes in Acute Infection Results in No Functional Impairment or Viremia Reduction.
- HIV-1 gag Genetic Variation in a Single Acutely Infected Participant Defined by High-Resolution Deep Sequencing.
- Engineering the human thymic microenvironment to support thymopoiesis in vivo.
- HDL redox activity is increased in HIV-infected men in association with macrophage activation and noncalcified coronary atherosclerotic plaque.
- Long-term human immune system reconstitution in non-obese diabetic (NOD)-Rag (-)-γ chain (-) (NRG) mice is similar but not identical to the original stem cell donor.
- Antigen Presenting Cell Candidates for HIV-1 Transmission in Human Distal Colonic Mucosa Defined By CD207 Dendritic Cells and CD209 Macrophages.
- HIV-1 Gag Cytotoxic T Lymphocyte Epitopes Vary in Presentation Kinetics Relative to HLA Class I Downregulation.
- Untrained Young Men Have Dysfunctional HDL Compared to Strength Trained Men Irrespective of Overweight/Obesity Status.
- Introduction of Exogenous T-cell Receptors Into Human Hematopoietic Progenitors Results in Exclusion of Endogenous T-cell Receptor Expression.
- Combination of allele-specific detection techniques to quantify minority resistance variants in hepatitis B infection: A novel approach.
- Biomimetic enzyme nanocomplexes and their use as antidotes and preventive measures for alcohol intoxication.
- Perturbations of circulating levels of RANKL-Osteoprotegerin Axis in Relation to Lipids and Progression of Atherosclerosis in HIV-Infected and -Uninfected Adults: ACTG...
- Biomarkers of microbial translocation and macrophage activation: association with progression of subclinical atherosclerosis in HIV-1 infection.
- Preferential depletion of gut CD4-expressing iNKT cells contributes to systemic immune activation in HIV-1 infection.
- Early HLA-B*57-Restricted CD8+ T Lymphocyte Responses Predict HIV-1 Disease Progression.
- Effects of lipid-probe interactions in biochemical fluorometric methods that assess HDL redox activity.
- Immune selection in vitro reveals human immunodeficiency virus type 1 Nef sequence motifs important for its immune evasion function in vivo.
- Partial Escape of HIV-1 from Cytotoxic T Lymphocytes during Chronic Infection.
- Epitope targeting and viral inoculum are determinants of Nef-mediated immune evasion of HIV-1 from CTLs.
- Hydrodynamic stretching of single cells for large population mechanical phenotyping.
- Definition of the viral targets of protective HIV-1-specific T cell responses.
- A Biochemical Fluorometric Method for Assessing the Oxidative Properties of HDL.
- Functional Analysis of HIV-1 Nef Gene Variants from Adolescent and Adult Survivors of Perinatal Infection.
- Combination therapy with daptomycin, linezolid, and rifampin as treatment option for MRSA meningitis and bacteremia.
- Human leukocyte antigen class I haplotypes of human immunodeficiency virus-1-infected persons on Likoma Island, Malawi.
- Increasing CTL targeting of conserved sequences during early HIV-1 infection is correlated to decreasing viremia.
- Antiviral activity of human immunodeficiency virus type 1 Gag-specific cytotoxic T lymphocyte targeting is not necessarily intrinsically superior to envelope targeting.
- Rapid T cell receptor delineation reveals clonal expansion limitation of the magnitude of the HIV-1-specific CD8+ T cell response.
- Interleukin-2-unresponsive immune defects in good syndrome: Letter to the Editor.
- Protection against bronchiolitis obliterans syndrome is associated with allograft CCR7+ CD45RA- T regulatory cells.
- Predicting the impact of blocking human immunodeficiency virus type 1 Nef in vivo.
- Assessing the antiviral activity of HIV-1-specific cytotoxic T lymphocytes.
- Candidate vaccine sequences to represent intra- and inter-clade HIV-1 variation.
- Telomerase-based pharmacologic enhancement of antiviral function of human CD8+ T lymphocytes.
- Proliferation and foxp3 expression in virus-specific memory CD8+ T lymphocytes.
- Bronchoalveolar immunologic profile of acute human lung transplant allograft rejection.
- Functional adaptation of Nef to the immune milieu of HIV-1 infection in vivo.
- Primary human immunodeficiency virus type 1 (HIV-1) infection during HIV-1 Gag vaccination.
- Cross-clade detection of HIV-1-specific cytotoxic T lymphocytes does not reflect cross-clade antiviral activity.
- Delayed reconstitution of CD4+ iNKT cells after effective HIV type 1 therapy.
- Availability of a diversely avid CD8+ T cell repertoire specific for the subdominant HLA-A2-restricted HIV-1 Gag p2419-27 epitope.
- Pediatric HIV-1-specific cytotoxic T-lymphocyte responses suggesting ongoing viral replication despite combination antiretroviral therapy.
- Epitope-dependent avidity thresholds for cytotoxic T-lymphocyte clearance of virus-infected cells.
- Paradoxical effects of two theta-defensins on HIV type 1 infection.
- Gag-specific CD8+ T lymphocytes recognize infected cells before AIDS-virus integration and viral protein expression.
- Nef interference with HIV-1-specific CTL antiviral activity is epitope specific.
- gp120-independent HIV infection of cells derived from the female reproductive tract, brain, and colon.
- Degeneracy and repertoire of the human HIV-1 Gag p17(77-85) CTL response.
- HIV-1 adapts to a retrocyclin with cationic amino acid substitutions that reduce fusion efficiency of gp41.
- Genetic and stochastic influences on the interaction of human immunodeficiency virus type 1 and cytotoxic T lymphocytes in identical twins.
- How many human immunodeficiency virus type 1-infected target cells can a cytotoxic T-lymphocyte kill?
- Effects of HIV-1 infection on lymphocyte phenotypes in blood versus lymph nodes.
- Parallel human immunodeficiency virus type 1-specific CD8+ T-lymphocyte responses in blood and mucosa during chronic infection.
- Decreased perforin and granzyme B expression in senescent HIV-1-specific cytotoxic T lymphocytes.
- Human immunodeficiency virus type 1 clade B superinfection: evidence for differential immune containment of distinct clade B strains.
- Ethanol stimulation of HIV infection of oral epithelial cells.
- Culturing of HIV-1-specific cytotoxic T lymphocytes with interleukin-7 and interleukin-15.
- Beyond help: direct effector functions of human immunodeficiency virus type 1-specific CD4(+) T cells.
- Impacts of epitope expression kinetics and class I downregulation on the antiviral activity of human immunodeficiency virus type 1-specific cytotoxic T lymphocytes.
- Epitope escape mutation and decay of human immunodeficiency virus type 1-specific CTL responses.
- Broadly increased sensitivity to cytotoxic T lymphocytes resulting from Nef epitope escape mutations.
- Impacts of avidity and specificity on the antiviral efficiency of HIV-1-specific CTL.
- The theta-defensin, retrocyclin, inhibits HIV-1 entry.
- Determinant of HIV-1 mutational escape from cytotoxic T lymphocytes.
- Differential impairment of lytic and cytokine functions in senescent human immunodeficiency virus type 1-specific cytotoxic T lymphocytes.
- Chimeric immune receptor T cells bypass class I requirements and recognize multiple cell types relevant in HIV-1 infection.
- TCR gamma delta+ and CD161+ thymocytes express HIV-1 in the SCID-hu mouse, potentially contributing to immune dysfunction in HIV infection.
- Immunologic profile of highly exposed yet HIV type 1-seronegative men.
- gp120-Independent infection of CD4(-) epithelial cells and CD4(+) T-cells by HIV-1.
- Monocyte chemoattractant protein-2 (CC chemokine ligand 8) inhibits replication of human immunodeficiency virus type 1 via CC chemokine receptor 5.
- Retrocyclin: a primate peptide that protects cells from infection by T- and M-tropic strains of HIV-1.
- Nef-mediated resistance of human immunodeficiency virus type 1 to antiviral cytotoxic T lymphocytes.
- A High Throughput Biochemical Fluorometric Method for Measuring Lipid Peroxidation in HDL.
- HIV-1 Quasispecies Delineation by Tag Linkage Deep Sequencing.
- Differential Blood and Mucosal Immune Responses against an HIV-1 Vaccine Administered via Inguinal or Deltoid Injection.
- Natural killer T cells in advanced melanoma patients treated with tremelimumab.
- A Stochastic Multi-Scale Model of HIV-1 Transmission for Decision-Making: Application to a MSM Population.
- HIV-1 Nef Sequence and Functional Compartmentalization in the Gut Is Not Due to Differential Cytotoxic T Lymphocyte Selective Pressure.
- Supranormal thymic output up to 2 decades after HIV-1 infection.
Schools
The Warren Alpert Medical School Of Brown University
New York University
Massachusetts General Hospital
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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Massachusetts General Hospital -
NYU School of Medicine -
Warren Alpert Medical School of Brown University
Hospital
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Ronald Reagan UCLA Medical Center -
Santa Monica-UCLA Medical Center and Orthopaedic Hospital
Drug Facts
| NPI NUMBER | 1780604298 | |
| NPPES Provider LastName | YANG | |
| NPPES Provider FirstName | OTTO | |
| NPPES Provider ZIPCode | 900953075 | |
| NPPES Provider State | CA | |
| Specialty Description | Infectious Disease | |
| Total Claim Count | 64.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1780604298 |
| Last Name Of The Provider | YANG |
| First Name Of The Provider | OTTO |
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