Pathology at Harvard
Pathology
Dr. Omer H. Yilmaz, MD
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Omer Yilmaz practices Pathology. He went to University of Michigan Medical School. Omer H. Yilmaz, MD is a published doctor. He published 27 publications, including: 'Colorectal cancer models for novel drug discovery.' He accepts Medicare.
Publications
- Colorectal cancer models for novel drug discovery.
- Depletion of Rictor, an essential protein component of mTORC2, decreases male lifespan.
- Atg16L1 T300A variant decreases selective autophagy resulting in altered cytokine signaling and decreased antibacterial defense.
- Dietary and Metabolic Control of Stem Cell Function in Physiology and Cancer.
- Combination PI3K/MEK inhibition promotes tumor apoptosis and regression in PIK3CA wild-type, KRAS mutant colorectal cancer.
- ZFHX4 Interacts with the NuRD Core Member CHD4 and Regulates the Glioblastoma Tumor-Initiating Cell State.
- Atg16l1 is Required for Autophagy in Intestinal Epithelial Cells and Protection of Mice From Salmonella Infection.
- Abatacept: A New Treatment Option for Refractory Adult Autoimmune Enteropathy.
- Pten-Null Tumors Cohabiting the Same Lung Display Differential AKT Activation and Sensitivity to Dietary Restriction.
- Cables1 is a tumor suppressor gene that regulates intestinal tumor progression in Apc (Min) mice.
- Peutz-Jeghers syndrome: a critical look at colonic Peutz-Jeghers polyps.
- MCT1-mediated transport of a toxic molecule is an effective strategy for targeting glycolytic tumors.
- Heterogeneity in signaling pathways of gastroenteropancreatic neuroendocrine tumors: a critical look at notch signaling pathway.
- Cystic papillary pattern in pancreatic ductal adenocarcinoma: a heretofore undescribed morphologic pattern that mimics intraductal papillary mucinous carcinoma.
- mTOR activation induces tumor suppressors that inhibit leukemogenesis and deplete hematopoietic stem cells after Pten deletion.
- Persistence of leukemia-initiating cells in a conditional knockin model of an imatinib-responsive myeloproliferative disorder.
- Aldehyde dehydrogenase 1a1 is dispensable for stem cell function in the mouse hematopoietic and nervous systems.
- The PI-3kinase pathway in hematopoietic stem cells and leukemia-initiating cells: a mechanistic difference between normal and cancer stem cells.
- CD150- cells are transiently reconstituting multipotent progenitors with little or no stem cell activity.
- Enhanced purification of fetal liver hematopoietic stem cells using SLAM family receptors.
- SLAM family markers are conserved among hematopoietic stem cells from old and reconstituted mice and markedly increase their purity.
- CD144 (VE-cadherin) is transiently expressed by fetal liver hematopoietic stem cells.
- Spatial differences in hematopoiesis but not in stem cells indicate a lack of regional patterning in definitive hematopoietic stem cells.
- SLAM family receptors distinguish hematopoietic stem and progenitor cells and reveal endothelial niches for stem cells.
- Intestinal Colonization by Candida albicans Alters Inflammatory Responses in Bruton's Tyrosine Kinase-Deficient Mice.
- Pten dependence distinguishes haematopoietic stem cells from leukaemia-initiating cells.
Schools
University of Mi Med Sch
Doctors Specialties
- Pathology
Accepted Insurances
Education
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Massachusetts General Hospital -
University of Michigan Medical School
Hospital
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Massachusetts General Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1417116864 |
| Last Name Of The Provider | YILMAZ |
| First Name Of The Provider | OMER |
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