Associate Professor, Medicine, University of Colorado School of Medicine
Endocrinology
Dr. Neda Rasouli, MD
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Neda Rasouli is an expert in the field of Endocrinology. She attended University of Arkansas for Medical Sciences. University of Arkansas for Medical Sciences is ranked 83/35 in Research/PrimaryCare. She received awards:"CMS Meaningful Use Stage 1 Certification" and "Most Compassionate Doctor". Neda Rasouli is a published doctor and has 50 publications published. The lastest was: 'Led Astray by Hemoglobin A1c: A Case of Misdiagnosis of Diabetes by Falsely Elevated Hemoglobin A1c.' Neda Rasouli accepts all Medicare patients.
Publications
- Integrative mRNA-microRNA analyses reveal novel interactions related to insulin sensitivity in human adipose tissue.
- The triglyceride to high-density lipoprotein cholesterol (TG/HDL-C) ratio as a predictor of β-cell function in African American women.
- Glycemic control and outcomes of hospitalization in noncritically ill patients with type 2 Diabetes admitted with cardiac problems or infections.
- The Effects of Temperature and Seasons on Subcutaneous White Adipose Tissue in Humans: Evidence for Thermogenic Gene Induction.
- Transitional care clinic for uninsured and medicaid-covered patients with Diabetes mellitus discharged from the hospital: a pilot quality improvement study.
- Effect of Glargine Insulin Delivery Method (Pen-Device vs. Vial-Syringe) on Glycemic Control and Patient Preferences in Patients with Type 1 and Type 2 Diabetes.
- Pathways to quality inpatient management of hyperglycemia and Diabetes: a call to action.
- An ACACB Variant Implicated in Diabetic Nephropathy Associates With Body Mass Index and Gene Expression in Obese Subjects.
- Cystic Fibrosis-Related Diabetes in Adults: Inpatient Management of 121 Patients during 410 Admissions.
- Subcutaneous Administration of Glargine to Diabetic Patients Receiving Insulin Infusion Prevents Rebound Hyperglycemia.
- Led Astray by Hemoglobin A1c: A Case of Misdiagnosis of Diabetes by Falsely Elevated Hemoglobin A1c.
- Pioglitazone Treatment Reduces Adipose Tissue Inflammation through Reduction of Mast Cell and Macrophage Number and by Improving Vascularity.
- Comparison of 70/30 biphasic insulin With glargine/lispro regimen in non-critically ill diabetic patients on continuous enteral nutrition therapy.
- Adipose tissue extracellular matrix and vascular Abnormalities in obesity and insulin resistance.
- Global gene expression profiles of subcutaneous adipose and muscle from glucose-tolerant, insulin-sensitive, and insulin-resistant individuals matched for BMI.
- Adipose triglyceride lipase expression in human adipose tissue and muscle. Role in insulin resistance and response to training and pioglitazone.
- Adipose tissue macrophages in insulin-resistant subjects are associated With collagen VI and fibrosis and demonstrate alternative activation.
- Insulin resistance in African-American and Caucasian women: differences in lipotoxicity, adipokines, and gene expression in adipose tissue and muscle.
- Matrix metalloproteinase-9 is increased in obese subjects and decreases in response to pioglitazone.
- Association of scavenger receptors in adipose tissue With insulin resistance in nondiabetic humans.
- Muscle inflammatory response and insulin resistance: synergistic interaction between macrophages and fatty acids leads to impaired insulin action.
- Adipocytokines and the metabolic complications of Obesity.
- Endoplasmic reticulum stress markers are associated With obesity in nondiabetic subjects.
- Stearoyl-coenzyme A desaturase 1 gene expression increases after pioglitazone treatment and is associated With peroxisomal proliferator-activated receptor-gamma respon...
- Effect of pioglitazone treatment on endoplasmic reticulum stress response in human adipose and in palmitate-induced stress in human liver and adipose cell lines.
- Intermuscular Lipid: a marker of disordered fat partitioning or the consequence of obesity?
- Calcium is involved in formation of high molecular weight adiponectin.
- A review of thiazolidinediones and metformin in the treatment of type 2 Diabetes with focus on cardiovascular complications.
- Thrombospondin-1 is an adipokine associated With obesity, adipose inflammation, and insulin resistance.
- Impact of family history of Diabetes and ethnicity on -cell function in obese, glucose-tolerant individuals.
- Retinol binding protein 4 expression in humans: relationship to insulin resistance, inflammation, and response to pioglitazone.
- Transcription factor 7-like 2 polymorphisms and type 2 Diabetes, glucose homeostasis traits and gene expression in US participants of European and African descent.
- Ectopic fat accumulation and metabolic Syndrome.
- OXPAT/PAT-1 is a PPAR-induced Lipid droplet protein that promotes fatty acid utilization.
- Human visfatin expression: relationship to insulin sensitivity, intramyocellular Lipids, and inflammation.
- Lipin expression is attenuated in adipose tissue of insulin-resistant human subjects and increases With peroxisome proliferator-activated receptor gamma activation.
- Effects of pioglitazone and metformin on beta-cell function in nondiabetic subjects at high risk for type 2 Diabetes.
- The prevention and treatment of metabolic Syndrome and high-risk obesity.
- The lipogenic enzymes DGAT1, FAS, and LPL in adipose tissue: effects of Obesity, insulin resistance, and TZD treatment.
- Pioglitazone increases secretion of high-molecular-weight adiponectin from adipocytes.
- Pioglitazone induces apoptosis of macrophages in human adipose tissue.
- Oxidative stress in Diabetes: a mechanistic overview of its effects on atherogenesis and myocardial dysfunction.
- Increased plasma adiponectin in response to pioglitazone does not result from increased gene expression.
- Effects of short-term experimental insulin resistance and family history of Diabetes on pancreatic beta-cell function in nondiabetic individuals.
- Expression of CD68 and macrophage chemoattractant protein-1 genes in human adipose and muscle tissues: association With cytokine expression, insulin resistance, and re...
- Pioglitazone improves insulin sensitivity through reduction in muscle Lipid and redistribution of Lipid into adipose tissue.
- Improved glycemic control in subjects with atypical Diabetes results from restored insulin secretion, but not improved insulin sensitivity.
- Divergent effects of peroxisome proliferator-activated receptor gamma agonists and tumor necrosis factor alpha on adipocyte ApoE expression.
- Peroxisome proliferator-activated receptor ligands as antiatherogenic agents: panacea or another Pandora's box?
Schools
Michigan State University Program
University Of Arkansas For Medical Sciences Program
Conditions Treated
- Abnormal Thyroid
- Adrenal Insufficiency
- Cushing's Syndrome
- Diabetes Mellitus, Secondary
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Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Most Compassionate Doctor
Education
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University of Arkansas for Medical Sciences -
Tehran University of Medical Sciences School of Medicine
Hospital
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University of Colorado Hospital
Drug Facts
| NPI NUMBER | 1336247295 | |
| NPPES Provider LastName | RASOULI | |
| NPPES Provider FirstName | NEDA | |
| NPPES Provider ZIPCode | 800452545 | |
| NPPES Provider State | CO | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 289.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1336247295 |
| Last Name Of The Provider | RASOULI |
| First Name Of The Provider | NEDA |
| View All |
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