Physician

Neurology

Michel Toledano, MD works in the field of Neurology. Michel Toledano is a published doctor. He published 47 publications, including: 'A new antioxidant with alkyl hydroperoxide defense properties in yeast.' He accepts Medicare payments and is listed with Medicare.gov.

Publications

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• Adaptive aneuploidy protects against thiol peroxidase deficiency by increasing respiration via key mitochondrial proteins.
• Teaching NeuroImages: diagnostic utility of FDG-PET in neurolymphomatosis.
• [Are mild ovarian stimulations for IVF-ET a significant progress in assisted reproductive technologies?].
• Dental composites reinforced with hydroxyapatite: mechanical behavior and absorption/elution characteristics.
• Hydrolytic stability of experimental hydroxyapatite-filled dental composite materials.
• Reversal of compromised bonding in bleached enamel.
• Reversal of compromised bonding to oxidized etched dentin.
• Spatially resolved photopolymerization kinetics and oxygen inhibition in dental adhesives.
• Interaction between iron metabolism and 2,3,7,8-tetrachlorodibenzo-p-dioxin in mice with variants of the Ahr gene: a hepatic oxidative mechanism.
• A new antioxidant with alkyl hydroperoxide defense properties in yeast.
• Yap1 and Skn7 control two specialized oxidative stress response regulons in yeast.
• A genetic investigation of the essential role of glutathione: mutations in the proline biosynthesis pathway are the only suppressors of glutathione auxotrophy in yeast.
• The H2O2 stimulon in Saccharomyces cerevisiae.
• A proteome analysis of the cadmium response in Saccharomyces cerevisiae.
• H2O2 sensing through oxidation of the Yap1 transcription factor.
• Yap8p activation in Saccharomyces cerevisiae under arsenic conditions.
• Ionizing radiation induces a Yap1-dependent peroxide stress response in yeast.
• Visualization of ribonucleotide reductase catalytic oxidation establishes thioredoxins as its major reductants in yeast.
• alpha1-Antitrypsin monotherapy induces immune tolerance during islet allograft transplantation in mice.
• Life span extension and H(2)O(2) resistance elicited by caloric restriction require the peroxiredoxin Tsa1 in Saccharomyces cerevisiae.
• Glutathione revisited: a vital function in iron metabolism and ancillary role in thiol-redox control.
• Nitric oxide activates an Nrf2/sulfiredoxin antioxidant pathway in macrophages.
• Sulfiredoxin protects mice from lipopolysaccharide-induced endotoxic shock.
• Proteome screens for Cys residues oxidation: the redoxome.
• Reining in H(2)O(2) for safe signaling.
• Activation of NRF2 by nitrosative agents and H2O2 involves KEAP1 disulfide formation.
• H2O2 activates the nuclear localization of Msn2 and Maf1 through thioredoxins in Saccharomyces cerevisiae.
• The guardian recruits cops: the p53-p21 axis delegates prosurvival duties to the Keap1-Nrf2 stress pathway.
• Dug1p Is a Cys-Gly peptidase of the gamma-glutamyl cycle of Saccharomyces cerevisiae and represents a novel family of Cys-Gly peptidases.
• Protein-thiol oxidation, from single proteins to proteome-wide analyses.
• The dual functions of thiol-based peroxidases in H2O2 scavenging and signaling.
• Regulation of peroxiredoxins by nitric oxide in immunostimulated macrophages.
• ROS as signalling molecules: mechanisms that generate specificity in ROS homeostasis.
• The system biology of thiol redox system in Escherichia coli and yeast: differential functions in oxidative stress, iron metabolism and DNA synthesis.
• Disulfide Bond-mediated multimerization of Ask1 and its reduction by thioredoxin-1 regulate H(2)O(2)-induced c-Jun NH(2)-terminal kinase activation and apoptosis.
• Cytoplasmic glutathione redox status determines survival upon exposure to the thiol-oxidant 4,4''-dipyridyl disulfide.
• The Arabidopsis thaliana sulfiredoxin is a plastidic cysteine-sulfinic acid reductase involved in the photooxidative stress response.
• The S. cerevisiae Yap1 and Yap2 transcription factors share a common cadmium-sensing domain.
• The Saccharomyces cerevisiae proteome of oxidized protein thiols: contrasted functions for the thioredoxin and glutathione pathways.
• A cysteine-sulfinic acid in peroxiredoxin regulates H2O2-sensing by the antioxidant Pap1 pathway.
• Microbial H2O2 sensors as archetypical redox signaling modules.
• Mammalian antioxidant defenses are not inducible by H2O2.
• ATP-dependent reduction of cysteine-sulphinic acid by S. cerevisiae sulphiredoxin.
• Two redox centers within Yap1 for H2O2 and thiol-reactive chemicals signaling.
• A thiol peroxidase is an H2O2 receptor and redox-transducer in gene activation.
• The control of the yeast H2O2 response by the Msn2/4 transcription factors.

Conditions Treated

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• Convulsions
• Epilepsy
• Gait Abnormality
• Headache
• View All

Doctors Specialties

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• Neurology

Accepted Insurances

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Education

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• Mayo School of Graduate Medical Education

Hospital

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• Mayo Clinic - Rochester, Minnesota

Drug Facts

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NPI NUMBER		1649581166
NPPES Provider LastName		TOLEDANO
NPPES Provider FirstName		MICHEL
NPPES Provider ZIPCode		559050001
NPPES Provider State		MN
Specialty Description		Neurology
Total Claim Count		51.0
Distinct Opioid Count		0.0
Opioid Claim Count		0.0
Percent Opioid Claims		0.0

Helpful Reviews

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Medicare Facts

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National Provider Identifier [NPI]	1649581166
Last Name Of The Provider	TOLEDANO
First Name Of The Provider	MICHEL
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