Professor Emeritus
Pathology
Dr. Michael Kashgarian, MD
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Michael Kashgarian works in the field of Pathology. He attended Yale University School of Medicine. Yale University School of Medicine is one of the top medical schools in the US. Yale University School of Medicine is ranked 7/57 in Research/PrimaryCare. He received awards:"Fellow (ASNF)" and "Whos Who in Medicine and Healthcare". Michael Kashgarian is a published doctor. He has 55 publications published. The most current publication "Lupus nephritis: lessons from the path lab." He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Akt Substrate of 160 kD Regulates Na+,K+-ATPase Trafficking in Response to Energy Depletion and Renal Ischemia.
- B Cell-Specific MHC Class II Deletion Reveals Multiple Nonredundant Roles for B Cell Antigen Presentation in Murine Lupus.
- Local Triggering of the ICOS Coreceptor by CD11c(+) Myeloid Cells Drives Organ Inflammation in Lupus.
- Mutation of POLB Causes Lupus in Mice.
- Exacerbated Autoimmunity in the Absence of TLR9 in MRL.Faslpr Mice Depends on Ifnar1.
- Signals via the Adaptor MyD88 in B Cells and DCs Make Distinct and Synergistic Contributions to Immune Activation and Tissue Damage in Lupus.
- An Incompletely Penetrant Novel Mutation in COL7A1 Causes Epidermolysis Bullosa Pruriginosa and Dominant Dystrophic Epidermolysis Bullosa Phenotypes in an Extended Kin...
- NADPH oxidase inhibits the pathogenesis of systemic lupus erythematosus.
- Acute Podocyte Vascular Endothelial Growth Factor (VEGF-A) Knockdown Disrupts alpha(V)beta(3) Integrin Signaling in the Glomerulus.
- The contribution of mesangial cell collagen synthesis to the pathogenesis of diabetic nephropathy.
- Lupus nephritis: lessons from the path lab.
- Benign fibroblastic polyps of the colon: a histologic, immunohistochemical, and ultrastructural study.
- Abrogation of skin disease in LUPUS-prone MRL/FASlpr mice by means of a novel tylophorine analog.
- The contribution of quantitative techniques including morphometry to renal diagnosis.
- Increased Tubular Proliferation as an Adaptive Response to Glomerular Albuminuria.
- Autoantibody responses and pathology regulated by B7-1 and B7-2 costimulation in MRL/lpr lupus.
- ICOS controls effector function but not trafficking receptor expression of kidney-infiltrating effector T cells in murine lupus.
- Hsp27 associates with actin and limits injury in energy depleted renal epithelia.
- A lupus-like syndrome develops in mice lacking the Ro 60-kDa protein, a major lupus autoantigen.
- A small-molecule macrophage migration inhibitory factor antagonist protects against glomerulonephritis in lupus-prone NZB/NZW F1 and MRL/lpr mice.
- Paracellular Cl- permeability is regulated by WNK4 kinase: insight into normal physiology and hypertension.
- Met and the epidermal growth factor receptor act cooperatively to regulate final nephron number and maintain collecting duct morphology.
- Induction of podocyte VEGF164 overexpression at different stages of development causes congenital nephrosis or steroid-resistant nephrotic syndrome.
- B7 costimulation in the development of lupus: autoimmunity arises either in the absence of B7.1/B7.2 or in the presence of anti-b7.1/B7.2 blocking antibodies.
- Preactivation of AMPK by metformin may ameliorate the epithelial cell damage caused by renal ischemia.
- Depletion of B cells in murine lupus: efficacy and resistance.
- Altered renal proximal tubular endocytosis and histology in mice lacking myosin-VI.
- Bone marrow stem cells contribute to repair of the ischemically injured renal tubule.
- Toll-like receptor 9 controls anti-DNA autoantibody production in murine lupus.
- Circumscribed palmar hypokeratosis: two cases and a review of the literature.
- Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damage.
- Toll-like receptor 7 and TLR9 dictate autoantibody specificity and have opposing inflammatory and regulatory roles in a murine model of lupus.
- B cell-derived IL-10 does not regulate spontaneous systemic autoimmunity in MRL.Fas(lpr) mice.
- TLR9 regulates TLR7- and MyD88-dependent autoantibody production and disease in a murine model of lupus.
- Differential inhibition of HSP72 and HSP25 produces profound impairment of cellular integrity.
- A case of an acute kidney injury secondary to an implanted aminoglycoside.
- Functional activation of heat shock factor and hypoxia-inducible factor in the kidney.
- The commonly used beta-actin-GFP transgenic mouse strain develops a distinct type of glomerulosclerosis.
- Modulation of renal cell injury by heat shock proteins: lessons learned from the immature kidney.
- A cluster of metabolic defects caused by mutation in a mitochondrial tRNA.
- Induction, differentiation, and remodeling of blood vessels after transplantation of Bcl-2-transduced endothelial cells.
- Endothelial cell seeding influences lesion development following arterial injury in the cholesterol-fed rabbit.
- The soft triangle revisited.
- Anatomy of the Caucasian alar groove.
- Developmental expression of HSP-72 and ischemic tolerance of the immature kidney.
- Resolution of proteinuria in a patient with focal segmental glomerulosclerosis following BiPAP initiation for obesity hypoventilation syndrome.
- Reduced tolerance of immature renal tubules to anoxia by HSF-1 decoy.
- Betaglycan inhibits TGF-beta signaling by preventing type I-type II receptor complex formation. Glycosaminoglycan modifications alter betaglycan function.
- PECAM-1 modulates thrombin-induced tissue factor expression on endothelial cells.
- A novel form of human mendelian hypertension featuring nonglucocorticoid-remediable aldosteronism.
- Ultrastructural characteristics of rat peritoneal mast cells undergoing differential release of serotonin without histamine and without degranulation.
- Islet transplantation is associated with improvement of renal function among uremic patients with type I diabetes mellitus and kidney transplants.
- Disruption of Myosin 1e promotes podocyte injury.
- Membranoproliferative glomerulonephritis type II (dense deposit disease): an update.
Schools
Yale University
Barnes Jewish Hospital
Yale Med Center
University Goettingen
Doctors Specialties
- Pathology
Accepted Insurances
Awards
- Fellow (ASNF)
- Whos Who in Medicine and Healthcare
Education
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Yale-New Haven Hospital -
Washington University - Barnes-Jewish Hospital -
Yale University School of Medicine
Hospital
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Yale-New Haven Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1538158175 |
| Last Name Of The Provider | KASHGARIAN |
| First Name Of The Provider | MICHAEL |
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