Instructor, Pathology, University of Chicago, Pritzker School of Medicine
Pathology
Dr. Megan E. McNerney, MD
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Megan McNerney is an expert in the field of Pathology. She graduated from University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine is ranked 10/19 in Research/PrimaryCare. Megan McNerney is a published doctor. She published 21 publications, including: 'Cooperative loss of RAS feedback regulation drives myeloid leukemogenesis.'
Publications
- Cooperative loss of RAS feedback regulation drives myeloid leukemogenesis.
- Nfkb1 is a haploinsufficient DNA damage-specific tumor suppressor.
- The spectrum of somatic mutations in high-risk acute myeloid leukaemia with -7/del(7q).
- Bionimbus: a cloud for managing, analyzing and sharing large genomics datasets.
- Dominant Role of Oncogene Dosage and Absence of Tumor Suppressor Activity in Nras-Driven Hematopoietic Transformation.
- Oral cavity tumors in younger patients show a poor prognosis and do not contain viral RNA.
- CUX1 is a haploinsufficient tumor suppressor gene on chromosome 7 frequently inactivated in acute myeloid leukemia.
- An integrated genomic approach to the assessment and treatment of acute myeloid leukemia.
- Requirement of homotypic NK-cell interactions through 2B4(CD244)/CD48 in the generation of NK effector functions.
- 2B4 (CD244)-CD48 interactions provide a novel MHC class I-independent system for NK-cell self-tolerance in mice.
- Targeted disruption of the 2B4 gene in mice reveals an in vivo role of 2B4 (CD244) in the rejection of B16 melanoma cells.
- 2B4 acts as a non-major histocompatibility complex binding inhibitory receptor on mouse natural killer cells.
- Widespread genetic epistasis among cancer genes.
- Role of natural killer cell subsets in cardiac allograft rejection.
- The CD2 family of natural killer cell receptors.
- Juvenile animal studies: testing strategies and design.
- A new self: MHC-class-I-independent natural-killer-cell self-tolerance.
- Development of warm auto- and allo-antibodies in a 3-year old boy with sickle cell haemoglobinopathy following his first transfusion of a single unit of red blood cells.
- 2B4 (CD244) is a non-MHC binding receptor with multiple functions on natural killer cells and CD8+ T cells.
- Genetic pathways leading to therapy-related myeloid neoplasms.
Schools
U Of Chgo Div Of Bio Sci Pritzker Sch Of Med
Doctors Specialties
- Clinical Pathology
- Pathology
Accepted Insurances
Education
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University of Chicago -
University of Chicago Pritzker School of Medicine
Hospital
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University of Chicago Medical Center
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