Professor, Medicine, Boston University School of Medicine
Infectious Disease
Dr. Lee M. Wetzler, MD
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Doctor Lee M. Wetzler, MD practices Infectious Disease. He attended State University of New York Upstate Medical University. Lee M. Wetzler, MD has written publications. He has 21 publications published with PubMed. The lastest publication is: 'In Vivo and In Vitro Characterization of the Immune Stimulating Activity of the Neisserial Porin PorB.' Lee M. Wetzler, MD is registered with Medicare.
Publications
- Macrophage-Specific TLR2 Signaling Mediates Pathogen-Induced TNF-Dependent Inflammatory Oral Bone Loss.
- Analysis of parameters associated with prevention of cellular apoptosis by pathogenic neisseriae and purified porins.
- The Nature of an In Vivo Anti-Capsular Polysaccharide Response Is Markedly Influenced by the Composition and/or Architecture of the Bacterial Subcapsular Domain.
- Toll-like receptor 2 induces mucosal homing receptor expression and IgA production by human B cells.
- Human airway epithelial cell responses to Neisseria lactamica and purified porin via Toll-like receptor 2-dependent signaling.
- Meningococcal porin PorB prevents cellular apoptosis in a toll-like receptor 2- and NF-kappaB-independent manner.
- Bronchus-associated lymphoid tissue (BALT) and survival in a vaccine mouse model of tularemia.
- Neisseria meningitidis PorB, a Toll-like receptor 2 ligand, improves the capacity of Francisella tularensis lipopolysaccharide to protect mice against experimental tul...
- Role of protein tyrosine kinase and Erk1/2 activities in the Toll-like receptor 2-induced cellular activation of murine B cells by neisserial porin.
- Identification of immunologic and pathologic parameters of death versus survival in respiratory tularemia.
- T cell activation by TLRs: a role for TLRs in the adaptive immune response.
- Neisseria meningitidis PorB, a TLR2 ligand, induces an antigen-specific eosinophil recall response: potential adjuvant for helminth vaccines?
- Induction of cell signaling events by the cholera toxin B subunit in antigen-presenting cells.
- Toll-like receptor 2-mediated human B cell differentiation.
- Meningococcal porin PorB binds to TLR2 and requires TLR1 for signaling.
- Improved purification of native meningococcal porin PorB and studies on its structure/function.
- The gonococcal Fur-regulated tbpA and tbpB genes are expressed during natural mucosal gonococcal infection.
- Neisserial porin-induced dendritic cell activation is MyD88 and TLR2 dependent.
- Cutting edge: Immune stimulation by neisserial porins is toll-like receptor 2 and MyD88 dependent.
- In Vivo and In Vitro Characterization of the Immune Stimulating Activity of the Neisserial Porin PorB.
Schools
SUNY Upstate Medical University Syracuse
University Of Michigan Med Center Int
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Education
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University of Michigan Hospitals and Health Centers -
State University of New York Upstate Medical University
Hospital
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Boston Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1629026752 |
| Last Name Of The Provider | WETZLER |
| First Name Of The Provider | LEE |
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