Children's Hospital of Dallas University of Texas Southwestern Medical Center at Dallas

Pediatrics

Margaret Schwarz is an expert in the field of Pediatrics. He went to University of Missouri - Kansas City School of Medicine. Dr. Margaret Schwarz is also a published doctor. He has 27 publication published. The most recent publication is: N-terminus of pro-EMAP II regulates its binding with C-terminus, Arginyl-tRNA Synthetase, and Neurofilament light protein.

Publications

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• N-terminus of pro-EMAP II regulates its binding with C-terminus, Arginyl-tRNA Synthetase, and Neurofilament light protein.
• Nintedanib, a triple angiokinase inhibitor, enhances cytotoxic therapy response in pancreatic cancer.
• Blockade of EMAP II protects cardiac function after chronic myocardial infarction by inducing angiogenesis.
• Enhancement of Nab-paclitaxel Antitumor Activity through Addition of Multitargeting Antiangiogenic Agents in Experimental Pancreatic Cancer.
• The dual PI3K/mTOR inhibitor NVP-BEZ235 enhances nab-paclitaxel antitumor response in experimental gastric cancer.
• Superior antitumor activity of nanoparticle albumin-bound Paclitaxel in experimental gastric cancer.
• Cavin-3 dictates the balance between ERK and Akt signaling.
• Potential role for antiangiogenic proteins in the evolution of bronchopulmonary dysplasia.
• Potential role for antiangiogenic proteins in the myocardial infarction repair process.
• Endothelial-monocyte activating polypeptide II alters fibronectin based endothelial cell adhesion and matrix assembly via alpha5 beta1 integrin.
• Identification of protease-sensitive sites in Human Endothelial-Monocyte Activating Polypeptide II protein.
• Proteasome inhibition enhances antitumour effects of gemcitabine in experimental pancreatic cancer.
• In vivo therapy of local tumor progression by targeting vascular endothelium with EMAP-II.
• Epithelial-mesenchymal interactions are linked to neovascularization.
• The efficacy of a novel, dual PI3K/mTOR inhibitor NVP-BEZ235 to enhance chemotherapy and antiangiogenic response in pancreatic cancer.
• Antitumour activity of sunitinib in combination with gemcitabine in experimental pancreatic cancer.
• Androgen receptor blockade in experimental combination therapy of pancreatic cancer.
• Smac mimetic-derived augmentation of chemotherapeutic response in experimental pancreatic cancer.
• Endothelial monocyte activating polypeptide II interferes with VEGF-induced proangiogenic signaling.
• An antiendothelial combination therapy strategy to increase survival in experimental pancreatic cancer.
• Enhancing cytotoxic agent activity in experimental pancreatic cancer through EMAP II combination therapy.
• EMAP II-based antiangiogenic-antiendothelial in vivo combination therapy of pancreatic cancer.
• Combination effects of bortezomib with gemcitabine and EMAP II in experimental pancreatic cancer.
• Lung self-assembly is modulated by tissue surface tensions.
• Antitumor effects of EMAP II against pancreatic cancer through inhibition of fibronectin-dependent proliferation.
• Emerging pulmonary vasculature lacks fate specification.

Schools

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• University of Missouri Kansas City Campus

Doctors Specialties

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• Pediatric Critical Care Medicine
• Pediatrics

Accepted Insurances

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• Cigna
• Commercial Insurance Company
• HealthFirst
• Medicaid
• Medicare MCR
• Kaiser PHCS Network PPO
• MultiPlan PPO
• PHCS Network PPO
• United Healthcare

Education

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• University of Pittsburgh Medical Center
• Nationwide Children's Hospital
• University of Missouri - Kansas City School of Medicine

Hospital

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• University of Texas Southwestern Medical Center
• Children's Health System of Texas

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