Professor, Internal Medicine, Yale University School of Medicine
Cardiology
Dr. Lawrence I. Young, MD
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Lawrence Young works in the field of Cardiology. He went to Yale University School of Medicine. Yale University School of Medicine is a one of the best medicals schools. Yale University School of Medicine has a rank of 7 in research and a 57 in primary care He has 2 awards "CMS Meaningful Use Stage 1 Certification" and "Top MD". Lawrence Young is a published doctor and has 47 publications published. The lastest was: 'miR-182 Modulates Myocardial Hypertrophic Response Induced by Angiogenesis in Heart.' Dr. Lawrence I. Young is registered with Medicare and accepts Medicare payments.
Publications
- AMPK is critical for mitochondrial function during reperfusion after myocardial ischemia.
- Urocortin 2 autocrine/paracrine and pharmacologic effects to activate AMP-activated protein kinase in the heart.
- The heart rate response to adenosine: A simple predictor of adverse cardiac outcomes in asymptomatic patients with type 2 diabetes.
- Bevacizumab: finding its niche in the treatment of heart failure secondary to liver vascular malformations in hereditary hemorrhagic telangiectasia.
- Limiting Cardiac Ischemic Injury by Pharmacological Augmentation of Macrophage Migration Inhibitory Factor-AMP-Activated Protein Kinase Signal Transduction.
- Gender-based divergence of cardiovascular outcomes in asymptomatic patients with type 2 diabetes: Results from the DIAD study.
- AMP-activated protein kinase (AMPK) activation and glycogen synthase kinase-3β (GSK-3β) inhibition induce Ca2+-independent deposition of tight junction components at...
- A small molecule AMPK activator protects the heart against ischemia-reperfusion injury.
- Risk stratification in diabetes: not all patients or perfusion defects are the same.
- Diet-induced obesity obstructs insulin signaling in the heart.
- Cardiac macrophage migration inhibitory factor inhibits JNK pathway activation and injury during ischemia/reperfusion.
- Lessons learned from the detection of ischemia in asymptomatic diabetics (DIAD) study.
- A crystallized view of AMPK activation.
- AMP-activated protein kinase conducts the ischemic stress response orchestra.
- Infusion of a biotinylated bis-glucose photolabel: a new method to quantify cell surface GLUT4 in the intact mouse heart.
- Aging-associated reductions in AMP-activated protein kinase activity and mitochondrial biogenesis.
- Diabetes mellitus and cardiovascular disease.
- AMP-activated protein kinase regulates the assembly of epithelial tight junctions.
- Activation of AMPK alpha- and gamma-isoform complexes in the intact ischemic rat heart.
- Value of peripheral vascular endothelial function in the detection of relative myocardial ischemia in asymptomatic type 2 diabetic patients who underwent myocardial pe...
- AMP-activated protein kinase activates p38 mitogen-activated protein kinase by increasing recruitment of p38 MAPK to TAB1 in the ischemic heart.
- AMP-activated protein kinase: a key stress signaling pathway in the heart.
- Dual mechanisms regulating AMPK kinase action in the ischemic heart.
- Evaluating the cardiovascular effects of the thiazolidinediones and their place in the management of type 2 diabetes in relation to the metabolic syndrome.
- Role of the nitric oxide pathway in AMPK-mediated glucose uptake and GLUT4 translocation in heart muscle.
- Brachial artery reactivity in asymptomatic patients with type 2 diabetes mellitus and microalbuminuria (from the Detection of Ischemia in Asymptomatic Diabetics-brachi...
- Diastolic function and type 1 diabetes.
- AMP-activated protein kinase mediates ischemic glucose uptake and prevents postischemic cardiac dysfunction, apoptosis, and injury.
- Cardiac myocyte-specific HIF-1alpha deletion alters vascularization, energy availability, calcium flux, and contractility in the normoxic heart.
- Cardiac abnormalities in diabetic patients with neuropathy: effects of aldose reductase inhibitor administration.
- Thiazolidinedione use, fluid retention, and congestive heart failure: a consensus statement from the American Heart Association and American Diabetes Association.
- Insulin resistance and the effects of thiazolidinediones on cardiac metabolism.
- Physiological role of AMP-activated protein kinase in the heart: graded activation during exercise.
- Diagnosis of CAD in patients with diabetes: who to evaluate.
- AMP kinase is required for mitochondrial biogenesis in skeletal muscle in response to chronic energy deprivation.
- Cardiac outcomes after myocardial infarction in elderly patients with diabetes mellitus.
- Acute myocardial infarction in the elderly with diabetes.
- Correlation of myocardial p-(123)I-iodophenylpentadecanoic acid retention with (18)F-FDG accumulation during experimental low-flow ischemia.
- miR-182 Modulates Myocardial Hypertrophic Response Induced by Angiogenesis in Heart.
- Five-year outcomes in high-risk participants in the Detection of Ischemia in Asymptomatic Diabetics (DIAD) study: a post hoc analysis.
- Thiazolidinedione use, fluid retention, and congestive heart failure: a consensus statement from the American Heart Association and American Diabetes Association. Octo...
- Detection of silent myocardial ischemia in asymptomatic diabetic subjects: the DIAD study.
- Macrophage migration inhibitory factor stimulates AMP-activated protein kinase in the ischaemic heart.
- Screening for coronary artery disease in patients with diabetes.
- Resolution of asymptomatic myocardial ischemia in patients with type 2 diabetes in the Detection of Ischemia in Asymptomatic Diabetics (DIAD) study.
- Cardiac outcomes after screening for asymptomatic coronary artery disease in patients with type 2 diabetes: the DIAD study: a randomized controlled trial.
Schools
University of North Carolina At Chapel Hill
North Carolina Memorial Hospital
Doctors Specialties
- Anesthesiology
- Cardiology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Top MD
Education
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Yale-New Haven Hospital -
Yale University School of Medicine
Hospital
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Yale-New Haven Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1548236383 |
| Last Name Of The Provider | YOUNG |
| First Name Of The Provider | LAWRENCE |
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