Associate Professor of Pediatrics, University of Chicago Medicine
Pediatric Hematology & Oncology
Dr. Kenan Onel, MD
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Kenan Onel works in the field of Pediatric Hematology & Oncology. He went to Weill Cornell Medical College of Cornell University. Weill Cornell Medical College of Cornell University is ranked 18 in research and 49 in primary care medicine Kenan Onel is a published physician. He has 38 papers published. The most current publication "Levels of soluble Fas/APO-1/CD95 in systemic lupus erythematosus and juvenile rheumatoid arthritis."
Publications
- Identification and Utilization of Donor and Recipient Genetic Variants to Predict Survival After HCT: Are We Ready for Primetime?
- Radiation-induced apoptosis varies among individuals and is modified by sex and age.
- A recurrent germline PAX5 mutation confers susceptibility to pre-B Cell acute lymphoblastic leukemia.
- Evolving risk of therapy-related acute myeloid leukemia following cancer chemotherapy among adults in the United States, 1975-2008.
- Leveraging epidemiology and clinical studies of cancer outcomes: recommendations and opportunities for translational research.
- Proposal for the clinical detection and management of patients and their family members with familial myelodysplastic syndrome/acute leukemia predisposition syndromes.
- Quality of life in children with systemic lupus erythematosus: a review.
- Combined intravenous methotrexate and cyclophosphamide for refractory childhood lupus nephritis.
- Relationship of quality of life and physical function measures with Disease activity in children with systemic lupus erythematosus.
- Levels of soluble Fas/APO-1/CD95 in systemic lupus erythematosus and juvenile rheumatoid arthritis.
- Do children with lupus have fewer male siblings?
- Advances in the medical treatment of juvenile rheumatoid arthritis.
- Quality of life in paediatric lupus.
- Expression and function of the murine CD95/FasR/APO-1 receptor in relation to B Cell ontogeny.
- Lack of association of the TP53 Arg72Pro SNP and the MDM2 SNP309 with systemic lupus erythematosus in Caucasian, African American, and Asian children and adults.
- Characterization of NOL7 gene point mutations, promoter methylation, and protein expression in cervical cancer.
- A genome-wide meta-analysis of nodular sclerosing Hodgkin lymphoma identifies risk loci at 6p21.32.
- Variants at 6q21 implicate PRDM1 in the etiology of therapy-induced second Malignancies after Hodgkin's lymphoma.
- An integrated genomic approach to the assessment and treatment of acute myeloid leukemia.
- Natural selection and functional genetic variation in the p53 pathway.
- Identification and molecular characterization of a novel 3′ mutation in RUNX1 in a family with familial platelet disorder.
- Heritability of hematologic Malignancies: from pedigrees to genomics.
- Anti-tumor necrosis factor therapy and cancer risk in patients with autoimmune Disorders.
- CBL is frequently altered in lung cancers: its relationship to mutations in MET and EGFR tyrosine kinases.
- Translating genetic questions into clinical answers in acute myeloid leukemia.
- Genome-wide association study to identify novel loci associated with therapy-related myeloid leukemia susceptibility.
- MDM2 SNP309 and TP53 Arg72Pro interact to alter therapy-related acute myeloid leukemia susceptibility.
- Genetic predictors of long-term toxicities after radiation therapy for breast cancer.
- The broken genome: genetic and pharmacologic approaches to breaking DNA.
- The TP53 mutational spectrum and frequency of CHEK2*1100delC in Li-Fraumeni-like kindreds.
- A single nucleotide polymorphism in the MDM2 promoter attenuates the p53 tumor suppressor pathway and accelerates tumor formation in humans.
- The genetics of familial lymphomas.
- MDM2 and prognosis.
- Differential recruitment of caspase 8 to cFlip confers sensitivity or resistance to Fas-mediated apoptosis in a subset of familial lymphoma patients.
- Pediatric AML: past, present, and future.
- Breast Cancer Risk in Childhood Cancer Survivors Without a History of Chest Radiotherapy: A Report From the Childhood Cancer Survivor Study.
- A variant at 9p21.3 functionally implicates CDKN2B in paediatric B-Cell precursor acute lymphoblastic leukaemia aetiology.
Schools
Joan Sanford I Weill Medical College Of Cornell University
Columbus Hospital
Mskcc
Conditions Treated
- Anemia
- Coagulation Disorders (incl. Hemophilia)
- Neutropenia
- Osteosarcoma
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Doctors Specialties
- Pediatric Hematology
- Oncology
Accepted Insurances
Education
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New York Presbyterian Hospital -
Weill Cornell Medical College of Cornell University
Hospital
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University of Chicago Medical Center
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