Associate Professor of Dermatology and Pathology Yale University
Dermatology
Dr. Marcus Bosenberg, MD
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Dr. Marcus Bosenberg is an expert in the field of Dermatology. He graduated from Weill Cornell Medical College of Cornell University. Weill Cornell Medical College of Cornell University is ranked 18 in research and 49 in primary care medicine Doctor Marcus Bosenberg, MD is a published doctor. He published 80 publications, including: 'Roles of the RAM and ANK domains in signaling by the C. elegans GLP-1 receptor.'
Publications
- Loss of PTEN Promotes Resistance to T Cell-Mediated Immunotherapy.
- Integrative discovery of CD98 as a Melanoma Biomarker.
- DNMT3b Modulates Melanoma Growth by Controlling Levels of mTORC2 Component RICTOR.
- Melanoma Treatments: Advances and Mechanisms.
- SMAC mimetic Debio 1143 synergizes with taxanes, topoisomerase inhibitors and bromodomain inhibitors to impede growth of lung adenocarcinoma cells.
- Transcriptional repression of IFNβ1 by ATF2 confers melanoma resistance to therapy.
- Therapy-induced tumour secretomes promote resistance and tumour progression.
- PDK1 and SGK3 Contribute to the Growth of BRAF-Mutant Melanomas and Are Potential Therapeutic Targets.
- mTORC1 activation blocks BrafV600E-induced growth arrest but is insufficient for melanoma formation.
- County-Level Association of Melanoma and Papillary Thyroid Cancer: Evidence of Shared Environmental Risk?
- Mitochondrial function in melanoma.
- Immune-Based Antitumor Effects of BRAF Inhibitors Rely on Signaling by CD40L and IFNγ.
- Melanoma metastasis: new concepts and evolving paradigms.
- Response to BRAF Inhibition in Melanoma Is Enhanced When Combined with Immune Checkpoint Blockade.
- Isolation and Molecular Characterization of Circulating Melanoma Cells.
- Phenformin enhances the therapeutic benefit of BRAFV600E inhibition in melanoma.
- Genetic inactivation or pharmacological inhibition of Pdk1 delays development and inhibits metastasis of Braf(V600E)::Pten(-/-) melanoma.
- Low-copy piggyBac transposon mutagenesis in mice identifies genes driving melanoma.
- MERTK controls melanoma cell migration and survival and differentially regulates cell behavior relative to AXL.
- Meeting report from the Society for Melanoma Research 2012 Congress, Hollywood, California.
- The Hematopoietic Stem Cell Regulatory Gene Latexin has Tumor Suppressive Properties in Malignant Melanoma.
- Genotype-selective combination therapies for melanoma identified by high-throughput drug screening.
- Inhibition of melanoma development in the Nras((Q61K)) ::Ink4a(-/-) mouse model by the small molecule BI-69A11.
- Familial multiple pilomatrixomas as a presentation of attenuated adenomatosis polyposis coli.
- Spontaneous genomic alterations in a chimeric model of colorectal cancer enable metastasis and guide effective combinatorial therapy.
- beta-catenin signaling controls metastasis in Braf-activated Pten-deficient melanomas.
- Norcantharidin induces melanoma cell apoptosis through activation of TR3 dependent pathway.
- piggyBac transposon somatic mutagenesis with an activated reporter and tracker (PB-SMART) for genetic screens in mice.
- Investigative pathology: leading the post-genomic revolution.
- Proinvasion metastasis drivers in early-stage melanoma are oncogenes.
- Coordinated activation of Wnt in epithelial and melanocyte stem cells initiates pigmented hair regeneration.
- Phosphoproteomic screen identifies potential therapeutic targets in melanoma.
- Unwelcome guests: macrophages promote UV-induced melanoma.
- Meeting report from the 7th International Melanoma Congress, Sydney, November, 2010.
- A role for ATF2 in regulating MITF and melanoma development.
- Mouse melanoma models and cell lines.
- A transient epidermolysis bullosa simplex-like phenotype associated with bexarotene treatment in a G138E KRT5 heterozygote.
- Differential roles of the pRb and Arf/p53 pathways in murine naevus and melanoma genesis.
- Cooperative interactions of PTEN deficiency and RAS activation in melanoma metastasis.
- Redefining regulation of DNA methylation by RNA interference.
- Lineage-specific transcriptional regulation of DICER by MITF in melanocytes.
- A role for the JARID1B stem cell marker for continuous melanoma growth.
- Integrative genome comparison of primary and metastatic melanomas.
- Melanocyte homeostasis in vivo tolerates Rb1 loss in a developmentally independent fashion.
- Characterization of melanoma cells capable of propagating tumors from a single cell.
- Chimeric mouse tumor models reveal differences in pathway activation between ERBB family- and KRAS-dependent lung adenocarcinomas.
- Activated cAMP response element binding protein is overexpressed in human mesotheliomas and inhibits apoptosis.
- Melanotic schwannoma arising in association with nevus of Ota: 2 cases suggesting a shared mechanism.
- Autoimmune acrosyringitis with ductal cysts: reclassification of case of eruptive syringoma.
- Braf(V600E) cooperates with Pten loss to induce metastatic melanoma.
- Nifurtimox induces apoptosis of neuroblastoma cells in vitro and in vivo.
- Identification of a novel subgroup of melanomas with KIT/cyclin-dependent kinase-4 overexpression.
- A new mechanism of release from senescence: suppression of p16INK4a by beta-catenin.
- LKB1 deficiency sensitizes mice to carcinogen-induced tumorigenesis.
- Loss of xeroderma pigmentosum C (Xpc) enhances melanoma photocarcinogenesis in Ink4a-Arf-deficient mice.
- Ink4a/Arf tumor suppressor does not modulate the degenerative conditions or tumor spectrum of the telomerase-deficient mouse.
- Epigenetic silencing of novel tumor suppressors in malignant melanoma.
- Precancer in mice: animal models used to understand, prevent, and treat human precancers.
- Failure to prolyl hydroxylate hypoxia-inducible factor alpha phenocopies VHL inactivation in vivo.
- Characterization of melanocyte-specific inducible Cre recombinase transgenic mice.
- Amplification of CDK4 and MDM2 in malignant melanoma.
- Role of epidermal growth factor receptor signaling in RAS-driven melanoma.
- Absence of telomerase and shortened telomeres have minimal effects on skin and pancreatic carcinogenesis elicited by viral oncogenes.
- Deregulated Akt3 activity promotes development of malignant melanoma.
- Pleomorphic liposarcoma: clinicopathologic analysis of 57 cases.
- Mouse models of human cancers (part 3).
- Roles of the RAM and ANK domains in signaling by the C. elegans GLP-1 receptor.
- lag-1, a gene required for lin-12 and glp-1 signaling in Caenorhabditis elegans, is homologous to human CBF1 and Drosophila Su(H).
- Myelogenous leukemia cutis resembling stasis dermatitis.
- Components of the Rb pathway are critical targets of UV mutagenesis in a murine melanoma model.
- Genetic analysis of Pten and Ink4a/Arf interactions in the suppression of tumorigenesis in mice.
- Utility of MMP-1, p53, E-cadherin, and collagen IV immunohistochemical stains in the differential diagnosis of adenomas with misplaced epithelium versus adenomas with ...
- HIN-1, a putative cytokine highly expressed in normal but not cancerous mammary epithelial cells.
- Telomere dysfunction and evolution of intestinal carcinoma in mice and humans.
- Activated release of membrane-anchored TGF-alpha in the absence of cytosol.
- Juxtacrine cell signaling molecules.
- The cytoplasmic carboxy-terminal amino acid specifies cleavage of membrane TGF alpha into soluble growth factor.
- Cleavage of membrane-anchored growth factors involves distinct protease activities regulated through common mechanisms.
- Both products of the mouse Ink4a/Arf locus suppress melanoma formation in vivo.
Schools
Weill Cornell Medical College
Doctors Specialties
- Dermatopathology
- Dermatology
Accepted Insurances
Education
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Harvard Medical School -
Brigham and Women's Hospital -
Weill Cornell Medical College of Cornell University
Hospital
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Yale-New Haven Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1679595094 |
| Last Name Of The Provider | BOSENBERG |
| First Name Of The Provider | MARCUS |
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