Infectious Disease Division Massachusetts General Hospital
Internal Medicine
Dr. Mathias Lichterfeld, MD
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Mathias Lichterfeld is an expert in the field of Internal Medicine. Mathias Lichterfeld is a published MD. He has 71 papers published. The most current publication "Th1/17 Polarization of CD4 T Cells Supports HIV-1 Persistence during Antiretroviral Therapy." He is registered with Medicare and accepts Medicare payments.
Publications
- Th1/17 Polarization of CD4 T Cells Supports HIV-1 Persistence during Antiretroviral Therapy.
- Innate immune activity correlates with CD4 T cell-associated HIV-1 DNA decline during latency-reversing treatment with panobinostat.
- Dysfunctional HIV-Specific CD8(+) T Cell Proliferation Is Associated with Increased Caspase-8 Activity and Mediated by Necroptosis.
- Studies on quantitative phosphopeptide analysis by matrix-assisted laser desorption/ionization mass spectrometry without label, chromatography or calibration curves.
- Long-term Antiretroviral Treatment Initiated in Primary HIV-1 Infection Affects the Size, Composition and Decay Kinetics of the Reservoir of HIV-1 Infected CD4 T Cells.
- A Cell-Intrinsic Inhibitor of HIV-1 Reverse Transcription in CD4(+) T Cells from Elite Controllers.
- LILRB2 Interaction with HLA Class I Correlates with Control of HIV-1 Infection.
- HIV-1 persistence in CD4(+) T cells with stem cell-like properties.
- Blunted Response to Combination Antiretroviral Therapy in HIV Elite Controllers: An International HIV Controller Collaboration.
- Hepatitis C therapy with Interferon-α and Ribavirin reduces CD4 T cell-associated HIV-1 DNA in HIV-1/HCV co-infected patients.
- Susceptibility to CD8 T cell-mediated killing influences the reservoir of latently HIV-1 infected CD4 T cells.
- Functional Characterization of HLA-G(+) Regulatory T Cells in HIV-1 Infection.
- Shelterin Dysfunction and p16INK4a-Mediated Growth Inhibition in HIV-1-Specific CD8 T Cells.
- Decreased prevalence of Helicobacter pylori infection in HIV patients with AIDS defining diseases.
- Mobilization of CD34+ haematopoietic stem cells is associated with a functional inactivation of the integrin very late antigen 4.
- Long-term efficacy and safety of ritonavir/indinavir at 400/400 mg twice a day in combination with two nucleoside reverse transcriptase inhibitors as first line antire...
- Superior virological efficacy of ritonavir-boosted protease inhibitor regimens compared to single protease inhibitor therapy.
- [Fatty liver and increased serum lactate in a woman with HIV].
- Regulatory and accessory HIV-1 proteins: potential targets for HIV-1 vaccines?
- Structured treatment interruptions following immediate initiation of HAART in eight patients with acute HIV-1 seroconversion.
- Treatment of HIV-1-associated Kaposi''s sarcoma with pegylated liposomal doxorubicin and HAART simultaneously induces effective tumor remission and CD4+ T cell recovery.
- Liver histopathology in human immunodeficiency virus-hepatitis C virus co-infected patients with fatal liver disease.
- The majority of currently circulating human immunodeficiency virus type 1 clade B viruses fail to prime cytotoxic T-lymphocyte responses against an otherwise immunodom...
- T cell receptor cross-recognition of an HIV-1 CD8+ T cell epitope presented by closely related alleles from the HLA-A3 superfamily.
- Control of human immunodeficiency virus replication by cytotoxic T lymphocytes targeting subdominant epitopes.
- De novo generation of escape variant-specific CD8+ T-cell responses following cytotoxic T-lymphocyte escape in chronic human immunodeficiency virus type 1 infection.
- High degree of inter-clade cross-reactivity of HIV-1-specific T cell responses at the single peptide level.
- HLA-B63 presents HLA-B57/B58-restricted cytotoxic T-lymphocyte epitopes and is associated with low human immunodeficiency virus load.
- Sequential deregulation of NK cell subset distribution and function starting in acute HIV-1 infection.
- Loss of HIV-1-specific T cell proliferation in chronic HIV-1 infection: cause or consequence of viral replication?
- Limited sequence evolution within persistently targeted CD8 epitopes in chronic human immunodeficiency virus type 1 infection.
- The emerging role of leukocyte immunoglobulin-like receptors (LILRs) in HIV-1 infection.
- Dendritic cell dysfunction during primary HIV-1 infection.
- Induction of strong HIV-1-specific CD4+ T-cell responses using an HIV-1 gp120/NefTat vaccine adjuvanted with AS02A in antiretroviral-treated HIV-1-infected individuals.
- Elite control of HIV: p21 (waf-1/cip-1) at its best.
- Inhibition of HIV-1 integration in ex vivo-infected CD4 T cells from elite controllers.
- CD4+ T cells from elite controllers resist HIV-1 infection by selective upregulation of p21.
- Transcriptional profiling of CD4 T cells identifies distinct subgroups of HIV-1 elite controllers.
- Mutational escape in HIV-1 CTL epitopes leads to increased binding to inhibitory myelomonocytic MHC class I receptors.
- Soluble HLA-G inhibits myeloid dendritic cell function in HIV-1 infection by interacting with leukocyte immunoglobulin-like receptor B2.
- Leukocyte immunoglobulin-like receptors maintain unique antigen-presenting properties of circulating myeloid dendritic cells in HIV-1-infected elite controllers.
- Epigenetic regulation of telomerase expression in HIV-1-specific CD8+ T cells.
- HLA-B*35-Px-mediated acceleration of HIV-1 infection by increased inhibitory immunoregulatory impulses.
- High-dose daptomycin for the treatment of endocarditis caused by Staphylococcus aureus with intermediate susceptibility to glycopeptides.
- Telomerase activity of HIV-1-specific CD8+ T cells: constitutive up-regulation in controllers and selective increase by blockade of PD ligand 1 in progressors.
- A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells.
- Random T-cell receptor recruitment in human immunodeficiency virus type 1 (HIV-1)-specific CD8+ T cells from genetically identical twins infected with the same HIV-1 s...
- Decreased CXCR3+ CD8 T cells in advanced human immunodeficiency virus infection suggest that a homing defect contributes to cytotoxic T-lymphocyte dysfunction.
- Recognition of a defined region within p24 gag by CD8+ T cells during primary human immunodeficiency virus type 1 infection in individuals expressing protective HLA cl...
- Selective depletion of high-avidity human immunodeficiency virus type 1 (HIV-1)-specific CD8+ T cells after early HIV-1 infection.
- Mutually exclusive T-cell receptor induction and differential susceptibility to human immunodeficiency virus type 1 mutational escape associated with a two-amino-acid ...
- HLA Alleles Associated with Delayed Progression to AIDS Contribute Strongly to the Initial CD8(+) T Cell Response against HIV-1.
- Immunological and virological impact of highly active antiretroviral therapy initiated during acute HIV-1 infection.
- Clinical outcomes of HIV-HCV co-infection in a large cohort of hemophiliac patients.
- Immunodominance of HIV-1-specific CD8(+) T-cell responses in acute HIV-1 infection: at the crossroads of viral and host genetics.
- Limited durability of viral control following treated acute HIV infection.
- Increased natural killer cell activity in viremic HIV-1 infection.
- Loss of HIV-1-specific CD8+ T cell proliferation after acute HIV-1 infection and restoration by vaccine-induced HIV-1-specific CD4+ T cells.
- Rapid determination of the Delta32 deletion in the human CC-chemokine receptor 5 (CCR5) gene without DNA extraction by lightcycler real-time polymerase chain reaction.
- HIV-1 Nef is preferentially recognized by CD8 T cells in primary HIV-1 infection despite a relatively high degree of genetic diversity.
- Selection, transmission, and reversion of an antigen-processing cytotoxic T-lymphocyte escape mutation in human immunodeficiency virus type 1 infection.
- Differences in the expressed HLA class I alleles effect the differential clustering of HIV type 1-specific T cell responses in infected Chinese and caucasians.
- Comprehensive analysis of human immunodeficiency virus type 1-specific CD4 responses reveals marked immunodominance of gag and nef and the presence of broadly recogniz...
- HIV-1-specific cytotoxicity is preferentially mediated by a subset of CD8(+) T cells producing both interferon-gamma and tumor necrosis factor-alpha.
- Diagnosis of invasive septate mold infections. A correlation of microbiological culture and histologic or cytologic examination.
- The tandem-repeat polymorphism of the DC-SIGNR gene does not affect the susceptibility to HIV infection and the progression to AIDS.
- [Antiretroviral treatment of HIV infected patients].
- Pilot study of interferon alpha high-dose induction therapy in combination with ribavirin for chronic hepatitis C in HIV-co-infected patients.
- Reduced CC chemokine receptor (CCR) 1 and CCR5 surface expression on peripheral blood T lymphocytes from patients with chronic hepatitis C infection.
- Antiretroviral drug toxicity -- a challenge for the hepatologist?
Doctors Specialties
- Infectious Disease Medicine
- Internal Medicine
Accepted Insurances
Education
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Massachusetts General Hospital -
University of Bonn Faculty of Medicine
Hospital
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Brigham and Women's Hospital -
Massachusetts General Hospital
Drug Facts
| NPI NUMBER | 1578690921 | |
| NPPES Provider LastName | LICHTERFELD | |
| NPPES Provider FirstName | MATHIAS | |
| NPPES Provider ZIPCode | 02114 | |
| NPPES Provider State | MA | |
| Specialty Description | Infectious Disease | |
| Total Claim Count | 127.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1578690921 |
| Last Name Of The Provider | LICHTERFELD |
| First Name Of The Provider | MATHIAS |
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