Professor, Medicine, University of Minnesota Medical School
Nephrology
Dr. Mark S. Paller, MD
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Mark S. Paller, MD works in the field of Nephrology. He graduated from Northwestern University Feinberg School of Medicine. Northwestern University Feinberg School of Medicine has a rank of 19 in research and a 29 in primary care He has 1 awards "CMS Meaningful Use Stage 1 Certification". Mark Paller has written publications. He has 73 publications published with PubMed. The lastest publication is: 'Under new Medicare regulations, can we still educate for uncertainty?' He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Committed to cardiovascular Disease research.
- Investing in research: the impact of one academic health center's research grant program.
- A program to provide regulatory support for investigator-initiated clinical research.
- Clinical trials at AHCs: the perspective of an academic clinical trials office.
- Introducing residents to a career in management: the Physician Management Pathway.
- Reperfusion injury pathophysiology in sickle transgenic mice.
- Under new Medicare regulations, can we still educate for uncertainty?
- Acute renal failure: controversies, clinical trials, and future directions.
- Nitric oxide-mediated Renal epithelial cell injury during hypoxia and reoxygenation.
- Purinergic receptors mediate cell proliferation and enhanced recovery from Renal ischemia by adenosine triphosphate.
- Effects of integrins on proliferation and apoptosis of renal epithelial cells after Acute injury.
- Integrins and repair after Acute renal injury.
- Acute renal failure in the 21st century: recommendations for management and outcomes assessment.
- A proposal for funding clinical research trials by health care insurers.
- The cell biology of reperfusion injury in the Kidney.
- Lateral diffusion of lipids in Renal cells: effects of hypoxia and reoxygenation and role of cytoskeleton.
- A randomized double-blind trial of the use of human recombinant superoxide dismutase in Renal transplantation.
- Heme oxygenase is not expressed as a stress protein after Renal ischemia.
- Calcium and free radicals in hypoxia/reoxygenation injury of renal epithelial cells.
- Bone marrow transplantation Nephropathy.
- Cytochrome P-450 mediates tissue-damaging hydroxyl radical formation during reoxygenation of the Kidney.
- Role of calcium in reperfusion injury of the Kidney.
- Iron loading enhances susceptibility to Renal ischemia in rats.
- Hazards of antioxidant combinations containing superoxide dismutase.
- Lateral mobility of Na,K-ATPase and membrane lipids in Renal cells. Importance of cytoskeletal integrity.
- Glomerular Disease and lung transplantation.
- Extracellular iron chelators protect Kidney cells from hypoxia/reoxygenation.
- Hemodynamic effects of alterations in potassium.
- Pathogenesis of sodium and water retention in edematous Disorders.
- Mechanism of decreased vascular reactivity to angiotensin II in conscious, potassium-deficient rats.
- Role of vasopressin in support of blood pressure in potassium deficient rats.
- Oxygen free radicals in ischemic Acute renal failure in the rat.
- Mechanism of decreased pressor responsiveness to ANG II, NE, and vasopressin in pregnant rats.
- Anticytoplasmic antibodies in antinuclear antibody-negative lupus erythematosus. Correlation With clinical course.
- Role of angiotensin II, alpha-adrenergic system, and arginine vasopressin on arterial pressure in rat.
- Cardiovascular and endocrine effects of potassium in spontaneously Hypertensive rats.
- Free radical scavengers in mercuric chloride-induced Acute renal failure in the rat.
- Effect of cyclosporine administration on Renal hemodynamics in conscious rats.
- Renal dysfunction in animal models of cyclosporine toxicity.
- Ethane production as a measure of lipid peroxidation after Renal ischemia.
- Hypothyroidism protects against free radical damage in ischemic Acute renal failure.
- The role of intensive dialysis in Acute renal failure.
- Beneficial effects of Renal denervation and prazosin on GFR and Renal blood flow after cyclosporine in rats.
- Effects of Nva2-cyclosporine on glomerular filtration rate and Renal blood flow in the rat.
- Decreased pressor responsiveness in pregnancy: studies in experimental animals.
- Dietary protein increases plasma renin and reduces pressor reactivity to angiotensin II.
- Hemoglobin- and myoglobin-induced Acute renal failure in rats: role of iron in nephrotoxicity.
- Renal work, glutathione and susceptibility to free radical-mediated postischemic injury.
- Role of iron in postischemic Renal injury in the rat.
- The prostaglandin E1 analog misoprostol reverses Acute cyclosporine nephrotoxicity.
- Effects of the prostaglandin E1 analog misoprostol on cyclosporine nephrotoxicity.
- Decreased pressor reactivity to angiotensin II in cirrhotic rats. Evidence for a post-receptor defect in angiotensin action.
- Effect of neutrophil depletion on ischemic Renal injury in the rat.
- Potassium depletion and regulation of angiotensin II receptors in vascular smooth muscle cells.
- Pressor resistance to vasopressin in sodium depletion, potassium depletion, and cirrhosis.
- Pressor responsiveness in pseudopregnant and pregnant rats: role of Maternal factors.
- Severe flank pain as the predominant symptom of IgA Nephropathy.
- Long-term effects of cyclosporine on Renal function in organ transplant recipients.
- Cyclosporine augments Renal but not systemic vascular reactivity.
- Cyclosporine nephrotoxicity and the role of cyclosporine in living-related donor transplantation.
- Drug-induced nephropathies.
- Dietary deficiency of antioxidants exacerbates ischemic injury in the rat Kidney.
- Radical thoughts about Kidneys.
- Differential gene expression in the recovery from ischemic Renal injury.
- Oxygen free radicals in Acute renal failure.
- Reactive oxygen species and rat Renal epithelial cells during hypoxia and reoxygenation.
- Hydrogen peroxide and ischemic Renal injury: effect of catalase inhibition.
- Protective effects of glutathione, glycine, or alanine in an in vitro model of Renal anoxia.
- Free radical-mediated postischemic injury in Renal transplantation.
- Prostaglandins protect Kidneys against ischemic and toxic injury by a cellular effect.
- Henoch-Schönlein vasculitis as a manifestation of IgA-associated Disease in cirrhosis.
- Xanthine oxidase produces O2-. in posthypoxic injury of Renal epithelial cells.
Procedures Preformed
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Alkalosis
- Calcium Metabolism Disorders
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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University of Colorado -
Case Western Reserve University - University Hospitals -
Northwestern University Feinberg School of Medicine
Hospital
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University of Minnesota Medical Center, Fairview
Drug Facts
| NPI NUMBER | 1841304359 | |
| NPPES Provider LastName | PALLER | |
| NPPES Provider FirstName | MARK | |
| NPPES Provider ZIPCode | 55455 | |
| NPPES Provider State | MN | |
| Specialty Description | Nephrology | |
| Total Claim Count | 519.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1841304359 |
| Last Name Of The Provider | PALLER |
| First Name Of The Provider | MARK |
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