Physician
Nephrology
Dr. Mark F. Bevan, MD
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Dr. Mark F. Bevan practices Nephrology. He researched medicine at Duke University School of Medicine. Duke University School of Medicine is a top medical college. Duke University School of Medicine has a rank of 8 in research and a 29 in primary care Dr. Mark F. Bevan is a published doctor as well. He has 42 publications published. The latest was: Microcircuitry of the direct and indirect pathways of the basal ganglia.
Publications
- Heterosynaptic regulation of external globus pallidus inputs to the subthalamic nucleus by the motor cortex.
- Short-term Depression of External Globus Pallidus-Subthalamic Nucleus Synaptic Transmission and Implications for Patterning Subthalamic Activity.
- Introduction to the special edition: function and dysfunction of the basal ganglia.
- Microcircuitry of the direct and indirect pathways of the basal ganglia.
- The subthalamic nucleus and the external pallidum: two tightly interconnected structures that control the output of the basal ganglia in the monkey.
- Efferent connections of the internal globus pallidus in the squirrel monkey: I. Topography and synaptic organization of the pallidothalamic projection.
- Intrinsic dynamics and synaptic inputs control the activity patterns of subthalamic nucleus neurons in health and in Parkinson's Disease.
- Synaptic organisation of the basal ganglia.
- Glutamate-enriched cholinergic synaptic terminals in the entopeduncular nucleus and subthalamic nucleus of the rat.
- The glutamate-enriched cortical and thalamic input to neurons in the subthalamic nucleus of the rat: convergence With GABA-positive terminals.
- The substantia nigra as a site of synaptic integration of functionally diverse information arising from the ventral pallidum and the globus pallidus in the rat.
- Neurons projecting from the entopeduncular nucleus to the thalamus receive convergent synaptic inputs from the subthalamic nucleus and the neostriatum in the rat.
- Dopamine regulates the impact of the cerebral cortex on the subthalamic nucleus-globus pallidus network.
- Convergent synaptic input from the neostriatum and the subthalamus onto identified nigrothalamic neurons in the rat.
- Cholinergic, GABAergic, and glutamate-enriched inputs from the mesopontine tegmentum to the subthalamic nucleus in the rat.
- Glutamate-enriched inputs from the mesopontine tegmentum to the entopeduncular nucleus in the rat.
- Regulation of the timing and pattern of action potential generation in rat subthalamic neurons in vitro by GABA-A IPSPs.
- Equilibrium potential of GABA(A) current and implications for rebound burst firing in rat subthalamic neurons in vitro.
- Relationship of activity in the subthalamic nucleus-globus pallidus network to cortical electroencephalogram.
- Mechanisms underlying spontaneous oscillation and rhythmic firing in rat subthalamic neurons.
- Selective innervation of neostriatal interneurons by a subclass of neuron in the globus pallidus of the rat.
- Synaptic integration of functionally diverse pallidal information in the entopeduncular nucleus and subthalamic nucleus in the rat.
- Selective participation of somatodendritic HCN channels in inhibitory but not excitatory synaptic integration in neurons of the subthalamic nucleus.
- Cellular mechanisms underlying burst firing in substantia nigra dopamine neurons.
- Sparse but selective and potent synaptic transmission from the globus pallidus to the subthalamic nucleus.
- Autonomous initiation and propagation of action potentials in neurons of the subthalamic nucleus.
- D2-like dopamine receptor-mediated modulation of activity-dependent plasticity at GABAergic synapses in the subthalamic nucleus.
- D2-like dopamine receptors modulate SKCa channel function in subthalamic nucleus neurons through inhibition of Cav2.2 channels.
- Accumulation of cytoplasmic Calcium, but not apamin-sensitive afterhyperpolarization current, during high frequency firing in rat subthalamic nucleus cells.
- GABAergic control of the subthalamic nucleus.
- Synaptic activation of dendritic AMPA and NMDA receptors generates transient high-frequency firing in substantia nigra dopamine neurons in vitro.
- Cellular principles underlying normal and pathological activity in the subthalamic nucleus.
- Enhancement of excitatory synaptic integration by GABAergic inhibition in the subthalamic nucleus.
- Ionic mechanisms underlying autonomous action potential generation in the somata and dendrites of GABAergic substantia nigra pars reticulata neurons in vitro.
- Globus pallidus neurons dynamically regulate the activity pattern of subthalamic nucleus neurons through the frequency-dependent activation of postsynaptic GABAA and G...
- Synaptic release of dopamine in the subthalamic nucleus.
- Synchronous unit activity and local field potentials evoked in the subthalamic nucleus by cortical stimulation.
- A model of reverse spike frequency adaptation and repetitive firing of subthalamic nucleus neurons.
- Apamin-sensitive small conductance Calcium-activated potassium channels, through their selective coupling to voltage-gated Calcium channels, are critical determinants ...
- "The little engine that could": voltage-dependent Na(+) channels and the subthalamic nucleus.
- Move to the rhythm: oscillations in the subthalamic nucleus-external globus pallidus network.
Procedures Preformed
- Arteriovenostomy for Renal Dialysis
- Hemodialysis
- Peritoneal Dialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Alkalosis
- Calcium Metabolism Disorders
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Doctors Specialties
- Nephrology
Accepted Insurances
Education
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Duke University School of Medicine
Drug Facts
| NPI NUMBER | 1720185788 | |
| NPPES Provider LastName | BEVAN | |
| NPPES Provider FirstName | MARK | |
| NPPES Provider ZIPCode | 874016589 | |
| NPPES Provider State | NM | |
| Specialty Description | Nephrology | |
| Total Claim Count | 7140.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 216.0 | |
| Percent Opioid Claims | 3.03 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1720185788 |
| Last Name Of The Provider | BEVAN |
| First Name Of The Provider | MARK |
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