Professor of Medicine, Indiana University
Endocrinology
Dr. Michael J. Econs, MD
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Michael J. Econs works in the field of Endocrinology. He went to University of California San Francisco School of Medicine. University of California San Francisco School of Medicine is a top medical college. University of California San Francisco School of Medicine is ranked 3/3 in Research/PrimaryCare. He received 4 awards: "America's Top Doctors", "Indianapolis Monthly", "Elected Member" and "Fellow (FACP)". Michael J. Econs is also published. He has 82 publications published. The lastest publication: "Serum fibroblast growth factor 23, serum iron and bone mineral density in premenopausal women.' Michael Econs accepts Medicare.
Publications
- Genome-wide association study of serum iron phenoTypes in premenopausal women of European descent.
- Osteoblast-Specific Overexpression of Human WNT16 Increases Both Cortical and Trabecular Bone Mass and Structure in Mice.
- Interferon Gamma, but not Calcitriol Improves the Osteopetrotic PhenoTypes in ADO2 Mice.
- Genetic Rescue of Glycosylation-deficient Fgf23 in the Galnt3 Knockout Mouse.
- Hypophosphatemic Rickets: Revealing Novel Control Points for Phosphate Homeostasis.
- Sibling Family Genes And Bone Mineral Density: Association And Allele-Specific Expression In Humans.
- High-Resolution Genome Screen for Bone Mineral Density in Heterogeneous Stock Rat.
- Iron and fibroblast growth factor 23 in X-linked hypophosphatemia.
- Generation of the first autosomal dominant osteopetrosis Type II (ADO2) disease models.
- Intronic deletions in the SLC34A3 gene: A cautionary tale for mutation analysis of hereditary hypophosphatemic rickets With hypercalciuria.
- Dosage Effect of a Phex Mutation in a Murine Model of X-Linked Hypophosphatemia.
- Meta-analysis of genome-wide studies identifies WNT16 and ESR1 SNPs associated With bone mineral density in premenopausal women.
- regSNPs: a strategy for prioritizing regulatory single nucleotide substitutions.
- Dietary Phosphate Restriction Normalizes Biochemical and Skeletal Abnormalities in a Murine Model of Tumoral Calcinosis.
- A Phex mutation in a murine model of X-linked hypophosphatemia alters phosphate responsiveness of bone cells.
- Iron Modifies Plasma FGF23 Differently in Autosomal Dominant Hypophosphatemic Rickets and Healthy Humans.
- Heterogeneous stock rat: A unique animal model for mapping genes influencing bone fragility.
- Replication of previous genome-wide association studies of bone mineral density in premenopausal American women.
- Establishment of sandwich ELISA for soluble alpha-Klotho measurement: Age-dependent change of soluble alpha-Klotho levels in healthy subjects.
- Identification of genes influencing skeletal phenoTypes in congenic P/NP rats.
- Mosaicism in osteopathia striata With cranial sclerosis.
- Treatment of X-linked hypophosphatemia With calcitriol and phosphate increases circulating fibroblast growth factor 23 concentrations.
- Genome-wide association study of bone mineral density in premenopausal European-American women and replication in African-American women.
- Clinical variability of familial tumoral calcinosis caused by novel GALNT3 mutations.
- Phosphaturic mesenchymal tumor, mixed connective tissue variant, of the mandible: report of a case and review of the literature.
- Hypophosphatemic rickets with hypercalciuria due to mutation in SLC34A3/Type IIc sodium-phosphate cotransporter: presentation as hypercalciuria and nephrolithiasis.
- Bone mineral density variation in men is influenced by sex-specific and non sex-specific quantitative trait loci.
- Ablation of the Galnt3 gene leads to low-circulating intact fibroblast growth factor 23 (Fgf23) concentrations and hyperphosphatemia despite increased Fgf23 expression.
- Epistasis between QTLs for bone density variation in Copenhagen x dark agouti F2 rats.
- Genetics of familial tumoral calcinosis.
- Association of adenylate cyclase 10 (ADCY10) polymorphisms and bone mineral density in healthy adults.
- CLCN7 polymorphisms and bone mineral density in healthy premenopausal white women and in white men.
- Genomic expression analysis of rat chromosome 4 for skeletal traits at femoral neck.
- Identification of a linkage disequilibrium block in chromosome 1q associated With BMD in premenopausal white women.
- Mutational survey of the PHEX gene in patients With X-linked hypophosphatemic rickets.
- Linkage screen for BMD phenoTypes in male and female COP and DA rat strains.
- Sex-specific genetic loci for femoral neck bone mass and strength identified in inbred COP and DA rats.
- Efficient and stable gene expression into human osteoclasts using an HIV-1-based lentiviral vector.
- Genetic loci affecting bone structure and strength in inbred COP and DA rats.
- Epistatic effects contribute to variation in BMD in Fischer 344 x Lewis F2 rats.
- A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosis.
- Novel GALNT3 mutations causing hyperostosis-hyperphosphatemia syndrome result in low intact fibroblast growth factor 23 concentrations.
- FGF23 concentrations vary With disease status in autosomal dominant hypophosphatemic rickets.
- Autosomal dominant osteopetrosis: clinical severity and natural history of 94 subjects With a chloride channel 7 gene mutation.
- Meta-analysis of genome-wide scans provides evidence for sex- and site-specific regulation of bone mass.
- Tumoral calcinosis presenting With eyelid calcifications due to novel missense mutations in the glycosyl transferase domain of the GALNT3 gene.
- Intronic deletions in the SLC34A3 gene cause hereditary hypophosphatemic rickets With hypercalciuria.
- Disease status in autosomal dominant osteopetrosis Type 2 is determined by osteoclastic properties.
- Heritability of changes in bone size and bone mass With age in premenopausal white sisters.
- Sensitivity of fibroblast growth factor 23 measurements in tumor-induced osteomalacia.
- The roles of specific genes implicated as circulating factors involved in normal and disordered phosphate homeostasis: frizzled related protein-4, matrix extracellular...
- Human ALOX12, but not ALOX15, is associated With BMD in white men and women.
- Polymorphisms in the estrogen receptor beta (ESR2) gene are associated With bone mineral density in Caucasian men and women.
- Analysis of variation in expression of autosomal dominant osteopetrosis Type 2: searching for modifier genes.
- Sex-specific quantitative trait loci contribute to normal variation in bone structure at the proximal femur in men.
- Fibroblast growth factor 23: roles in health and disease.
- Whole-genome scan for linkage to bone strength and structure in inbred Fischer 344 and Lewis rats.
- Genome screen for bone mineral density phenoTypes in Fisher 344 and Lewis rat strains.
- Sex-specific and non-sex-specific quantitative trait loci contribute to normal variation in bone mineral density in men.
- A novel GALNT3 mutation in a pseudoautosomal dominant form of tumoral calcinosis: evidence that the disorder is autosomal recessive.
- Persistent tumor-induced osteomalacia confirmed by elevated postoperative levels of serum fibroblast growth factor-23 and 5-year follow-up of bone density changes.
- Mutations that cause osteoglophonic dysplasia define novel roles for FGFR1 in bone elongation.
- Contribution of the LRP5 gene to normal variation in peak BMD in women.
- False positive rates in association studies as a function of degree of stratification.
- FGF23 is processed by proprotein convertases but not by PHEX.
- Osteoclast-derived serum tartrate-resistant acid phosphatase 5b in Albers-Schonberg disease (Type II autosomal dominant osteopetrosis).
- Tumor-induced Rickets: usefulness of MR gradient echo recall imaging for tumor localization.
- Confirmation of linkage to chromosome 1q for peak vertebral bone mineral density in premenopausal white women.
- Most osteomalacia-associated mesenchymal tumors are a single histopathologic entity: an analysis of 32 cases and a comprehensive review of the literature.
- Renal phosphate wasting Disorders: clinical features and pathogenesis.
- FGF-23 in fibrous dysplasia of bone and its relationship to Renal phosphate wasting.
- Chloride channel 7 (ClCN7) gene mutations and autosomal dominant osteopetrosis, Type II.
- Autosomal dominant hyperostosis/osteosclerosis With high serum alkaline phosphatase activity.
- Fibroblast growth factor 23 in oncogenic osteomalacia and X-linked hypophosphatemia.
- Sibling pair linkage and association studies between peak bone mineral density and the gene locus for the osteoclast-specific subunit (OC116) of the vacuolar proton pu...
- Genetics of Osteoporosis.
- Measurement of tartrate-resistant acid phosphatase and the brain isoenzyme of creatine kinase accurately diagnoses Type II autosomal dominant osteopetrosis but does no...
- The genetics of absorptive hypercalciuria--a note of caution.
- Bone loss at the femoral neck in premenopausal white women: effects of weight change and sex-hormone levels.
- Case 29-2001: oncogenic hypophosphatemic osteomalacia.
- Serum fibroblast growth factor 23, serum iron and bone mineral density in premenopausal women.
Schools
University of California At San Francisco
University MD
Duke University Med Center
Conditions Treated
- Calcium Metabolism Disorders
- Diabetes Type 1
- Diabetes Type 2
- Diabetes With Renal Manifestations
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Doctors Specialties
- Endocrinology
Accepted Insurances
Awards
- America's Top Doctors
- Indianapolis Monthly
- Elected Member
- Fellow (FACP)
Education
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Duke University Hospital -
University of Maryland -
University of California San Francisco School of Medicine
Hospital
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Duke University Hospital
Drug Facts
| NPI NUMBER | 1619931441 | |
| NPPES Provider LastName | ECONS | |
| NPPES Provider FirstName | MICHAEL | |
| NPPES Provider ZIPCode | 462025233 | |
| NPPES Provider State | IN | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 175.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1619931441 |
| Last Name Of The Provider | ECONS |
| First Name Of The Provider | MICHAEL |
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